Non-obese non-alcoholic fatty liver disease (NAFLD) in Asia: an international registry study.


Journal

Metabolism: clinical and experimental
ISSN: 1532-8600
Titre abrégé: Metabolism
Pays: United States
ID NLM: 0375267

Informations de publication

Date de publication:
01 2022
Historique:
received: 04 06 2021
revised: 06 10 2021
accepted: 07 10 2021
pubmed: 15 10 2021
medline: 28 12 2021
entrez: 14 10 2021
Statut: ppublish

Résumé

A significant proportion of the non-alcoholic fatty liver disease (NAFLD) population is non-obese. Prior studies reporting the severity of NAFLD amongst non-obese patients were heterogenous. Our study, using data from the largest biopsy-proven NAFLD international registry within Asia, aims to characterize the demographic, metabolic and histological differences between non-obese and obese NAFLD patients. 1812 biopsy-proven NAFLD patients across nine countries in Asia assessed between 2006 and 2019 were pooled into a curated clinical registry. Demographic, metabolic and histological differences between non-obese and obese NAFLD patients were evaluated. The performance of Fibrosis-4 index for liver fibrosis (FIB-4) and NAFLD fibrosis score (NFS) to identify advanced liver disease across the varying obesity subgroups was compared. A random forest analysis was performed to identify novel predictors of fibrosis and steatohepatitis in non-obese patients. One-fifth (21.6%) of NAFLD patients were non-obese. Non-obese NAFLD patients had lower proportions of NASH (50.5% vs 56.5%, p = 0.033) and advanced fibrosis (14.0% vs 18.7%, p = 0.033). Metabolic syndrome in non-obese individuals was associated with NASH (OR 1.59, 95% CI 1.01-2.54, p = 0.047) and advanced fibrosis (OR 1.88, 95% CI 0.99-3.54, p = 0.051). FIB-4 performed better than the NFS score (AUROC 81.5% vs 73.7%, p < 0.001) when classifying patients with F2-4 fibrosis amongst non-obese NAFLD patients. Haemoglobin, GGT, waist circumference and cholesterol are additional variables found on random forest analysis useful for identifying non-obese NAFLD patients with advanced liver disease. A substantial proportion of non-obese NAFLD patients has NASH or advanced fibrosis. FIB-4, compared to NFS better identifies non-obese NAFLD patients with advanced liver disease. Serum GGT, cholesterol, haemoglobin and waist circumference, which are neither components of NFS nor FIB-4, are important biomarkers for advanced liver disease in non-obese patients.

Sections du résumé

BACKGROUND
A significant proportion of the non-alcoholic fatty liver disease (NAFLD) population is non-obese. Prior studies reporting the severity of NAFLD amongst non-obese patients were heterogenous. Our study, using data from the largest biopsy-proven NAFLD international registry within Asia, aims to characterize the demographic, metabolic and histological differences between non-obese and obese NAFLD patients.
METHODS
1812 biopsy-proven NAFLD patients across nine countries in Asia assessed between 2006 and 2019 were pooled into a curated clinical registry. Demographic, metabolic and histological differences between non-obese and obese NAFLD patients were evaluated. The performance of Fibrosis-4 index for liver fibrosis (FIB-4) and NAFLD fibrosis score (NFS) to identify advanced liver disease across the varying obesity subgroups was compared. A random forest analysis was performed to identify novel predictors of fibrosis and steatohepatitis in non-obese patients.
FINDINGS
One-fifth (21.6%) of NAFLD patients were non-obese. Non-obese NAFLD patients had lower proportions of NASH (50.5% vs 56.5%, p = 0.033) and advanced fibrosis (14.0% vs 18.7%, p = 0.033). Metabolic syndrome in non-obese individuals was associated with NASH (OR 1.59, 95% CI 1.01-2.54, p = 0.047) and advanced fibrosis (OR 1.88, 95% CI 0.99-3.54, p = 0.051). FIB-4 performed better than the NFS score (AUROC 81.5% vs 73.7%, p < 0.001) when classifying patients with F2-4 fibrosis amongst non-obese NAFLD patients. Haemoglobin, GGT, waist circumference and cholesterol are additional variables found on random forest analysis useful for identifying non-obese NAFLD patients with advanced liver disease.
CONCLUSION
A substantial proportion of non-obese NAFLD patients has NASH or advanced fibrosis. FIB-4, compared to NFS better identifies non-obese NAFLD patients with advanced liver disease. Serum GGT, cholesterol, haemoglobin and waist circumference, which are neither components of NFS nor FIB-4, are important biomarkers for advanced liver disease in non-obese patients.

