Interferon Lambda Regulates Cellular and Humoral Immunity in Pristane-Induced Lupus.
Ifnlr1
SLE
autoimmunity
interferon lambda
lupus
pristane
type III interferons
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
29 Oct 2021
29 Oct 2021
Historique:
received:
21
09
2021
revised:
21
10
2021
accepted:
26
10
2021
entrez:
13
11
2021
pubmed:
14
11
2021
medline:
21
12
2021
Statut:
epublish
Résumé
A pivotal role of type I interferons in systemic lupus erythematosus (SLE) is widely accepted. Type III interferons (IFN-λ) however, the most recently discovered cytokines grouped within the interferon family, have not been extensively studied in lupus disease models yet. Growing evidence suggests a role for IFN-λ in regulating both innate and adaptive immune responses, and increased serum concentrations have been described in multiple autoimmune diseases including SLE. Using the pristane-induced lupus model, we found that mice with defective IFN-λ receptors (
Identifiants
pubmed: 34769174
pii: ijms222111747
doi: 10.3390/ijms222111747
pmc: PMC8584021
pii:
doi:
Substances chimiques
IFNLR1 protein, mouse
0
Receptors, Interferon
0
Terpenes
0
pristane
26HZV48DT1
Interferons
9008-11-1
Interferon Lambda
0
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : Deutsche Forschungsgemeinschaft
ID : CRC 1160 project 12 505 and TRR 130, project 12
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