Predictors of neutralizing antibody response to BNT162b2 vaccination in allogeneic hematopoietic stem cell transplant recipients.


Journal

Journal of hematology & oncology
ISSN: 1756-8722
Titre abrégé: J Hematol Oncol
Pays: England
ID NLM: 101468937

Informations de publication

Date de publication:
24 10 2021
Historique:
received: 05 08 2021
accepted: 13 10 2021
entrez: 25 10 2021
pubmed: 26 10 2021
medline: 16 11 2021
Statut: epublish

Résumé

Factors affecting response to SARS-CoV-2 mRNA vaccine in allogeneic hematopoietic stem cell transplantation (allo-HCT) recipients remain to be elucidated. Forty allo-HCT recipients were included in a study of immunization with BNT162b2 mRNA vaccine at days 0 and 21. Binding antibodies (Ab) to SARS-CoV-2 receptor binding domain (RBD) were assessed at days 0, 21, 28, and 49 while neutralizing Ab against SARS-CoV-2 wild type (NT50) were assessed at days 0 and 49. Results observed in allo-HCT patients were compared to those obtained in 40 healthy adults naive of SARS-CoV-2 infection. Flow cytometry analysis of peripheral blood cells was performed before vaccination to identify potential predictors of Ab responses. Three patients had detectable anti-RBD Ab before vaccination. Among the 37 SARS-CoV-2 naive patients, 20 (54%) and 32 (86%) patients had detectable anti-RBD Ab 21 days and 49 days postvaccination. Comparing anti-RBD Ab levels in allo-HCT recipients and healthy adults, we observed significantly lower anti-RBD Ab levels in allo-HCT recipients at days 21, 28 and 49. Further, 49% of allo-HCT patients versus 88% of healthy adults had detectable NT50 Ab at day 49 while allo-HCT recipients had significantly lower NT50 Ab titers than healthy adults (P = 0.0004). Ongoing moderate/severe chronic GVHD (P < 0.01) as well as rituximab administration in the year prior to vaccination (P < 0.05) correlated with low anti-RBD and NT50 Ab titers at 49 days after the first vaccination in multivariate analyses. Compared to healthy adults, allo-HCT patients without chronic GVHD or rituximab therapy had comparable anti-RBD Ab levels and NT50 Ab titers at day 49. Flow cytometry analyses before vaccination indicated that Ab responses in allo-HCT patients were strongly correlated with the number of memory B cells and of naive CD4 Chronic GVHD and rituximab administration in allo-HCT recipients are associated with reduced Ab responses to BNT162b2 vaccination. Immunological markers could help identify allo-HCT patients at risk of poor Ab response to mRNA vaccination. The study was registered at clinicaltrialsregister.eu on 11 March 2021 (EudractCT # 2021-000673-83).

Sections du résumé

BACKGROUND
Factors affecting response to SARS-CoV-2 mRNA vaccine in allogeneic hematopoietic stem cell transplantation (allo-HCT) recipients remain to be elucidated.
METHODS
Forty allo-HCT recipients were included in a study of immunization with BNT162b2 mRNA vaccine at days 0 and 21. Binding antibodies (Ab) to SARS-CoV-2 receptor binding domain (RBD) were assessed at days 0, 21, 28, and 49 while neutralizing Ab against SARS-CoV-2 wild type (NT50) were assessed at days 0 and 49. Results observed in allo-HCT patients were compared to those obtained in 40 healthy adults naive of SARS-CoV-2 infection. Flow cytometry analysis of peripheral blood cells was performed before vaccination to identify potential predictors of Ab responses.
RESULTS
Three patients had detectable anti-RBD Ab before vaccination. Among the 37 SARS-CoV-2 naive patients, 20 (54%) and 32 (86%) patients had detectable anti-RBD Ab 21 days and 49 days postvaccination. Comparing anti-RBD Ab levels in allo-HCT recipients and healthy adults, we observed significantly lower anti-RBD Ab levels in allo-HCT recipients at days 21, 28 and 49. Further, 49% of allo-HCT patients versus 88% of healthy adults had detectable NT50 Ab at day 49 while allo-HCT recipients had significantly lower NT50 Ab titers than healthy adults (P = 0.0004). Ongoing moderate/severe chronic GVHD (P < 0.01) as well as rituximab administration in the year prior to vaccination (P < 0.05) correlated with low anti-RBD and NT50 Ab titers at 49 days after the first vaccination in multivariate analyses. Compared to healthy adults, allo-HCT patients without chronic GVHD or rituximab therapy had comparable anti-RBD Ab levels and NT50 Ab titers at day 49. Flow cytometry analyses before vaccination indicated that Ab responses in allo-HCT patients were strongly correlated with the number of memory B cells and of naive CD4
CONCLUSIONS
Chronic GVHD and rituximab administration in allo-HCT recipients are associated with reduced Ab responses to BNT162b2 vaccination. Immunological markers could help identify allo-HCT patients at risk of poor Ab response to mRNA vaccination.
TRIAL REGISTRATION
The study was registered at clinicaltrialsregister.eu on 11 March 2021 (EudractCT # 2021-000673-83).

