Inhibition of Severe Acute Respiratory Syndrome Coronavirus 2 Replication by Hypertonic Saline Solution in Lung and Kidney Epithelial Cells.


Journal

ACS pharmacology & translational science
ISSN: 2575-9108
Titre abrégé: ACS Pharmacol Transl Sci
Pays: United States
ID NLM: 101721411

Informations de publication

Date de publication:
08 Oct 2021
Historique:
received: 15 03 2021
entrez: 15 10 2021
pubmed: 16 10 2021
medline: 16 10 2021
Statut: epublish

Résumé

An unprecedented global health crisis has been caused by a new virus called severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). We performed experiments to test if a hypertonic saline solution was capable of inhibiting virus replication. Our data show that 1.2% NaCl inhibited virus replication by 90%, achieving 100% of inhibition at 1.5% in the nonhuman primate kidney cell line Vero, and 1.1% of NaCl was sufficient to inhibit the virus replication by 88% in human epithelial lung cell line Calu-3. Furthermore, our results indicate that the inhibition is due to an intracellular mechanism and not to the dissociation of the spike SARS-CoV-2 protein and its human receptor. NaCl depolarizes the plasma membrane causing a low energy state (high ADP/ATP concentration ratio) without impairing mitochondrial function, supposedly associated with the inhibition of the SARS-CoV-2 life cycle. Membrane depolarization and intracellular energy deprivation are possible mechanisms by which the hypertonic saline solution efficiently prevents virus replication

Identifiants

pubmed: 34651104
doi: 10.1021/acsptsci.1c00080
pmc: PMC8442612
doi:

Types de publication

Journal Article

Langues

eng

Pagination

1514-1527

Informations de copyright

© 2021 American Chemical Society.

Déclaration de conflit d'intérêts

The authors declare no competing financial interest.

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Auteurs

Rafael R G Machado (RRG)

Department of Microbiology, Institute of Biomedical Sciences, University of São Paulo, São Paulo 05508, Brazil.

Talita Glaser (T)

Department of Biochemistry, Institute of Chemistry, University of São Paulo, São Paulo 05508, Brazil.

Danielle B Araujo (DB)

Department of Microbiology, Institute of Biomedical Sciences, University of São Paulo, São Paulo 05508, Brazil.
Hospital Israelita Albert Einstein, São Paulo 05652, Brazil.

Lyvia Lintzmaier Petiz (LL)

Department of Biochemistry, Institute of Chemistry, University of São Paulo, São Paulo 05508, Brazil.

Danielle B L Oliveira (DBL)

Department of Microbiology, Institute of Biomedical Sciences, University of São Paulo, São Paulo 05508, Brazil.
Hospital Israelita Albert Einstein, São Paulo 05652, Brazil.
Development and Innovation Center, Laboratory of Virology, Butantan Institute, São Paulo 05503, Brazil.

Giuliana S Durigon (GS)

Medical School Clinical Hospital, University of São Paulo, São Paulo 05508, Brazil.

Alessandra L Leal (AL)

Gaspar Vianna Clinic Hospital Foundation, Belém 66083, Brazil.

João Renato R Pinho (JRR)

Hospital Israelita Albert Einstein, São Paulo 05652, Brazil.
LIM-03, Central Laboratories Division, Clinics Hospital, São Paulo School of Medicine, University of São Paulo, São Paulo 05508, Brazil.
LIM-07, Institute of Tropical Medicine, Department of Gastroenterology, University of São Paulo School of Medicine, São Paulo 05508, Brazil.

Luis C S Ferreira (LCS)

Department of Microbiology, Institute of Biomedical Sciences, University of São Paulo, São Paulo 05508, Brazil.
Scientific Platform Pasteur USP, São Paulo 05508, Brazil.

Henning Ulrich (H)

Department of Biochemistry, Institute of Chemistry, University of São Paulo, São Paulo 05508, Brazil.

Edison L Durigon (EL)

Department of Microbiology, Institute of Biomedical Sciences, University of São Paulo, São Paulo 05508, Brazil.
Scientific Platform Pasteur USP, São Paulo 05508, Brazil.

Cristiane Rodrigues Guzzo (CR)

Department of Microbiology, Institute of Biomedical Sciences, University of São Paulo, São Paulo 05508, Brazil.

Classifications MeSH