Interleukin 6 predicts increased neural response during face processing in a sample of individuals with schizophrenia and healthy participants: A functional magnetic resonance imaging study.


Journal

NeuroImage. Clinical
ISSN: 2213-1582
Titre abrégé: Neuroimage Clin
Pays: Netherlands
ID NLM: 101597070

Informations de publication

Date de publication:
2021
Historique:
received: 13 07 2021
revised: 12 09 2021
accepted: 04 10 2021
pubmed: 12 10 2021
medline: 20 1 2022
entrez: 11 10 2021
Statut: ppublish

Résumé

Deficits in facial emotion recognition are a core feature of schizophrenia and predictive of functional outcome. Higher plasma levels of the cytokine interleukin 6 (IL-6) have recently been associated with poorer facial emotion recognition in individuals with schizophrenia and healthy participants, but the neural mechanisms affected remain poorly understood. Forty-nine individuals with schizophrenia or schizoaffective disorder and 158 healthy participants were imaged using functional magnetic resonance imaging during a dynamic facial emotion recognition task. Plasma IL-6 was measured from blood samples taken outside the scanner. Multiple regression was used in statistical parametric mapping software to test whether higher plasma IL-6 predicted increased neural response during task performance. Higher plasma IL-6 predicted increased bilateral medial prefrontal response during neutral face processing compared to angry face processing in the total sample (N = 207, t These findings suggest that higher peripheral IL-6 levels predict altered neural response within brain regions involved in social cognition and emotion during facial emotion recognition. This is consistent with recent neuroimaging research on IL-6 and suggesting a possible neural mechanism by which this cytokine might affect facial emotion recognition accuracy.

Sections du résumé

BACKGROUND
Deficits in facial emotion recognition are a core feature of schizophrenia and predictive of functional outcome. Higher plasma levels of the cytokine interleukin 6 (IL-6) have recently been associated with poorer facial emotion recognition in individuals with schizophrenia and healthy participants, but the neural mechanisms affected remain poorly understood.
METHODS
Forty-nine individuals with schizophrenia or schizoaffective disorder and 158 healthy participants were imaged using functional magnetic resonance imaging during a dynamic facial emotion recognition task. Plasma IL-6 was measured from blood samples taken outside the scanner. Multiple regression was used in statistical parametric mapping software to test whether higher plasma IL-6 predicted increased neural response during task performance.
RESULTS
Higher plasma IL-6 predicted increased bilateral medial prefrontal response during neutral face processing compared to angry face processing in the total sample (N = 207, t
CONCLUSIONS
These findings suggest that higher peripheral IL-6 levels predict altered neural response within brain regions involved in social cognition and emotion during facial emotion recognition. This is consistent with recent neuroimaging research on IL-6 and suggesting a possible neural mechanism by which this cytokine might affect facial emotion recognition accuracy.

Identifiants

pubmed: 34634589
pii: S2213-1582(21)00295-3
doi: 10.1016/j.nicl.2021.102851
pmc: PMC8515297
pii:
doi:

Substances chimiques

IL6 protein, human 0
Interleukin-6 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

102851

Informations de copyright

Copyright © 2021 The Author(s). Published by Elsevier Inc. All rights reserved.

Auteurs

David Mothersill (D)

Department of Psychology, School of Business, National College of Ireland, Dublin, Ireland; Center for Neuroimaging and Cognitive Genomics (NICOG), School of Psychology, National University of Ireland Galway, Ireland; Department of Psychiatry, Trinity College Dublin, St. James's Hospital, Dublin, Ireland.

Sinead King (S)

Center for Neuroimaging and Cognitive Genomics (NICOG), School of Psychology, National University of Ireland Galway, Ireland.

Laurena Holleran (L)

Center for Neuroimaging and Cognitive Genomics (NICOG), School of Psychology, National University of Ireland Galway, Ireland.

Maria Dauvermann (M)

Center for Neuroimaging and Cognitive Genomics (NICOG), School of Psychology, National University of Ireland Galway, Ireland; Institute of Psychiatry, Psychology & Neuroscience, King's College London, London, England, UK.

Saahithh Patlola (S)

Center for Neuroimaging and Cognitive Genomics (NICOG), School of Psychology, National University of Ireland Galway, Ireland.

Karolina Rokita (K)

Center for Neuroimaging and Cognitive Genomics (NICOG), School of Psychology, National University of Ireland Galway, Ireland.

Ross McManus (R)

Department of Psychiatry, Trinity College Dublin, St. James's Hospital, Dublin, Ireland.

Marcus Keynon (M)

Department of Psychiatry, Trinity College Dublin, St. James's Hospital, Dublin, Ireland.

Colm McDonald (C)

Department of Psychiatry, Clinical Science Institute, National University of Ireland Galway, Ireland.

Brian Hallahan (B)

Department of Psychiatry, Clinical Science Institute, National University of Ireland Galway, Ireland.

Aiden Corvin (A)

Department of Psychiatry, Trinity College Dublin, St. James's Hospital, Dublin, Ireland.

Derek Morris (D)

School of Natural Sciences, National University of Ireland Galway, Ireland.

John Kelly (J)

Pharmacology & Therapeutics, National University of Ireland Galway, Ireland.

Declan McKernan (D)

Pharmacology & Therapeutics, National University of Ireland Galway, Ireland.

Gary Donohoe (G)

Center for Neuroimaging and Cognitive Genomics (NICOG), School of Psychology, National University of Ireland Galway, Ireland. Electronic address: gary.donohoe@nuigalway.ie.

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