Age-related demethylation of the TDP-43 autoregulatory region in the human motor cortex.


Journal

Communications biology
ISSN: 2399-3642
Titre abrégé: Commun Biol
Pays: England
ID NLM: 101719179

Informations de publication

Date de publication:
21 09 2021
Historique:
received: 21 01 2021
accepted: 01 09 2021
entrez: 22 9 2021
pubmed: 23 9 2021
medline: 15 12 2021
Statut: epublish

Résumé

In amyotrophic lateral sclerosis (ALS), TAR DNA-binding protein 43 (TDP-43), which is encoded by TARDBP, forms aggregates in the motor cortex. This aggregate formation may be triggered by an increase in the TDP-43 level with aging. However, the amount of TDP-43 is autoregulated by alternative splicing of the TARDBP 3'UTR, and how this autoregulation is affected by aging remains to be elucidated. We found that DNA demethylation in the autoregulatory region in the TARDBP 3'UTR reduced alternative splicing and increased TARDBP mRNA expression. Furthermore, in the human motor cortex, we found that this region was demethylated with aging, resulting in increased expression of TARDBP mRNA. The acceleration of DNA demethylation in the motor cortex was associated with the age of ALS onset. In summary, the dysregulation of TDP-43 autoregulation by age-related DNA demethylation in the motor cortex may explain the contribution of aging and motor system selectivity in ALS.

Identifiants

pubmed: 34548609
doi: 10.1038/s42003-021-02621-0
pii: 10.1038/s42003-021-02621-0
pmc: PMC8455575
doi:

Substances chimiques

DNA-Binding Proteins 0
TARDBP protein, human 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1107

Informations de copyright

© 2021. The Author(s).

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Auteurs

Yuka Koike (Y)

Department of Neurology, Brain Research Institute, Niigata University, Niigata city, Japan.

Akihiro Sugai (A)

Department of Molecular Neuroscience, Center for Bioresource-based Research, Brain Research Institute, Niigata University, Niigata city, Japan. akihiro.sugai@bri.niigata-u.ac.jp.

Norikazu Hara (N)

Department of Molecular Genetics, Center for Bioresource-based Research, Brain Research Institute, Niigata University, Niigata city, Japan.

Junko Ito (J)

Department of Pathology, Brain Research Institute, Niigata University, Niigata city, Japan.

Akio Yokoseki (A)

Department of Inter-Organ Communication Research, Niigata University Graduate School of Medical and Dental Sciences, Niigata city, Japan.

Tomohiko Ishihara (T)

Department of Neurology, Brain Research Institute, Niigata University, Niigata city, Japan.

Takuma Yamagishi (T)

Department of Neurology, Brain Research Institute, Niigata University, Niigata city, Japan.

Shintaro Tsuboguchi (S)

Department of Neurology, Brain Research Institute, Niigata University, Niigata city, Japan.

Mari Tada (M)

Department of Pathology Neuroscience, Center for Bioresource-based Research, Brain Research Institute, Niigata University, Niigata city, Japan.

Takeshi Ikeuchi (T)

Department of Molecular Genetics, Center for Bioresource-based Research, Brain Research Institute, Niigata University, Niigata city, Japan.

Akiyoshi Kakita (A)

Department of Pathology, Brain Research Institute, Niigata University, Niigata city, Japan.

Osamu Onodera (O)

Department of Neurology, Brain Research Institute, Niigata University, Niigata city, Japan. onodera@bri.niigata-u.ac.jp.

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