Unraveling the physiological roles of retinoic acid receptor-related orphan receptor α.


Journal

Experimental & molecular medicine
ISSN: 2092-6413
Titre abrégé: Exp Mol Med
Pays: United States
ID NLM: 9607880

Informations de publication

Date de publication:
09 2021
Historique:
received: 14 06 2021
accepted: 13 07 2021
revised: 12 07 2021
pubmed: 1 10 2021
medline: 30 3 2022
entrez: 30 9 2021
Statut: ppublish

Résumé

Retinoic acid receptor-related orphan receptor-α (RORα) is a member of the orphan nuclear receptor family and functions as a transcriptional activator in response to circadian changes. Circadian rhythms are complex cellular mechanisms regulating diverse metabolic, inflammatory, and tumorigenic gene expression pathways that govern cyclic cellular physiology. Disruption of circadian regulators, including RORα, plays a critical role in tumorigenesis and facilitates the development of inflammatory hallmarks. Although RORα contributes to overall fitness among anticancer, anti-inflammatory, lipid homeostasis, and circadian clock mechanisms, the molecular mechanisms underlying the mode of transcriptional regulation by RORα remain unclear. Nonetheless, RORα has important implications for pharmacological prevention of cancer, inflammation, and metabolic diseases, and understanding context-dependent RORα regulation will provide an innovative approach for unraveling the functional link between cancer metabolism and rhythm changes.

Identifiants

pubmed: 34588606
doi: 10.1038/s12276-021-00679-8
pii: 10.1038/s12276-021-00679-8
pmc: PMC8492739
doi:

Substances chimiques

Nuclear Receptor Subfamily 1, Group F, Member 1 0
RORA protein, human 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

1278-1286

Informations de copyright

© 2021. The Author(s).

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Auteurs

Ji Min Lee (JM)

Department of Molecular Bioscience, College of Biomedical Sciences, Kangwon National University, Chuncheon, 24341, Republic of Korea.

Hyunkyung Kim (H)

Department of Biochemistry and Molecular Biology, Korea University College of Medicine, Seoul, 02841, Republic of Korea. hyunkkim@korea.ac.kr.
BK21 Graduate Program, Department of Biomedical Sciences, Korea University College of Medicine, Seoul, 02841, Republic of Korea. hyunkkim@korea.ac.kr.

Sung Hee Baek (SH)

Creative Research Initiatives Center for Epigenetic Code and Diseases, Department of Biological Sciences, Seoul National University, Seoul, 08826, Republic of Korea. sbaek@snu.ac.kr.

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