Identifiants

pubmed: 34689821
doi: 10.1186/s13045-021-01190-3
pii: 10.1186/s13045-021-01190-3
pmc: PMC8542409
doi:

Substances chimiques

Antibodies, Neutralizing 0
COVID-19 Vaccines 0
BNT162 Vaccine N38TVC63NU

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

174

Subventions

Organisme : Université de Liège
ID : Crédits Sectoriels de Recherche en Sciences de la Santé (FSR 2021)

Informations de copyright

© 2021. The Author(s).

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Auteurs

Lorenzo Canti (L)

Laboratory of Hematology, GIGA-I3, University of Liege and CHU of Liège, Liege, Belgium.

Stéphanie Humblet-Baron (S)

Department of Microbiology, Immunology and Transplantation, Laboratory of Adaptive Immunology, KU Leuven, Leuven, Belgium.

Isabelle Desombere (I)

SD Infectious Diseases in Humans, Sciensano, 642 Engelandstraat, 1180, Ukkel, Belgium.

Julika Neumann (J)

Department of Microbiology, Immunology and Transplantation, Laboratory of Adaptive Immunology, KU Leuven, Leuven, Belgium.

Pieter Pannus (P)

SD Infectious Diseases in Humans, Sciensano, 642 Engelandstraat, 1180, Ukkel, Belgium.

Leo Heyndrickx (L)

Virology Unit, Department of Biomedical Sciences, Institute of Tropical Medicine, 155 Nationalestraat, 2000, Antwerp, Belgium.

Aurélie Henry (A)

Division of Hematology, Department of Medicine, CHU of Liège, Liège, Belgium.

Sophie Servais (S)

Laboratory of Hematology, GIGA-I3, University of Liege and CHU of Liège, Liege, Belgium.
Division of Hematology, Department of Medicine, CHU of Liège, Liège, Belgium.

Evelyne Willems (E)

Division of Hematology, Department of Medicine, CHU of Liège, Liège, Belgium.

Grégory Ehx (G)

Laboratory of Hematology, GIGA-I3, University of Liege and CHU of Liège, Liege, Belgium.

Stanislas Goriely (S)

Institute for Medical Immunology and ULB Center for Research in Immunology (U-CRI), Université Libre de Bruxelles (ULB), Gosselies, Belgium.

Laurence Seidel (L)

Department of Biostatistics, University Hospital of Liège, Liège, Belgium.

Johan Michiels (J)

Virology Unit, Department of Biomedical Sciences, Institute of Tropical Medicine, 155 Nationalestraat, 2000, Antwerp, Belgium.

Betty Willems (B)

Virology Unit, Department of Biomedical Sciences, Institute of Tropical Medicine, 155 Nationalestraat, 2000, Antwerp, Belgium.

Adrian Liston (A)

Department of Microbiology, Immunology and Transplantation, Laboratory of Adaptive Immunology, KU Leuven, Leuven, Belgium.
Laboratory of Lymphocyte Signalling and Development, The Babraham Institute, Cambridge, UK.

Kevin K Ariën (KK)

Virology Unit, Department of Biomedical Sciences, Institute of Tropical Medicine, 155 Nationalestraat, 2000, Antwerp, Belgium.

Yves Beguin (Y)

Laboratory of Hematology, GIGA-I3, University of Liege and CHU of Liège, Liege, Belgium.
Division of Hematology, Department of Medicine, CHU of Liège, Liège, Belgium.

Maria E Goossens (ME)

SD Infectious Diseases in Humans, Sciensano, 642 Engelandstraat, 1180, Ukkel, Belgium.

Arnaud Marchant (A)

Institute for Medical Immunology and ULB Center for Research in Immunology (U-CRI), Université Libre de Bruxelles (ULB), Gosselies, Belgium.

Frédéric Baron (F)

Laboratory of Hematology, GIGA-I3, University of Liege and CHU of Liège, Liege, Belgium. f.baron@uliege.be.
Division of Hematology, Department of Medicine, CHU of Liège, Liège, Belgium. f.baron@uliege.be.
Department of Hematology, University of Liège, CHU Sart-Tilman, 4000, Liège, Belgium. f.baron@uliege.be.

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