Identifiants

pubmed: 34648769
pii: S0026-0495(21)00211-0
doi: 10.1016/j.metabol.2021.154911
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

154911

Informations de copyright

Copyright © 2021. Published by Elsevier Inc.

Déclaration de conflit d'intérêts

Declaration of competing interest No conflicts of interest exist for any of the above authors.

Auteurs

Eunice Xiang-Xuan Tan (EX)

Division of Gastroenterology and Hepatology, National University Health System, Singapore; Department of Medicine, Yong Loo Lin School of Medicine, National University of Singapore, Singapore.

Jonathan Wei-Jie Lee (JW)

Division of Gastroenterology and Hepatology, National University Health System, Singapore; Department of Medicine, Yong Loo Lin School of Medicine, National University of Singapore, Singapore.

Nur Halisah Jumat (NH)

Department of Medicine, Yong Loo Lin School of Medicine, National University of Singapore, Singapore.

Wah-Kheong Chan (WK)

University of Malaya, Kuala Lumpur, Malaysia.

Sombat Treeprasertsuk (S)

Chulalongkorn University, Bangkok, Thailand.

George Boon-Bee Goh (GB)

Singapore General Hospital, Singapore.

Jian-Gao Fan (JG)

Shanghai Jiaotong University School of Medicine, Shanghai, China.

Myeong Jun Song (MJ)

The Catholic University Korea, Republic of Korea.

Phunchai Charatcharoenwitthaya (P)

Mahidol University, Bangkok, Thailand.

Ajay Duseja (A)

Post Graduate Institute of Medical Education and Research, Chandigarh, India.

Kento Imajo (K)

Yokohama City University Graduate School of Medicine, Yokohama, Japan.

Atsushi Nakajima (A)

Yokohama City University Graduate School of Medicine, Yokohama, Japan.

Yosuke Seki (Y)

Yotsuya Medical Cube, Tokyo, Japan.

Kazunori Kasama (K)

Yotsuya Medical Cube, Tokyo, Japan.

Satoru Kakizaki (S)

Department of Gastroenterology and Hepatology, Gunma University Graduate School of Medicine, Japan.

Laurentius A Lesmana (LA)

Medistra Hospital, Jakarta, Indonesia.

Kenneth I Zheng (KI)

NAFLD Research Center, Department of Hepatology, the First Affiliated Hospital of Wenzhou Medical University, Wenzhou, China.

Ming-Hua Zheng (MH)

NAFLD Research Center, Department of Hepatology, the First Affiliated Hospital of Wenzhou Medical University, Wenzhou, China.

Calvin J Koh (CJ)

Division of Gastroenterology and Hepatology, National University Health System, Singapore; Department of Medicine, Yong Loo Lin School of Medicine, National University of Singapore, Singapore.

Khek-Yu Ho (KY)

Division of Gastroenterology and Hepatology, National University Health System, Singapore; Department of Medicine, Yong Loo Lin School of Medicine, National University of Singapore, Singapore.

Khean-Lee Goh (KL)

University of Malaya, Kuala Lumpur, Malaysia.

Vincent Wai-Sun Wong (VW)

The Chinese University of Hong Kong, Hong Kong.

Yock-Young Dan (YY)

Division of Gastroenterology and Hepatology, National University Health System, Singapore; Department of Medicine, Yong Loo Lin School of Medicine, National University of Singapore, Singapore. Electronic address: mdcdyy@nus.edu.sg.

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