PRDX-1 Supports the Survival and Antitumor Activity of Primary and CAR-Modified NK Cells under Oxidative Stress.
Journal
Cancer immunology research
ISSN: 2326-6074
Titre abrégé: Cancer Immunol Res
Pays: United States
ID NLM: 101614637
Informations de publication
Date de publication:
02 2022
02 2022
Historique:
received:
23
12
2020
revised:
15
09
2021
accepted:
30
11
2021
pubmed:
3
12
2021
medline:
30
4
2022
entrez:
2
12
2021
Statut:
ppublish
Résumé
Oxidative stress, caused by the imbalance between reactive species generation and the dysfunctional capacity of antioxidant defenses, is one of the characteristic features of cancer. Here, we quantified hydrogen peroxide in the tumor microenvironment (TME) and demonstrated that hydrogen peroxide concentrations are elevated in tumor interstitial fluid isolated from murine breast cancers
Identifiants
pubmed: 34853030
pii: 2326-6066.CIR-20-1023
doi: 10.1158/2326-6066.CIR-20-1023
pmc: PMC9414282
doi:
Substances chimiques
Antioxidants
0
Interleukin-15
0
Hydrogen Peroxide
BBX060AN9V
Peroxiredoxins
EC 1.11.1.15
Prdx1 protein, mouse
EC 1.11.1.15
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
228-244Informations de copyright
©2021 The Authors; Published by the American Association for Cancer Research.
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