Drug resistance in Candida albicans isolates and related changes in the structural domain of Mdr1 protein.


Journal

Journal of infection and public health
ISSN: 1876-035X
Titre abrégé: J Infect Public Health
Pays: England
ID NLM: 101487384

Informations de publication

Date de publication:
Dec 2021
Historique:
received: 12 10 2021
revised: 26 10 2021
accepted: 01 11 2021
pubmed: 20 11 2021
medline: 15 12 2021
entrez: 19 11 2021
Statut: ppublish

Résumé

The increasing azole drug resistance in fungal pathogens poses a pressing threat to global health care. The coexistence of drug-resistant Candida albicans with tuberculosis patients and the failure of several drugs to treat C. albicans infection extend hospital stay, economic burden, and death. The misuse or abuse of azole-derived antifungals, chronic use of TB drugs, different immune-suppressive drugs, and diseases like HIV, COVID-19, etc., have aggravated the situation. So it is vital to understand the molecular changes in drug-resistant genes to modify the treatment to design an alternative mechanism. C. albicans isolated from chronic tuberculosis patients were screened for antifungal sensitivity studies using disk diffusion assay. The multidrug-resistant C. albicans were further screened for molecular-level changes in drug resistance using MDR1 gene sequencing and compared with Gen bank data of similar species using the BLAST tool. The investigation proved that the isolated C. albicans from TB patients are significantly resistant to the action of six drugs. The molecular changes in MDR1 genes showed differences in seven nucleotide base pairs that interfered with the efflux pump.

Sections du résumé

BACKGROUND BACKGROUND
The increasing azole drug resistance in fungal pathogens poses a pressing threat to global health care. The coexistence of drug-resistant Candida albicans with tuberculosis patients and the failure of several drugs to treat C. albicans infection extend hospital stay, economic burden, and death. The misuse or abuse of azole-derived antifungals, chronic use of TB drugs, different immune-suppressive drugs, and diseases like HIV, COVID-19, etc., have aggravated the situation. So it is vital to understand the molecular changes in drug-resistant genes to modify the treatment to design an alternative mechanism.
METHOD METHODS
C. albicans isolated from chronic tuberculosis patients were screened for antifungal sensitivity studies using disk diffusion assay. The multidrug-resistant C. albicans were further screened for molecular-level changes in drug resistance using MDR1 gene sequencing and compared with Gen bank data of similar species using the BLAST tool.
RESULTS RESULTS
The investigation proved that the isolated C. albicans from TB patients are significantly resistant to the action of six drugs. The molecular changes in MDR1 genes showed differences in seven nucleotide base pairs that interfered with the efflux pump.

Identifiants

pubmed: 34794907
pii: S1876-0341(21)00363-4
doi: 10.1016/j.jiph.2021.11.002
pii:
doi:

Substances chimiques

ATP Binding Cassette Transporter, Subfamily B, Member 1 0
Antifungal Agents 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1848-1853

Informations de copyright

Copyright © 2021 The Authors. Published by Elsevier Ltd.. All rights reserved.

Auteurs

P Dhasarathan (P)

Department of Biotechnology, Prathyusha Engineering College, Thiruvallur 602 025, Tamil Nadu, India.

Mohamad S AlSalhi (MS)

Department of Physics and Astronomy, College of Science, King Saud University, P.O. Box-2455, Riyadh, 11451, Saudi Arabia. Electronic address: malsalhi@ksu.edu.sa.

Sandhanasamy Devanesan (S)

Department of Physics and Astronomy, College of Science, King Saud University, P.O. Box-2455, Riyadh, 11451, Saudi Arabia.

Jeeva Subbiah (J)

Department Center for Inflammation, Immunity & Infection, Institute for Biomedical Sciences, Georgia State University, Atlanta, GA 30303, USA.

A J A Ranjitsingh (AJA)

Department of Biotechnology, Prathyusha Engineering College, Thiruvallur 602 025, Tamil Nadu, India. Electronic address: ranjitspkc@gmail.com.

Mohammed Binsalah (M)

Department of Oral and Maxillofacial Surgery, College of Dentistry, King Saud University, Riyadh, 11451, Saudi Arabia.

Akram A Alfuraydi (AA)

Department of Botany and Microbiology, College of Science, King Saud University, P.O. Box-2455, Riyadh, 11451, Saudi Arabia.

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Classifications MeSH