Loss of CDX2 in colorectal cancer is associated with histopathologic subtypes and microsatellite instability but is prognostically inferior to hematoxylin-eosin-based morphologic parameters from the WHO classification.


Journal

British journal of cancer
ISSN: 1532-1827
Titre abrégé: Br J Cancer
Pays: England
ID NLM: 0370635

Informations de publication

Date de publication:
12 2021
Historique:
received: 09 06 2021
accepted: 16 09 2021
revised: 30 08 2021
pubmed: 8 10 2021
medline: 11 1 2022
entrez: 7 10 2021
Statut: ppublish

Résumé

Immunohistochemical loss of CDX2 has been proposed as a biomarker of dismal survival in colorectal carcinoma (CRC), especially in UICC Stage II/III. However, it remains unclear, how CDX2 expression is related to central hematoxylin-eosin (HE)-based morphologic parameters defined by 2019 WHO classification and how its prognostic relevance is compared to these parameters. We evaluated CDX2 expression in 1003 CRCs and explored its prognostic relevance compared to CRC subtypes, tumour budding and WHO grade in the overall cohort and in specific subgroups. CDX2-low/absent CRCs were enriched in specific morphologic subtypes, right-sided and microsatellite-instable (MSI-H) CRCs (P < 0.001) and showed worse survival characteristics in the overall cohort/UICC Stage II/III (e.g. DFS: P = 0.005) and in microsatellite stable and left-sided CRCs, but not in MSI-H or right-sided CRCs. Compared with CDX2, all HE-based markers showed a significantly better prognostic discrimination in all scenarios. In multivariate analyses including all morphologic parameters, CDX2 was not an independent prognostic factor. CDX2 loss has some prognostic impact in univariate analyses, but its prognostic relevance is considerably lower compared to central HE-based morphologic parameters defined by the WHO classification and vanishes in multivariate analyses incorporating these factors.

Sections du résumé

BACKGROUND
Immunohistochemical loss of CDX2 has been proposed as a biomarker of dismal survival in colorectal carcinoma (CRC), especially in UICC Stage II/III. However, it remains unclear, how CDX2 expression is related to central hematoxylin-eosin (HE)-based morphologic parameters defined by 2019 WHO classification and how its prognostic relevance is compared to these parameters.
METHODS
We evaluated CDX2 expression in 1003 CRCs and explored its prognostic relevance compared to CRC subtypes, tumour budding and WHO grade in the overall cohort and in specific subgroups.
RESULTS
CDX2-low/absent CRCs were enriched in specific morphologic subtypes, right-sided and microsatellite-instable (MSI-H) CRCs (P < 0.001) and showed worse survival characteristics in the overall cohort/UICC Stage II/III (e.g. DFS: P = 0.005) and in microsatellite stable and left-sided CRCs, but not in MSI-H or right-sided CRCs. Compared with CDX2, all HE-based markers showed a significantly better prognostic discrimination in all scenarios. In multivariate analyses including all morphologic parameters, CDX2 was not an independent prognostic factor.
CONCLUSION
CDX2 loss has some prognostic impact in univariate analyses, but its prognostic relevance is considerably lower compared to central HE-based morphologic parameters defined by the WHO classification and vanishes in multivariate analyses incorporating these factors.

Identifiants

pubmed: 34616012
doi: 10.1038/s41416-021-01553-0
pii: 10.1038/s41416-021-01553-0
pmc: PMC8651779
doi:

Substances chimiques

CDX2 Transcription Factor 0
CDX2 protein, human 0
Eosine Yellowish-(YS) TDQ283MPCW
Hematoxylin YKM8PY2Z55

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1632-1646

Informations de copyright

© 2021. The Author(s).

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Auteurs

Björn Konukiewitz (B)

Institute of Pathology, Technical University Munich, Munich, Germany.
Institute of Pathology, Christian-Albrecht University of Kiel, Kiel, Germany.

Maxime Schmitt (M)

Institute of Pathology, Technical University Munich, Munich, Germany.

Miguel Silva (M)

II Medizinische Klinik, Klinikum rechts der Isar, Technical University Munich, Munich, Germany.

Junika Pohl (J)

Institute of Pathology, Technical University Munich, Munich, Germany.

Corinna Lang (C)

Institute of Pathology, Technical University Munich, Munich, Germany.

Katja Steiger (K)

Institute of Pathology, Technical University Munich, Munich, Germany.

Kathrin Halfter (K)

Munich Cancer Registry (MCR), at the University Hospital of Munich, Institute for Medical Information Processing, Biometry, and Epidemiology (IBE), Ludwig-Maximilian-University (LMU), Munich, Germany.

Jutta Engel (J)

Munich Cancer Registry (MCR), at the University Hospital of Munich, Institute for Medical Information Processing, Biometry, and Epidemiology (IBE), Ludwig-Maximilian-University (LMU), Munich, Germany.

Anna Melissa Schlitter (AM)

Institute of Pathology, Technical University Munich, Munich, Germany.

Melanie Boxberg (M)

Institute of Pathology, Technical University Munich, Munich, Germany.

Nicole Pfarr (N)

Institute of Pathology, Technical University Munich, Munich, Germany.

Dirk Wilhelm (D)

Department of Surgery, Klinikum rechts der Isar, Technical University Munich, Munich, Germany.

Sebastian Foersch (S)

Institute of Pathology, University Hospital Mainz, Mainz, Germany.

Markus Tschurtschenthaler (M)

II Medizinische Klinik, Klinikum rechts der Isar, Technical University Munich, Munich, Germany.
Institute for Translational Cancer Research, German Cancer Consortium (DKTK), Partner Site Munich, Munich, Germany.

Wilko Weichert (W)

Institute of Pathology, Technical University Munich, Munich, Germany.
German Cancer Consortium (DKTK), Partner Site Munich, Munich, Germany.
Bavarian Cancer Center (BZKF), Munich, Germany.

Moritz Jesinghaus (M)

Institute of Pathology, Technical University Munich, Munich, Germany. moritz.jesinghaus@uni-marburg.de.
Institute of Pathology, University Hospital Marburg, Marburg, Germany. moritz.jesinghaus@uni-marburg.de.

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Classifications MeSH