Burden of liver disease progression in hospitalized patients with type 2 diabetes mellitus.


Journal

Journal of hepatology
ISSN: 1600-0641
Titre abrégé: J Hepatol
Pays: Netherlands
ID NLM: 8503886

Informations de publication

Date de publication:
02 2022
Historique:
received: 20 01 2021
revised: 20 09 2021
accepted: 21 09 2021
pubmed: 5 10 2021
medline: 23 2 2022
entrez: 4 10 2021
Statut: ppublish

Résumé

There are uncertainties regarding the burden of liver disease in patients with type 2 diabetes (T2D). Thus, we aimed to quantify the burden of liver disease, identify risk factors, and estimate attributable risks in patients with T2D. We measured adjusted hazard ratios of liver disease progression to hepatocellular carcinoma and/or decompensated cirrhosis in a 2010-2020 retrospective, bicentric, longitudinal, cohort of 52,066 hospitalized patients with T2D. Mean age was 64±14 years and 58% were men. Alcohol use disorders accounted for 57% of liver-related complications and were associated with all liver-related risk factors. Non-metabolic liver-related risk factors accounted for 37% of the liver burden. T2D control was not associated with liver disease progression. The incidence (95% CI) of liver-related complications and of competing mortality were 3.9 (3.5-4.3) and 27.8 (26.7-28.9) per 1,000 person-years at risk, respectively. The cumulative incidence of liver disease progression exceeded the cumulative incidence of competing mortality only in the presence of well-identified risk factors of liver disease progression, including alcohol use. The incidence of hepatocellular carcinoma was 0.3 (95% CI 0.1-0.5) per 1,000 person-years in patients with obesity and it increased with age. The adjusted hazard ratios of liver disease progression were 55.7 (40.5-76.6), 3.5 (2.3-5.2), 8.9 (6.9-11.5), and 1.5 (1.1-2.1), for alcohol-related liver disease, alcohol use disorders without alcohol-related liver disease, non-metabolic liver-related risk factors, and obesity, respectively. The attributable fractions of alcohol use disorders, non-metabolic liver-related risk factors, and obesity to the liver burden were 55%, 14%, and 7%, respectively. In this analysis of data from 2 hospital-based cohorts of patients with T2D, alcohol use disorders, rather than obesity, contributed to most of the liver burden. These results suggest that patients with T2D should be advised to drink minimal amounts of alcohol. There is uncertainty on the burden of liver-related complications in patients with type 2 diabetes. We studied the risks of liver cancer and complications of liver disease in over 50,000 patients with type 2 diabetes. We found that alcohol was the main factor associated with complications of liver disease. This finding has major implications on the alcohol advice given to patients with type 2 diabetes.

Sections du résumé

BACKGROUND & AIMS
There are uncertainties regarding the burden of liver disease in patients with type 2 diabetes (T2D). Thus, we aimed to quantify the burden of liver disease, identify risk factors, and estimate attributable risks in patients with T2D.
METHODS
We measured adjusted hazard ratios of liver disease progression to hepatocellular carcinoma and/or decompensated cirrhosis in a 2010-2020 retrospective, bicentric, longitudinal, cohort of 52,066 hospitalized patients with T2D.
RESULTS
Mean age was 64±14 years and 58% were men. Alcohol use disorders accounted for 57% of liver-related complications and were associated with all liver-related risk factors. Non-metabolic liver-related risk factors accounted for 37% of the liver burden. T2D control was not associated with liver disease progression. The incidence (95% CI) of liver-related complications and of competing mortality were 3.9 (3.5-4.3) and 27.8 (26.7-28.9) per 1,000 person-years at risk, respectively. The cumulative incidence of liver disease progression exceeded the cumulative incidence of competing mortality only in the presence of well-identified risk factors of liver disease progression, including alcohol use. The incidence of hepatocellular carcinoma was 0.3 (95% CI 0.1-0.5) per 1,000 person-years in patients with obesity and it increased with age. The adjusted hazard ratios of liver disease progression were 55.7 (40.5-76.6), 3.5 (2.3-5.2), 8.9 (6.9-11.5), and 1.5 (1.1-2.1), for alcohol-related liver disease, alcohol use disorders without alcohol-related liver disease, non-metabolic liver-related risk factors, and obesity, respectively. The attributable fractions of alcohol use disorders, non-metabolic liver-related risk factors, and obesity to the liver burden were 55%, 14%, and 7%, respectively.
CONCLUSIONS
In this analysis of data from 2 hospital-based cohorts of patients with T2D, alcohol use disorders, rather than obesity, contributed to most of the liver burden. These results suggest that patients with T2D should be advised to drink minimal amounts of alcohol.
LAY SUMMARY
There is uncertainty on the burden of liver-related complications in patients with type 2 diabetes. We studied the risks of liver cancer and complications of liver disease in over 50,000 patients with type 2 diabetes. We found that alcohol was the main factor associated with complications of liver disease. This finding has major implications on the alcohol advice given to patients with type 2 diabetes.

