Emerging roles of N-linked glycosylation in brain physiology and disorders.

Alzheimer’s disease N-linked glycosylation carbohydrate metabolism neurodegeneration neuroinflammation

Journal

Trends in endocrinology and metabolism: TEM
ISSN: 1879-3061
Titre abrégé: Trends Endocrinol Metab
Pays: United States
ID NLM: 9001516

Informations de publication

Date de publication:
12 2021
Historique:
received: 27 07 2021
revised: 10 09 2021
accepted: 23 09 2021
pubmed: 11 11 2021
medline: 28 4 2022
entrez: 10 11 2021
Statut: ppublish

Résumé

N-linked glycosylation is a complex, co- and post-translational series of events that connects metabolism to signaling in almost all cells. Metabolic assembly of N-linked glycans spans multiple cellular compartments, and early N-linked glycan biosynthesis is a central mediator of protein folding and the unfolded protein response (UPR). In the brain, N-linked glycosylated proteins participate in a myriad of processes, from electrical gradients to neurotransmission. However, it is less clear how perturbations in N-linked glycosylation impact and even potentially drive aspects of neurological disorders. In this review, we discuss our current understanding of the metabolic origins of N-linked glycans in the brain, their role in modulating neuronal function, and how aberrant N-linked glycosylation can drive neurological disorders.

Identifiants

pubmed: 34756776
pii: S1043-2760(21)00221-6
doi: 10.1016/j.tem.2021.09.006
pmc: PMC8589112
mid: NIHMS1744697
pii:
doi:

Substances chimiques

Polysaccharides 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

980-993

Subventions

Organisme : NINDS NIH HHS
ID : R35 NS116824
Pays : United States
Organisme : NCI NIH HHS
ID : T32 CA165990
Pays : United States
Organisme : NCI NIH HHS
ID : F99 CA264165
Pays : United States
Organisme : NINDS NIH HHS
ID : P01 NS097197
Pays : United States
Organisme : NIA NIH HHS
ID : R01 AG066653
Pays : United States

Informations de copyright

Copyright © 2021 Elsevier Ltd. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of interests R.C.S. and M.S.G. are consultants for Maze Therapeutics, M.S.G. is a consultant for Enable Therapeutics, Glut1-Deficiency Syndrome Foundation, and Chelsea's Hope. M.S.G. and R.C.S. are founders of Atterogen, LLC.

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Auteurs

Lindsey R Conroy (LR)

Department of Neuroscience, University of Kentucky College of Medicine, Lexington, KY 40508-0536, USA; Markey Cancer Center, Lexington, KY 40508-0536, USA.

Tara R Hawkinson (TR)

Department of Neuroscience, University of Kentucky College of Medicine, Lexington, KY 40508-0536, USA.

Lyndsay E A Young (LEA)

Department of Molecular and Cellular Biochemistry University of Kentucky College of Medicine, Lexington, KY 40508-0536, USA.

Matthew S Gentry (MS)

Department of Molecular and Cellular Biochemistry University of Kentucky College of Medicine, Lexington, KY 40508-0536, USA.

Ramon C Sun (RC)

Department of Neuroscience, University of Kentucky College of Medicine, Lexington, KY 40508-0536, USA; Markey Cancer Center, Lexington, KY 40508-0536, USA; Sanders Brown Center for Aging, Lexington, KY 40508-0536, USA. Electronic address: ramon.sun@uky.edu.

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Classifications MeSH