Treatment of Device-Related Thrombosis After Left Atrial Appendage Occlusion: Initial Experience With Low-Dose Apixaban.


Journal

Cardiovascular revascularization medicine : including molecular interventions
ISSN: 1878-0938
Titre abrégé: Cardiovasc Revasc Med
Pays: United States
ID NLM: 101238551

Informations de publication

Date de publication:
07 2022
Historique:
received: 21 09 2021
revised: 01 11 2021
accepted: 04 11 2021
pubmed: 11 11 2021
medline: 28 6 2022
entrez: 10 11 2021
Statut: ppublish

Résumé

Left atrial appendage (LAA) closure devices require short-term postprocedural oral antithrombotic therapy (AT) to prevent device-related thrombosis (DRT) until device endothelialization occurs. Currently, there is no consensus regarding the optimal AT strategy for DRT prevention. The purpose of our case series is to summarize our experience using apixaban at reduced doses for effectively treating DRT. Among a total of 180 patients, 11 patients (6.1%) presented DRT and 4 were specifically treated with low-dose apixaban (2.5 mg/12 h). The mean CHA2DS2-VASc and HAS-BLED scores were high [5 (SD ±1.2) and 3.25 (SD ±0.5), respectively] and all patients had history of a major hemorrhagic event (BARC Score ≥3) mostly gastrointestinal (n = 3). An Amplazer Amulet device was implanted in 3 patients, and a LAmbre system in one patient. AT strategy at the time of DRT diagnosis was consistently single antiplatelet therapy in all patients. Following DRT diagnosis, reduced dose of apixaban was initiated in all the patients. No major or minor bleeding events occurred during apixaban administration. Apixaban low dose regimen could be a feasible option to prevent DRT while keeping a low risk of bleeding.

Sections du résumé

BACKGROUND
Left atrial appendage (LAA) closure devices require short-term postprocedural oral antithrombotic therapy (AT) to prevent device-related thrombosis (DRT) until device endothelialization occurs. Currently, there is no consensus regarding the optimal AT strategy for DRT prevention.
METHODS
The purpose of our case series is to summarize our experience using apixaban at reduced doses for effectively treating DRT.
RESULTS
Among a total of 180 patients, 11 patients (6.1%) presented DRT and 4 were specifically treated with low-dose apixaban (2.5 mg/12 h). The mean CHA2DS2-VASc and HAS-BLED scores were high [5 (SD ±1.2) and 3.25 (SD ±0.5), respectively] and all patients had history of a major hemorrhagic event (BARC Score ≥3) mostly gastrointestinal (n = 3). An Amplazer Amulet device was implanted in 3 patients, and a LAmbre system in one patient. AT strategy at the time of DRT diagnosis was consistently single antiplatelet therapy in all patients. Following DRT diagnosis, reduced dose of apixaban was initiated in all the patients. No major or minor bleeding events occurred during apixaban administration.
CONCLUSIONS
Apixaban low dose regimen could be a feasible option to prevent DRT while keeping a low risk of bleeding.

Identifiants

pubmed: 34753670
pii: S1553-8389(21)00734-X
doi: 10.1016/j.carrev.2021.11.007
pii:
doi:

Substances chimiques

Pyrazoles 0
Pyridones 0
apixaban 3Z9Y7UWC1J

Types de publication

Letter

Langues

eng

Sous-ensembles de citation

IM

Pagination

201-203

Informations de copyright

Copyright © 2021 Elsevier Inc. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest None.

Auteurs

Eduardo Flores-Umanzor (E)

Institut Clínic Cardiovascular, Hospital Clínic, Universitat de Barcelona, Spain.

Pedro Cepas-Guillen (P)

Institut Clínic Cardiovascular, Hospital Clínic, Universitat de Barcelona, Spain.

Ander Regueiro (A)

Institut Clínic Cardiovascular, Hospital Clínic, Universitat de Barcelona, Spain.

Laura Sanchis (L)

Institut Clínic Cardiovascular, Hospital Clínic, Universitat de Barcelona, Spain.

Felipe Unigarro (F)

Anesthesiology Department, Hospital Clinic, Spain.

Salvatore Brugaletta (S)

Institut Clínic Cardiovascular, Hospital Clínic, Universitat de Barcelona, Spain.

Marta Sitges (M)

Institut Clínic Cardiovascular, Hospital Clínic, Universitat de Barcelona, Spain; Institut d'Investigacions Biomèdiques August Pi i Sunyer (IDIBAPS), Spain; Centro de Investigación Biomédica en Red (CIBERCV), Instituto de Salud Carlos III, Spain.

Manel Sabaté (M)

Institut Clínic Cardiovascular, Hospital Clínic, Universitat de Barcelona, Spain; Centro de Investigación Biomédica en Red (CIBERCV), Instituto de Salud Carlos III, Spain.

Xavier Freixa (X)

Institut Clínic Cardiovascular, Hospital Clínic, Universitat de Barcelona, Spain. Electronic address: freixa@clinic.cat.

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Classifications MeSH