Serum Soluble OX40 as a Diagnostic and Prognostic Biomarker for Drug-Induced Hypersensitivity Syndrome/Drug Reaction with Eosinophilia and Systemic Symptoms.


Journal

The journal of allergy and clinical immunology. In practice
ISSN: 2213-2201
Titre abrégé: J Allergy Clin Immunol Pract
Pays: United States
ID NLM: 101597220

Informations de publication

Date de publication:
02 2022
Historique:
received: 09 05 2021
revised: 07 09 2021
accepted: 15 10 2021
pubmed: 11 11 2021
medline: 19 2 2022
entrez: 10 11 2021
Statut: ppublish

Résumé

Drug-induced hypersensitivity syndrome/drug reaction with eosinophilia and systemic symptoms (DIHS/DRESS) is a severe adverse drug reaction commonly associated with the reactivation of human herpesvirus 6 (HHV-6). There are currently no adequate biomarkers for the early diagnosis and detection of DIHS/DRESS. Notably, OX40 (CD134) has an important role in allergic inflammation and functions as a cellular receptor for HHV-6 entry. We previously reported that the membrane-bound form of OX40 in CD4 We sought to investigate the clinical significance of serum soluble OX40 (sOX40) in DIHS/DRESS. Serum sOX40 levels in patients with DIHS/DRESS (n = 39), maculopapular exanthema/erythema multiforme (n = 17), Stevens-Johnson syndrome/toxic epidermal necrolysis (n = 13), or autoimmune bullous diseases (n = 5), and levels in healthy volunteers (n = 5) were examined by enzyme-linked immunosorbent assay. Copy numbers of HHV-6, HHV-7, and cytomegalovirus in peripheral blood mononuclear cells were quantified using real-time PCR. Serum sOX40 levels in patients with DIHS/DRESS in the acute stage were elevated in parallel with high OX40 expression on CD4 Serum sOX40 levels can be a useful diagnostic marker for DIHS/DRESS that reflect disease severity. Elevated serum sOX40 levels also predict HHV-6 reactivation in patients with DIHS/DRESS.

Sections du résumé

BACKGROUND
Drug-induced hypersensitivity syndrome/drug reaction with eosinophilia and systemic symptoms (DIHS/DRESS) is a severe adverse drug reaction commonly associated with the reactivation of human herpesvirus 6 (HHV-6). There are currently no adequate biomarkers for the early diagnosis and detection of DIHS/DRESS. Notably, OX40 (CD134) has an important role in allergic inflammation and functions as a cellular receptor for HHV-6 entry. We previously reported that the membrane-bound form of OX40 in CD4
OBJECTIVE
We sought to investigate the clinical significance of serum soluble OX40 (sOX40) in DIHS/DRESS.
METHODS
Serum sOX40 levels in patients with DIHS/DRESS (n = 39), maculopapular exanthema/erythema multiforme (n = 17), Stevens-Johnson syndrome/toxic epidermal necrolysis (n = 13), or autoimmune bullous diseases (n = 5), and levels in healthy volunteers (n = 5) were examined by enzyme-linked immunosorbent assay. Copy numbers of HHV-6, HHV-7, and cytomegalovirus in peripheral blood mononuclear cells were quantified using real-time PCR.
RESULTS
Serum sOX40 levels in patients with DIHS/DRESS in the acute stage were elevated in parallel with high OX40 expression on CD4
CONCLUSIONS
Serum sOX40 levels can be a useful diagnostic marker for DIHS/DRESS that reflect disease severity. Elevated serum sOX40 levels also predict HHV-6 reactivation in patients with DIHS/DRESS.

Identifiants

pubmed: 34757063
pii: S2213-2198(21)01195-8
doi: 10.1016/j.jaip.2021.10.042
pii:
doi:

Substances chimiques

Pharmaceutical Preparations 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

558-565.e4

Informations de copyright

Copyright © 2021 American Academy of Allergy, Asthma & Immunology. Published by Elsevier Inc. All rights reserved.

Auteurs

Yasuhiro Mitsui (Y)

Department of Dermatology, Nara Medical University School of Medicine, Kashihara, Japan.

Satoru Shinkuma (S)

Department of Dermatology, Nara Medical University School of Medicine, Kashihara, Japan.

Yuki Nakamura-Nishimura (Y)

Department of Dermatology, Nara Medical University School of Medicine, Kashihara, Japan.

Rie Ommori (R)

Department of Dermatology, Nara Medical University School of Medicine, Kashihara, Japan.

Kohei Ogawa (K)

Department of Dermatology, Nara Medical University School of Medicine, Kashihara, Japan.

Fumi Miyagawa (F)

Department of Dermatology, Nara Medical University School of Medicine, Kashihara, Japan.

Yasuko Mori (Y)

Division of Clinical Virology, Center for Infectious Diseases, Kobe University Graduate School of Medicine, Kobe, Japan.

Mikiko Tohyama (M)

Department of Dermatology, National Hospital Organization Shikoku Cancer Center, Matsuyama, Japan.

Hideo Asada (H)

Department of Dermatology, Nara Medical University School of Medicine, Kashihara, Japan. Electronic address: asadah@naramed-u.ac.jp.

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