Plasma ADAM-10 levels and functional outcome of acute primary basal ganglia hemorrhage.


Journal

Clinica chimica acta; international journal of clinical chemistry
ISSN: 1873-3492
Titre abrégé: Clin Chim Acta
Pays: Netherlands
ID NLM: 1302422

Informations de publication

Date de publication:
01 Jan 2022
Historique:
received: 28 10 2021
revised: 23 11 2021
accepted: 25 11 2021
pubmed: 2 12 2021
medline: 22 12 2021
entrez: 1 12 2021
Statut: ppublish

Résumé

The a-secretase A disintegrin and metalloprotease-10 (ADAM-10) may have deleterious effects in acute brain injury. This study was designed to discern if a relationship between plasma ADAM-10 levels and functional outcome exists in patients with intracerebral hemorrhage (ICH). A total of 109 patients with basal ganglia hemorrhage and 100 healthy controls were included. Their plasma ADAM-10 levels were gauged. Ninety-day prognosis was assessed and poor outcome was defined as death or major disability (modified Rankin Scale score of 3 or greater). Plasma ADAM-10 levels were substantially elevated in patients, as compared to controls. ADAM-10 levels were independently correlated with hematoma size and National Institutes of Health Stroke Scale (NIHSS) score. Plasma ADAM-10, NIHSS score and hematoma size emerged as the independent predictors for 90-day poor outcome. Under receiver operating characteristic curve, plasma ADAM-10 levels exhibited similar prognostic capability, as compared to hematoma size and NIHSS score; moreover, it significantly improved prognostic abilities of NIHSS and hematoma size. Rising plasma ADAM-10 levels are independently related to increasing severity and poor long-term functional outcome after hemorrhagic stroke, substantializing serum ADAM-10 as a useful prognostic biomarker of ICH.

Sections du résumé

BACKGROUND BACKGROUND
The a-secretase A disintegrin and metalloprotease-10 (ADAM-10) may have deleterious effects in acute brain injury. This study was designed to discern if a relationship between plasma ADAM-10 levels and functional outcome exists in patients with intracerebral hemorrhage (ICH).
METHODS METHODS
A total of 109 patients with basal ganglia hemorrhage and 100 healthy controls were included. Their plasma ADAM-10 levels were gauged. Ninety-day prognosis was assessed and poor outcome was defined as death or major disability (modified Rankin Scale score of 3 or greater).
RESULTS RESULTS
Plasma ADAM-10 levels were substantially elevated in patients, as compared to controls. ADAM-10 levels were independently correlated with hematoma size and National Institutes of Health Stroke Scale (NIHSS) score. Plasma ADAM-10, NIHSS score and hematoma size emerged as the independent predictors for 90-day poor outcome. Under receiver operating characteristic curve, plasma ADAM-10 levels exhibited similar prognostic capability, as compared to hematoma size and NIHSS score; moreover, it significantly improved prognostic abilities of NIHSS and hematoma size.
CONCLUSIONS CONCLUSIONS
Rising plasma ADAM-10 levels are independently related to increasing severity and poor long-term functional outcome after hemorrhagic stroke, substantializing serum ADAM-10 as a useful prognostic biomarker of ICH.

Identifiants

pubmed: 34852263
pii: S0009-8981(21)00416-2
doi: 10.1016/j.cca.2021.11.026
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

18-24

Informations de copyright

Copyright © 2021 Elsevier B.V. All rights reserved.

Auteurs

Ma-Jing Feng (MJ)

Department of Neurosurgery, The Changxing People's Hospital, The Second Affiliated Hospital of Zhejiang University School of Medicine Changxing Campus, 66 Taihu Middle Road, Changxing 313100, China. Electronic address: ongiangang@163.com.

Wei Wang (W)

Department of Neurosurgery, The Changxing People's Hospital, The Second Affiliated Hospital of Zhejiang University School of Medicine Changxing Campus, 66 Taihu Middle Road, Changxing 313100, China.

Xue-Feng Zhang (XF)

Emergency Intensive Care Unit, The Changxing People's Hospital, The Second Affiliated Hospital of Zhejiang University School of Medicine Changxing Campus, 66 Taihu Middle Road, Changxing 313100, China.

Fang-Fang Che (FF)

Department of Clinical Laboratory, The Changxing People's Hospital, The Second Affiliated Hospital of Zhejiang University School of Medicine Changxing Campus, 66 Taihu Middle Road, Changxing 313100, China.

Jie Yang (J)

Department of Pathology, The Changxing People's Hospital, The Second Affiliated Hospital of Zhejiang University School of Medicine Changxing Campus, 66 Taihu Middle Road, Changxing 313100, China.

Wei-Bin Ning (WB)

Department of Radiology, The Changxing People's Hospital, The Second Affiliated Hospital of Zhejiang University School of Medicine Changxing Campus, 66 Taihu Middle Road, Changxing 313100, China.

Wei Gao (W)

Department of Neurosurgery, The Changxing People's Hospital, The Second Affiliated Hospital of Zhejiang University School of Medicine Changxing Campus, 66 Taihu Middle Road, Changxing 313100, China.

Jiang Chen (J)

Department of Neurosurgery, The Changxing People's Hospital, The Second Affiliated Hospital of Zhejiang University School of Medicine Changxing Campus, 66 Taihu Middle Road, Changxing 313100, China.

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