Lenvatinib versus Sorafenib as first-line treatment in hepatocellular carcinoma: A multi-institutional matched case-control study.

Lenvatinib Sorafenib hepatocarcinoma prognostic and predictor factors

Journal

Hepatology research : the official journal of the Japan Society of Hepatology
ISSN: 1386-6346
Titre abrégé: Hepatol Res
Pays: Netherlands
ID NLM: 9711801

Informations de publication

Date de publication:
Dec 2021
Historique:
revised: 16 09 2021
received: 18 06 2021
accepted: 23 09 2021
pubmed: 1 10 2021
medline: 1 10 2021
entrez: 30 9 2021
Statut: ppublish

Résumé

Advanced Hepatocarcinoma (HCC) is an important health problem worldwide. Recently, the REFLECT trial demonstrated the non-inferiority of Lenvatinib compared to Sorafenib in I line setting, thus leading to the approval of new first-line standard of care, along with Sorafenib. With aim to evaluate the optimal choice between Sorafenib and Lenvatinib as primary treatment in clinical practice, we performed a multicentric analysis with the propensity score matching on 184 HCC patients. The median overall survival (OS) were 15.2 and 10.5 months for Lenvatinib and Sorafenib arm, respectively. The median progression-free survival (PFS) was 7.0 and 4.5 months for Lenvatinib and Sorafenib arm, respectively. Patients treated with Lenvatinib showed a 36% reduction of death risk (p = 0.0156), a 29% reduction of progression risk (p = 0.0446), a higher response rate (p < 0.00001) and a higher disease control rate (p = 0.002). Sorafenib showed to be correlated with more hand-foot skin reaction and Lenvatinib with more hypertension and fatigue. We highlighted the prognostic role of Barcelona Clinic Liver Cancer (BCLC) stage, Eastern Cooperative Oncology Group Performance Status (ECOG-PS), bilirubin, alkaline phosphatase and eosinophils for Sorafenib. Conversely, albumin, aspartate aminotransferase (AST), alkaline phosphatase and Neutrophil-Lymphocyte Ratio (NLR) resulted prognostic in Lenvatinib arm. Finally, we highlighted the positive predictive role of albumin > Normal Value (NV), ECOG > 0, NLR < 3, absence of Hepatitis C Virus positivity, and presence of portal vein thrombosis in favor of Lenvatinib arm. Eosinophil < 50 and ECOG > 0 negatively predicted the response to Sorafenib. SLenvatinib showed to better perform in a real-word setting compared to Sorafenib. More researches are needed to validate the predictor factors of response to Lenvatinib rather than Sorafenib.

Sections du résumé

BACKGROUND BACKGROUND
Advanced Hepatocarcinoma (HCC) is an important health problem worldwide. Recently, the REFLECT trial demonstrated the non-inferiority of Lenvatinib compared to Sorafenib in I line setting, thus leading to the approval of new first-line standard of care, along with Sorafenib.
AIMS AND METHODS OBJECTIVE
With aim to evaluate the optimal choice between Sorafenib and Lenvatinib as primary treatment in clinical practice, we performed a multicentric analysis with the propensity score matching on 184 HCC patients.
RESULTS RESULTS
The median overall survival (OS) were 15.2 and 10.5 months for Lenvatinib and Sorafenib arm, respectively. The median progression-free survival (PFS) was 7.0 and 4.5 months for Lenvatinib and Sorafenib arm, respectively. Patients treated with Lenvatinib showed a 36% reduction of death risk (p = 0.0156), a 29% reduction of progression risk (p = 0.0446), a higher response rate (p < 0.00001) and a higher disease control rate (p = 0.002). Sorafenib showed to be correlated with more hand-foot skin reaction and Lenvatinib with more hypertension and fatigue. We highlighted the prognostic role of Barcelona Clinic Liver Cancer (BCLC) stage, Eastern Cooperative Oncology Group Performance Status (ECOG-PS), bilirubin, alkaline phosphatase and eosinophils for Sorafenib. Conversely, albumin, aspartate aminotransferase (AST), alkaline phosphatase and Neutrophil-Lymphocyte Ratio (NLR) resulted prognostic in Lenvatinib arm. Finally, we highlighted the positive predictive role of albumin > Normal Value (NV), ECOG > 0, NLR < 3, absence of Hepatitis C Virus positivity, and presence of portal vein thrombosis in favor of Lenvatinib arm. Eosinophil < 50 and ECOG > 0 negatively predicted the response to Sorafenib.
CONCLUSION CONCLUSIONS
SLenvatinib showed to better perform in a real-word setting compared to Sorafenib. More researches are needed to validate the predictor factors of response to Lenvatinib rather than Sorafenib.

