Intestinal Microbiota Play an Important Role in the Treatment of Type I Diabetes in Mice With BefA Protein.


Journal

Frontiers in cellular and infection microbiology
ISSN: 2235-2988
Titre abrégé: Front Cell Infect Microbiol
Pays: Switzerland
ID NLM: 101585359

Informations de publication

Date de publication:
2021
Historique:
received: 02 06 2021
accepted: 02 09 2021
entrez: 4 10 2021
pubmed: 5 10 2021
medline: 21 10 2021
Statut: epublish

Résumé

More and more studies have shown that the intestinal microbiota is the main factor in the pathogenesis of type 1 diabetes mellitus (T1DM). Beta cell expansion factor A (BefA) is a protein expressed by intestinal microorganisms. It has been proven to promote the proliferation of β-cells and has broad application prospects. However, as an intestinal protein, there have not been studies and reports on its application in diabetes and its mechanism of action. In this study, a T1DM model induced by multiple low-dose STZ (MLD-STZ) injections was established, and BefA protein was administered to explore its therapeutic effect in T1DM and the potential mechanism of intestinal microbiota. BefA protein significantly reduced the blood glucose, maintained the body weight, and improved the glucose tolerance of the mice. At the same time, the BefA protein significantly increased the expression of ZO-1, Occludin, and significantly reduced the expression of TLR-4, Myd88, and p-p65/p65. BefA protein significantly reduced the relative expression of pro-inflammatory cytokines IL-1β, IL-6 and TNF-α. In addition, our high-throughput sequencing shows for the first time that the good hypoglycemic effect of BefA protein is strongly related to the increase in the abundance of the beneficial gut bacteria

Identifiants

pubmed: 34604109
doi: 10.3389/fcimb.2021.719542
pmc: PMC8485065
doi:

Substances chimiques

factor A 0
Vitamin B 12 P6YC3EG204

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

719542

Informations de copyright

Copyright © 2021 Qin, Chen, Li, Wei, Zhou, Le, Hu and Chen.

Déclaration de conflit d'intérêts

Author QQ was employed by company Harbin Meihua Biotechnology Co., Ltd. The remaining authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.

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Auteurs

Qi Qin (Q)

National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.
Harbin Meihua Biotechnology Co., Ltd, Research and Development Center, Haerbin, China.
School of Life Sciences, Lanzhou University, Lanzhou, China.

Yan Chen (Y)

Department of Dialysis, Haifushan Hospital, Weifang, China.

Yongbo Li (Y)

Department of Orthopedics, Haifushan Hospital, Weifang, China.

Jing Wei (J)

National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.

Xiaoting Zhou (X)

National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.

Fuyin Le (F)

National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.

Hong Hu (H)

School of Life Sciences, Nanchang University, Nanchang, China.
Center for Reproductive Medicine, Qingyuan Peopler's Hospital, The Sixth Affiliated Hospital of Guangzhou Medical University, Qingyuan, China.

Tingtao Chen (T)

National Engineering Research Center for Bioengineering Drugs and the Technologies, Institute of Translational Medicine, Nanchang University, Nanchang, China.

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