Dietary Iron Overload and
fatigue
hemochromatosis
iron overload
liver
mitochondria
mitochondrial respiration
reactive oxygen species
Journal
Antioxidants (Basel, Switzerland)
ISSN: 2076-3921
Titre abrégé: Antioxidants (Basel)
Pays: Switzerland
ID NLM: 101668981
Informations de publication
Date de publication:
16 Nov 2021
16 Nov 2021
Historique:
received:
30
09
2021
revised:
04
11
2021
accepted:
13
11
2021
entrez:
27
11
2021
pubmed:
28
11
2021
medline:
28
11
2021
Statut:
epublish
Résumé
Iron is an essential co-factor for many cellular metabolic processes, and mitochondria are main sites of utilization. Iron accumulation promotes production of reactive oxygen species (ROS) via the catalytic activity of iron species. Herein, we investigated the consequences of dietary and genetic iron overload on mitochondrial function. C57BL/6N wildtype and
Identifiants
pubmed: 34829689
pii: antiox10111818
doi: 10.3390/antiox10111818
pmc: PMC8615072
pii:
doi:
Types de publication
Journal Article
Langues
eng
Subventions
Organisme : FWF funded doctoral program HOROS
ID : W-1253
Organisme : transnational doctoral program BI-DOC
ID : D151660-018-017
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