Receptor binding domain-IgG levels correlate with protection in residents facing SARS-CoV-2 B.1.1.7 outbreaks.

BNT162b2 vaccine COVID-19 SARS-CoV-2 antibody response efficacy neutralizing antibodies nucleocapsid antigenemia nursing homes residents symptoms

Journal

Allergy
ISSN: 1398-9995
Titre abrégé: Allergy
Pays: Denmark
ID NLM: 7804028

Informations de publication

Date de publication:
06 2022
Historique:
revised: 28 09 2021
received: 10 09 2021
accepted: 01 10 2021
pubmed: 16 10 2021
medline: 7 6 2022
entrez: 15 10 2021
Statut: ppublish

Résumé

Limited information exists on nursing home (NH) residents regarding BNT162b2 vaccine efficacy in preventing SARS-CoV-2 and severe COVID-19, and its association with post-vaccine humoral response. 396 residents from seven NHs suffering a SARS-CoV-2 B.1.1.7 (VOC-α) outbreak at least 14 days after a vaccine campaign were repeatedly tested using SARS-CoV-2 real-time reverse-transcriptase polymerase chain reaction on nasopharyngeal swab test (RT-qPCR). SARS-CoV-2 receptor-binding domain (RBD) of the S1 subunit (RBD-IgG) was measured in all residents. Nucleocapsid antigenemia (N-Ag) was measured in RT-qPCR-positive residents and serum neutralizing antibodies in vaccinated residents from one NH. The incidence of positive RT-qPCR was lower in residents vaccinated by two doses (72/317; 22.7%) vs one dose (10/31; 32.3%) or non-vaccinated residents (21/48; 43.7%; p < .01). COVID-19-induced deaths were observed in 5 of the 48 non-vaccinated residents (10.4%), in 2 of the 31 who had received one dose (6.4%), and in 3 of the 317 (0.9%) who had received two doses (p = .0007). Severe symptoms were more common in infected non-vaccinated residents (10/21; 47.6%) than in infected vaccinated residents (15/72; 21.0%; p = .002). Higher levels of RBD-IgG (n = 325) were associated with a lower SARS-CoV-2 incidence. No in vitro serum neutralization activity was found for RBD-IgG levels below 1050 AU/ml. RBD-IgG levels were inversely associated with N-Ag levels, found as a risk factor of severe COVID-19. Two BNT162b2 doses are associated with a 48% reduction of SARS-CoV-2 incidence and a 91.3% reduction of death risk in residents from NHs facing a VOC-α outbreak. Post-vaccine RBD-IgG levels correlate with BNT162b2 protection against SARS-CoV-2 B.1.1.7.

Sections du résumé

BACKGROUND
Limited information exists on nursing home (NH) residents regarding BNT162b2 vaccine efficacy in preventing SARS-CoV-2 and severe COVID-19, and its association with post-vaccine humoral response.
METHODS
396 residents from seven NHs suffering a SARS-CoV-2 B.1.1.7 (VOC-α) outbreak at least 14 days after a vaccine campaign were repeatedly tested using SARS-CoV-2 real-time reverse-transcriptase polymerase chain reaction on nasopharyngeal swab test (RT-qPCR). SARS-CoV-2 receptor-binding domain (RBD) of the S1 subunit (RBD-IgG) was measured in all residents. Nucleocapsid antigenemia (N-Ag) was measured in RT-qPCR-positive residents and serum neutralizing antibodies in vaccinated residents from one NH.
RESULTS
The incidence of positive RT-qPCR was lower in residents vaccinated by two doses (72/317; 22.7%) vs one dose (10/31; 32.3%) or non-vaccinated residents (21/48; 43.7%; p < .01). COVID-19-induced deaths were observed in 5 of the 48 non-vaccinated residents (10.4%), in 2 of the 31 who had received one dose (6.4%), and in 3 of the 317 (0.9%) who had received two doses (p = .0007). Severe symptoms were more common in infected non-vaccinated residents (10/21; 47.6%) than in infected vaccinated residents (15/72; 21.0%; p = .002). Higher levels of RBD-IgG (n = 325) were associated with a lower SARS-CoV-2 incidence. No in vitro serum neutralization activity was found for RBD-IgG levels below 1050 AU/ml. RBD-IgG levels were inversely associated with N-Ag levels, found as a risk factor of severe COVID-19.
CONCLUSIONS
Two BNT162b2 doses are associated with a 48% reduction of SARS-CoV-2 incidence and a 91.3% reduction of death risk in residents from NHs facing a VOC-α outbreak. Post-vaccine RBD-IgG levels correlate with BNT162b2 protection against SARS-CoV-2 B.1.1.7.

