IL-17C and IL-17RE Promote Wound Closure in a
Staphylococcus aureus
interleukin-17C
wound closure
wound infection
Journal
Microorganisms
ISSN: 2076-2607
Titre abrégé: Microorganisms
Pays: Switzerland
ID NLM: 101625893
Informations de publication
Date de publication:
27 Aug 2021
27 Aug 2021
Historique:
received:
15
06
2021
revised:
06
08
2021
accepted:
13
08
2021
entrez:
28
9
2021
pubmed:
29
9
2021
medline:
29
9
2021
Statut:
epublish
Résumé
The epithelial cytokine interleukin-17C (IL-17C) mediates inflammation through the interleukin 17 receptor E (IL-17RE). Prior studies showed a detrimental role of IL-17C in the pathogenesis of immune-mediated skin diseases (e.g., psoriasis). Here, we examined the role of IL-17C/IL-17RE in wound closure in a
Identifiants
pubmed: 34576717
pii: microorganisms9091821
doi: 10.3390/microorganisms9091821
pmc: PMC8469012
pii:
doi:
Types de publication
Journal Article
Langues
eng
Subventions
Organisme : DFG and Saarland University
ID : funding program Open Access Publishing
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