Cyclin-dependent kinase 1 as a potential target for lycorine against hepatocellular carcinoma.


Journal

Biochemical pharmacology
ISSN: 1873-2968
Titre abrégé: Biochem Pharmacol
Pays: England
ID NLM: 0101032

Informations de publication

Date de publication:
11 2021
Historique:
received: 08 07 2021
revised: 03 10 2021
accepted: 04 10 2021
pubmed: 22 10 2021
medline: 7 1 2022
entrez: 21 10 2021
Statut: ppublish

Résumé

The pathological changes and possible underlying molecular mechanisms of hepatocellular carcinoma (HCC) are currently unclear. Effective treatment of this pathological state remains a challenge. The purpose of this study is to obtain some key genes with diagnostic and prognostic meaning and to identify potential therapeutic agents for HCC treatment. Here, CDK1, CCNB1 and CCNB2 were found to be highly expressed in HCC patients and accompanied by poor prognosis, and knockdown of them by siRNA drastically induced autophagy and senescence in hepatoma cells. Simultaneously, the anti-HCC effect of lycorine was comparable to that of interfering with these three genes, and lycorine significantly promoted the decrease both in protein and mRNA expression of CDK1. Molecular validation mechanistically demonstrated that lycorine might attenuate the degradation rate of CDK1 via interaction with it, which had been confirmed by cellular thermal shift assay and drug affinity responsive targets stability assay. Taken together, these findings suggested that CDK1, CCNB1 and CCNB2 could be regarded as potential diagnostic and prognostic biomarkers for HCC, and CDK1 might serve as a promising therapeutic target for lycorine against HCC.

Identifiants

pubmed: 34673013
pii: S0006-2952(21)00422-6
doi: 10.1016/j.bcp.2021.114806
pii:
doi:

Substances chimiques

Amaryllidaceae Alkaloids 0
Antineoplastic Agents 0
CCNB1 protein, human 0
CCNB2 protein, human 0
Cyclin B1 0
Cyclin B2 0
Phenanthridines 0
CDC2 Protein Kinase EC 2.7.11.22
CDK1 protein, human EC 2.7.11.22
lycorine I9Q105R5BU

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

114806

Informations de copyright

Copyright © 2021 Elsevier Inc. All rights reserved.

Auteurs

Shuangshuang Yin (S)

State Key Laboratory of Component-based Chinese Medicine, Tianjin University of Traditional Chinese Medicine, Tianjin 300193, China.

Shenshen Yang (S)

State Key Laboratory of Component-based Chinese Medicine, Tianjin University of Traditional Chinese Medicine, Tianjin 300193, China.

Yanming Luo (Y)

State Key Laboratory of Component-based Chinese Medicine, Tianjin University of Traditional Chinese Medicine, Tianjin 300193, China.

Jia Lu (J)

State Key Laboratory of Component-based Chinese Medicine, Tianjin University of Traditional Chinese Medicine, Tianjin 300193, China.

Gaoyong Hu (G)

State Key Laboratory of Component-based Chinese Medicine, Tianjin University of Traditional Chinese Medicine, Tianjin 300193, China.

Kailong Wang (K)

State Key Laboratory of Component-based Chinese Medicine, Tianjin University of Traditional Chinese Medicine, Tianjin 300193, China.

Yingying Shao (Y)

State Key Laboratory of Component-based Chinese Medicine, Tianjin University of Traditional Chinese Medicine, Tianjin 300193, China.

Shiyue Zhou (S)

State Key Laboratory of Component-based Chinese Medicine, Tianjin University of Traditional Chinese Medicine, Tianjin 300193, China.

Sangho Koo (S)

Department of Chemistry, Myongji University, Yongin, Gyeonggi-Do 17058, South Korea.

Yuling Qiu (Y)

School of Pharmacy, Tianjin Medical University, Tianjin 300070, China. Electronic address: qiuyuling@tmu.edu.cn.

Tao Wang (T)

State Key Laboratory of Component-based Chinese Medicine, Tianjin University of Traditional Chinese Medicine, Tianjin 300193, China. Electronic address: wangtao@tjutcm.edu.cn.

Haiyang Yu (H)

State Key Laboratory of Component-based Chinese Medicine, Tianjin University of Traditional Chinese Medicine, Tianjin 300193, China. Electronic address: hyyu@tjutcm.edu.cn.

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Classifications MeSH