Circulating Monocyte Subsets Are Associated With Extent of Myocardial Injury but Not With Type of Myocardial Infarction.
acute coronary syndrome
biomarker
chronic coronary syndrome
inflammation
monocytes
troponin
Journal
Frontiers in cardiovascular medicine
ISSN: 2297-055X
Titre abrégé: Front Cardiovasc Med
Pays: Switzerland
ID NLM: 101653388
Informations de publication
Date de publication:
2021
2021
Historique:
received:
15
07
2021
accepted:
14
10
2021
entrez:
19
11
2021
pubmed:
20
11
2021
medline:
20
11
2021
Statut:
epublish
Résumé
Inflammation is a hallmark of the period after a myocardial infarction (MI) that is either promoted or resolved by distinct subtypes of circulating inflammatory cells. The three main monocyte subpopulations play different roles inflammation. This study examined whether the type of MI (type 1 or type 2) or the extent of myocardial injury is associated with differences in monocyte subpopulations. For this purpose, peripheral whole blood from patients with a suspected MI was used for flow cytometric measurements of the monocyte subpopulations, and myocardial injury was classified by cardiac troponin levels in serum. In patients with acute coronary syndrome (
Identifiants
pubmed: 34796212
doi: 10.3389/fcvm.2021.741890
pmc: PMC8592918
doi:
Types de publication
Journal Article
Langues
eng
Pagination
741890Informations de copyright
Copyright © 2021 Askari, Lipps, Voss, Staubach, Grün, Klingenberg, von Jeinsen, Wolter, Kriechbaum, Dörr, Nef, Liebetrau, Hamm and Keller.
Déclaration de conflit d'intérêts
The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
Références
Hellenic J Cardiol. 2019 Sep - Oct;60(5):311-321
pubmed: 31055050
Atherosclerosis. 2020 Aug;307:39-51
pubmed: 32711213
Cardiovasc Res. 2019 Jun 1;115(7):1117-1130
pubmed: 30825305
Am J Med Sci. 2019 May;357(5):421-434
pubmed: 31010468
Sci Rep. 2019 Nov 8;9(1):16304
pubmed: 31704966
Circulation. 2010 Jun 8;121(22):2437-45
pubmed: 20530020
Thromb Haemost. 2004 Aug;92(2):419-24
pubmed: 15269840
Thromb Haemost. 2016 Sep 27;116(4):626-37
pubmed: 27412877
Hellenic J Cardiol. 2021 Mar-Apr;62(2):182-183
pubmed: 32464277
Atherosclerosis. 2018 Feb;269:245-251
pubmed: 29407600
Medicine (Baltimore). 2016 May;95(18):e3466
pubmed: 27149446
Circulation. 1998 Mar 3;97(8):765-72
pubmed: 9498540
Circulation. 2000 Sep 12;102(11):1216-20
pubmed: 10982533
Nat Rev Immunol. 2011 Oct 10;11(11):762-74
pubmed: 21984070
Eur Heart J Cardiovasc Imaging. 2016 Oct;17(10):1128-37
pubmed: 26508517
Scand J Immunol. 2015 Sep;82(3):163-73
pubmed: 25997925
J Am Coll Cardiol. 2018 Oct 30;72(18):2231-2264
pubmed: 30153967
Eur J Prev Cardiol. 2020 Dec;27(19):2166-2169
pubmed: 31423815
Heart. 2013 Aug;99(16):1154-6
pubmed: 23781110
Blood. 2010 Oct 21;116(16):e74-80
pubmed: 20628149
J Thromb Haemost. 2012 Jul;10(7):1231-41
pubmed: 22212813
Mediators Inflamm. 2019 Apr 4;2019:7434376
pubmed: 31089324
Circ Cardiovasc Genet. 2012 Feb 1;5(1):122-31
pubmed: 22238190
Eur Heart J. 2020 Nov 7;41(42):4092-4099
pubmed: 32860034
Eur Heart J. 2007 Oct;28(20):2525-38
pubmed: 17951287
J Am Coll Cardiol. 2018 Jan 16;71(2):173-176
pubmed: 29325641
Coron Artery Dis. 2013 Aug;24(5):404-11
pubmed: 23612365
J Am Coll Cardiol. 2020 Oct 6;76(14):1660-1670
pubmed: 33004131
Int J Cardiol. 2020 Apr 1;304:144-147
pubmed: 32008845