Identifiants

pubmed: 34652831
doi: 10.1111/all.15142
pmc: PMC8652754
doi:

Substances chimiques

Antibodies, Viral 0
COVID-19 Vaccines 0
Immunoglobulin G 0
Vaccines 0
BNT162 Vaccine N38TVC63NU

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1885-1894

Informations de copyright

© 2021 European Academy of Allergy and Clinical Immunology and John Wiley & Sons Ltd.

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Auteurs

Hubert Blain (H)

Department of Internal Medicine and Geriatrics, MUSE University, Montpellier, France.

Edouard Tuaillon (E)

Pathogenesis and Control of Chronic and Emerging Infections, University of Montpellier, INSERM, EFS, Antilles University, University Hospital, Montpellier, France.

Lucie Gamon (L)

Clinical Research and Epidemiology Unit, University Hospital, Montpellier, France.

Amandine Pisoni (A)

Pathogenesis and Control of Chronic and Emerging Infections, University of Montpellier, INSERM, EFS, Antilles University, University Hospital, Montpellier, France.

Stéphanie Miot (S)

Department of Internal Medicine and Geriatrics, MUSE University, Montpellier, France.

Valentin Delpui (V)

Department of Internal Medicine and Geriatrics, MUSE University, Montpellier, France.

Nejm Si-Mohamed (N)

Department of Internal Medicine and Geriatrics, MUSE University, Montpellier, France.

Clémence Niel (C)

Pathogenesis and Control of Chronic and Emerging Infections, University of Montpellier, INSERM, EFS, Antilles University, University Hospital, Montpellier, France.

Yves Rolland (Y)

Gérontopôle de Toulouse, INSERM 1027, Toulouse, France.

Brigitte Montes (B)

Pathogenesis and Control of Chronic and Emerging Infections, University of Montpellier, INSERM, EFS, Antilles University, University Hospital, Montpellier, France.

Soraya Groc (S)

Pathogenesis and Control of Chronic and Emerging Infections, University of Montpellier, INSERM, EFS, Antilles University, University Hospital, Montpellier, France.

Sophia Rafasse (S)

CEMIPAI, University of Montpellier, UAR3725, CNRS, Montpellier, France.
Institute of Research in Infectiology of Montpellier (IRIM), University of Montpellier, UMR9004, CNRS, Montpellier, France.

Anne-Marie Dupuy (AM)

Biochemistry and Hormonology Laboratory, University Hospital, Montpellier, France.

Nathalie Gros (N)

CEMIPAI, University of Montpellier, UAR3725, CNRS, Montpellier, France.
Institute of Research in Infectiology of Montpellier (IRIM), University of Montpellier, UMR9004, CNRS, Montpellier, France.

Delphine Muriaux (D)

CEMIPAI, University of Montpellier, UAR3725, CNRS, Montpellier, France.
Institute of Research in Infectiology of Montpellier (IRIM), University of Montpellier, UMR9004, CNRS, Montpellier, France.

Marie-Christine Picot (MC)

Clinical Research and Epidemiology Unit, University Hospital, Montpellier, France.

Jean Bousquet (J)

Department of Dermatology and Allergy, Universitätsmedizin, Berlin, Germany.
University Hospital, Montpellier, France.

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