Atrial fibrillation after closure of patent foramen ovale in the REDUCE clinical study.


Journal

Catheterization and cardiovascular interventions : official journal of the Society for Cardiac Angiography & Interventions
ISSN: 1522-726X
Titre abrégé: Catheter Cardiovasc Interv
Pays: United States
ID NLM: 100884139

Informations de publication

Date de publication:
04 2022
Historique:
revised: 27 10 2021
received: 16 08 2021
accepted: 04 11 2021
pubmed: 14 11 2021
medline: 30 4 2022
entrez: 13 11 2021
Statut: ppublish

Résumé

To describe the occurrence of postprocedural atrial fibrillation (AF) among patients with cryptogenic stroke undergoing patent foramen ovale (PFO) closure in the REDUCE clinical study and analyze for potential risk factors for the development of postprocedural AF. AF is an adverse event that might potentially counterbalance the stroke prevention benefit from PFO closure. Data on AF after transcatheter PFO closure are sparse. We evaluated data from patients having PFO closure (Gore HELEX or Gore Cardioform Septal Occluder) in the REDUCE clinical trial (n = 408) in at post hoc explorative analysis. Median follow-up was 5.0 years. AF occurred in 30 patients (7.4%) after PFO closure with a total of 34 AF events. Most were reported as non-serious (68%), detected within 45 days post-procedure (79%), and resolved within 2 weeks of onset (63%). One subject with AF had recurrent stroke. Postprocedural AF occurred more frequently among subjects with higher age and large device sizes. Male sex was the only independent predictor of postprocedural AF. We found no association between the type of occluder (HELEX or Gore Cardioform Septal Occluder) or PFO anatomical characteristics and post-procedural AF. In the REDUCE clinical study, postprocedural atrial fibrillation was mostly early onset, transient and with no later recurrence. Postprocedural AF occurred more frequently among patients with higher age and larger devices. Male sex was the only independent predictor of postprocedural AF.

Sections du résumé

OBJECTIVES
To describe the occurrence of postprocedural atrial fibrillation (AF) among patients with cryptogenic stroke undergoing patent foramen ovale (PFO) closure in the REDUCE clinical study and analyze for potential risk factors for the development of postprocedural AF.
BACKGROUND
AF is an adverse event that might potentially counterbalance the stroke prevention benefit from PFO closure. Data on AF after transcatheter PFO closure are sparse.
METHODS
We evaluated data from patients having PFO closure (Gore HELEX or Gore Cardioform Septal Occluder) in the REDUCE clinical trial (n = 408) in at post hoc explorative analysis. Median follow-up was 5.0 years.
RESULTS
AF occurred in 30 patients (7.4%) after PFO closure with a total of 34 AF events. Most were reported as non-serious (68%), detected within 45 days post-procedure (79%), and resolved within 2 weeks of onset (63%). One subject with AF had recurrent stroke. Postprocedural AF occurred more frequently among subjects with higher age and large device sizes. Male sex was the only independent predictor of postprocedural AF. We found no association between the type of occluder (HELEX or Gore Cardioform Septal Occluder) or PFO anatomical characteristics and post-procedural AF.
CONCLUSION
In the REDUCE clinical study, postprocedural atrial fibrillation was mostly early onset, transient and with no later recurrence. Postprocedural AF occurred more frequently among patients with higher age and larger devices. Male sex was the only independent predictor of postprocedural AF.

Identifiants

pubmed: 34773685
doi: 10.1002/ccd.30019
doi:

Types de publication

Clinical Study Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1551-1557

Informations de copyright

© 2021 Wiley Periodicals LLC.

Références

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Auteurs

Asger Andersen (A)

Department of Cardiology, Aarhus University Hospital, Aarhus, Denmark.

Kristina Laut Matzen (KL)

Department of Cardiology, Aarhus University Hospital, Aarhus, Denmark.

Grethe Andersen (G)

Department of Neurology, Aarhus University Hospital, Aarhus, Denmark.

Magnus Settergren (M)

Department of Cardiology, Karolinska University Hospital, Stockholm, Sweden.

Christina Sjostrand (C)

Department of Neurology, Karolinska University Hospital, Stockholm, Sweden.

Helle K Iversen (HK)

Department of Neurology, Rigshospitalet, University of Copenhagen, Copenhagen, Denmark.

Risto O Roine (RO)

Department of Clinical Neurosciences, Turku University Hospital and University of Turku, Turku, Finland.

David Hildick-Smith (D)

Department of Cardiology, Sussex Cardiac Centre, Brighton & Sussex University Hospitals, Brighton, UK.

J David Spence (JD)

Stroke Prevention & Atherosclerosis Research Centre, Robarts Research Institute, Western University, London, Ontario, Canada.

John F Rhodes (JF)

Department of Cardiology, Medical University of South Carolina, Charleston, South Carolina, USA.

Scott E Kasner (SE)

Department of Neurology, University of Pennsylvania, Philadelphia, Pennsylvania, USA.

Lars Sondergaard (L)

Department of Cardiology, Rigshospitalet, University of Copenhagen, Copenhagen, Denmark.

Jens Erik Nielsen-Kudsk (JE)

Department of Cardiology, Aarhus University Hospital, Aarhus, Denmark.

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