Identifiants

pubmed: 34606913
pii: S0168-8278(21)02092-4
doi: 10.1016/j.jhep.2021.09.030
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

265-274

Investigateurs

Hélène Fontaine (H)
Marion Corouge (M)
Anaïs Vallet Pichard (AV)
Clémence Hollande (C)
Benoit Terris (B)
Etienne Larger (E)
Jérome Bertherat (J)
Vlad Ratziu (V)
Agnès Hartemann (A)

Informations de copyright

Copyright © 2021 European Association for the Study of the Liver. Published by Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Conflicts of interest VM: coinvestigator for Genfit, Intercept, Janssen, Novo-Nordisk, Galmed; travel fees from AbbVie. PS: coinvestigator: Genfit, Intercept, Janssen, Novo-Nordisk, Galmed; boards for MSD; travel fees AbbVie, Gilead, MSD. DT: boards for Gore, AbbVie, Gilead, Alfasigma, Shionogi, Sobi. SP: consulting and lecturing fees from Janssen, Gilead, MSD, AbbVie, Biotest, Shinogui, Viiv, LFB and grants from AbbVie, Gilead, Roche and MSD without relation to this manuscript. Please refer to the accompanying ICMJE disclosure forms for further details.

Auteurs

Vincent Mallet (V)

AP-HP.Centre Université de Paris, Groupe Hospitalier Cochin Port Royal, DMU Cancérologie et spécialités médico-chirurgicales, Département d'Hépatologie et d'Addictologie, Paris, France. Electronic address: vincent.mallet@aphp.fr.

Lucia Parlati (L)

AP-HP.Centre Université de Paris, Groupe Hospitalier Cochin Port Royal, DMU Cancérologie et spécialités médico-chirurgicales, Département d'Hépatologie et d'Addictologie, Paris, France.

Alessandro Martinino (A)

AP-HP.Centre Université de Paris, Groupe Hospitalier Cochin Port Royal, DMU Cancérologie et spécialités médico-chirurgicales, Département d'Hépatologie et d'Addictologie, Paris, France.

Juan Pablo Scarano Pereira (JP)

AP-HP.Centre Université de Paris, Groupe Hospitalier Cochin Port Royal, DMU Cancérologie et spécialités médico-chirurgicales, Département d'Hépatologie et d'Addictologie, Paris, France.

Carmen Navas Jimenez (CN)

AP-HP.Centre Université de Paris, Groupe Hospitalier Cochin Port Royal, DMU Cancérologie et spécialités médico-chirurgicales, Département d'Hépatologie et d'Addictologie, Paris, France.

Mehdi Sakka (M)

AP-HP.Sorbonne.Université, Hôpitaux Universitaires Pitié Salpétrière - Charles Foix, DMU BioGeM, Service de Biochimie Métabolique, F-75013, Paris, France.

Samir Bouam (S)

AP-HP.Centre Université de Paris, Groupe Hospitalier Cochin Port Royal, DMU PRIME, Unité d'Information Médicale, Paris, France.

Aurelia Retbi (A)

AP-HP.Sorbonne.Université, Hôpitaux Universitaires Pitié Salpêtrière - Saint-Antoine, DMU ESPRIT, Département d'Information Médicale, F75013 Paris, France.

Donika Krasteva (D)

AP-HP.Centre Université de Paris, Groupe Hospitalier Cochin Port Royal, DMU Cancérologie et spécialités médico-chirurgicales, Département d'Hépatologie et d'Addictologie, Paris, France.

Jean-François Meritet (JF)

AP-HP.Centre Université de Paris, Groupe Hospitalier Cochin Port Royal, DMU BIOPHYGEN, Service de Virologie, Paris, France.

Michaël Schwarzinger (M)

Department of methodology and innovation in prevention, Bordeaux university hospital, 33000 Bordeaux, France; University of Bordeaux, Inserm UMR 1219-Bordeaux Population Health, 33000 Bordeaux, France.

Dominique Thabut (D)

AP-HP.Sorbonne.Université, Hôpitaux Universitaires Pitié Salpêtrière - Charles Foix, DMU SAPERE, Service d'Hépatogastroentérologie, Paris France.

Pierre Rufat (P)

AP-HP.Sorbonne.Université, Hôpitaux Universitaires Pitié Salpêtrière - Saint-Antoine, DMU ESPRIT, Département d'Information Médicale, F75013 Paris, France.

Dominique Bonnefont-Rousselot (D)

AP-HP.Sorbonne.Université, Hôpitaux Universitaires Pitié Salpétrière - Charles Foix, DMU BioGeM, Service de Biochimie Métabolique, F-75013, Paris, France; Université de Paris, CNRS, INSERM, UTCBS, F-75006 Paris, France.

Philippe Sogni (P)

AP-HP.Centre Université de Paris, Groupe Hospitalier Cochin Port Royal, DMU Cancérologie et spécialités médico-chirurgicales, Département d'Hépatologie et d'Addictologie, Paris, France.

Stanislas Pol (S)

AP-HP.Centre Université de Paris, Groupe Hospitalier Cochin Port Royal, DMU Cancérologie et spécialités médico-chirurgicales, Département d'Hépatologie et d'Addictologie, Paris, France.

Emmanuel Tsochatzis (E)

UCL Institute for Liver and Digestive Health, Royal Free Hospital and UCL, London, United Kingdom.

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