Systems biology and machine learning approaches identify drug targets in diabetic nephropathy.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
06 12 2021
Historique:
received: 03 03 2021
accepted: 12 11 2021
entrez: 7 12 2021
pubmed: 8 12 2021
medline: 28 1 2022
Statut: epublish

Résumé

Diabetic nephropathy (DN), the leading cause of end-stage renal disease, has become a massive global health burden. Despite considerable efforts, the underlying mechanisms have not yet been comprehensively understood. In this study, a systematic approach was utilized to identify the microRNA signature in DN and to introduce novel drug targets (DTs) in DN. Using microarray profiling followed by qPCR confirmation, 13 and 6 differentially expressed (DE) microRNAs were identified in the kidney cortex and medulla, respectively. The microRNA-target interaction networks for each anatomical compartment were constructed and central nodes were identified. Moreover, enrichment analysis was performed to identify key signaling pathways. To develop a strategy for DT prediction, the human proteome was annotated with 65 biochemical characteristics and 23 network topology parameters. Furthermore, all proteins targeted by at least one FDA-approved drug were identified. Next, mGMDH-AFS, a high-performance machine learning algorithm capable of tolerating massive imbalanced size of the classes, was developed to classify DT and non-DT proteins. The sensitivity, specificity, accuracy, and precision of the proposed method were 90%, 86%, 88%, and 89%, respectively. Moreover, it significantly outperformed the state-of-the-art (P-value ≤ 0.05) and showed very good diagnostic accuracy and high agreement between predicted and observed class labels. The cortex and medulla networks were then analyzed with this validated machine to identify potential DTs. Among the high-rank DT candidates are Egfr, Prkce, clic5, Kit, and Agtr1a which is a current well-known target in DN. In conclusion, a combination of experimental and computational approaches was exploited to provide a holistic insight into the disorder for introducing novel therapeutic targets.

Identifiants

pubmed: 34873190
doi: 10.1038/s41598-021-02282-3
pii: 10.1038/s41598-021-02282-3
pmc: PMC8648918
doi:

Substances chimiques

MicroRNAs 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

23452

Informations de copyright

© 2021. The Author(s).

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Auteurs

Maryam Abedi (M)

Regenerative Medicine Research Center, Isfahan University of Medical Sciences, Isfahan, Iran.

Hamid Reza Marateb (HR)

Biomedical Engineering Department, Engineering Faculty, University of Isfahan, Isfahan, Iran.
Department of Automatic Control, Biomedical Engineering Research Center, Universitat Politècnica de Catalunya, BarcelonaTech (UPC), Barcelona, Spain.

Mohammad Reza Mohebian (MR)

Department of Electrical and Computer Engineering, University of Saskatchewan, Saskatoon, Canada.

Seyed Hamid Aghaee-Bakhtiari (SH)

Bioinformatics Research Group, Mashhad University of Medical Sciences, Mashhad, Iran.
Department of Medical Biotechnology and Nanotechnology, Faculty of Medicine, Mashhad University of Medical Sciences, Mashhad, Iran.

Seyed Mahdi Nassiri (SM)

Department of Clinical Pathology, Faculty of Veterinary Medicine, University of Tehran, Tehran, Iran.

Yousof Gheisari (Y)

Regenerative Medicine Research Center, Isfahan University of Medical Sciences, Isfahan, Iran. ygheisari@med.mui.ac.ir.
Department of Genetics and Molecular Biology, Isfahan University of Medical Sciences, Isfahan, Iran. ygheisari@med.mui.ac.ir.

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