Identifiants

pubmed: 34591334
doi: 10.1111/hepr.13718
doi:

Types de publication

Journal Article

Langues

eng

Pagination

1229-1241

Informations de copyright

© 2021 The Authors. Hepatology Research published by John Wiley & Sons Australia, Ltd on behalf of Japan Society of Hepatology.

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Auteurs

Margherita Rimini (M)

Division of Oncology, Department of Oncology and Hematology, University of Modena and Reggio Emilia, Modena, Italy.

Shigeo Shimose (S)

Division of Gastroenterology, Department of Medicine, Kurume University School of Medicine, Kurume, Japan.

Sara Lonardi (S)

Early Phase Clinical Trial Unit, Department of Oncology, Veneto Institute of Oncology IOV-IRCCS, Padua, Italy.
Medical Oncology Unit 1, Department of Oncology, Veneto Institute of Oncology IOV-IRCCS, Padua, Italy.

Toshifumi Tada (T)

Department of Internal Medicine, Japanese Red Cross Himeji Hospital, Himeji, Japan.

Gianluca Masi (G)

Unit of Medical Oncology, Pisa University Hospital, Pisa, Italy.

Hideki Iwamoto (H)

Division of Gastroenterology, Department of Medicine, Kurume University School of Medicine, Kurume, Japan.

Eleonora Lai (E)

Medical Oncology Unit, University Hospital and University of Cagliari, Cagliari, Italy.

Valentina Burgio (V)

Department of Medical Oncology, IRCCS San Raffaele Hospital, Milan, Italy.

Atsushi Hiraoka (A)

Gastroenterology Center, Ehime Prefectural Central Hospital, Matsuyama, Japan.

Toru Ishikawa (T)

Department of Gastroenterology, Saiseikai Niigata Hospital, Niigata, Japan.

Caterina Soldà (C)

Medical Oncology Unit 1, Department of Oncology, Veneto Institute of Oncology IOV-IRCCS, Padua, Italy.

Tomotake Shirono (T)

Division of Gastroenterology, Department of Medicine, Kurume University School of Medicine, Kurume, Japan.

Caterina Vivaldi (C)

Unit of Medical Oncology, Pisa University Hospital, Pisa, Italy.

Koichi Takaguchi (K)

Department of Hepatology, Kagawa Prefectural Central Hospital, Takamatsu, Japan.

Noritomo Shimada (N)

Division of Gastroenterology and Hepatology, Otakanomori Hospital, Kashiwa, Japan.

Giorgio Astara (G)

Medical Oncology Unit, University Hospital and University of Cagliari, Cagliari, Italy.

Hironori Koga (H)

Division of Gastroenterology, Department of Medicine, Kurume University School of Medicine, Kurume, Japan.

Kazuhiro Nouso (K)

Department of Gastroenterology, Okayama City Hospital, Okayama, Japan.

Kouji Joko (K)

Hepato-biliary Center, Matsuyama Red Cross Hospital, Matsuyama, Japan.

Takuji Torimura (T)

Division of Gastroenterology, Department of Medicine, Kurume University School of Medicine, Kurume, Japan.

Yoichi Hiasa (Y)

Department of Gastroenterology and Metabology, Ehime University Graduate School of Medicine, Matsuyama, Japan.

Francesca Salani (F)

Unit of Medical Oncology, Pisa University Hospital, Pisa, Italy.

Mario Scartozzi (M)

Medical Oncology Unit, University Hospital and University of Cagliari, Cagliari, Italy.

Stefano Cascinu (S)

Department of Medical Oncology, IRCCS San Raffaele Hospital, Milan, Italy.

Andrea Casadei-Gardini (A)

Department of Medical Oncology, IRCCS San Raffaele Hospital, Milan, Italy.

Classifications MeSH