Wnt2 Contributes to the Development of Atherosclerosis.

EndMT HAECs TGF-β Wnt atherosclerosis

Journal

Frontiers in cardiovascular medicine
ISSN: 2297-055X
Titre abrégé: Front Cardiovasc Med
Pays: Switzerland
ID NLM: 101653388

Informations de publication

Date de publication:
2021
Historique:
received: 02 08 2021
accepted: 21 10 2021
entrez: 13 12 2021
pubmed: 14 12 2021
medline: 14 12 2021
Statut: epublish

Résumé

Atherosclerosis, is a chronic inflammatory disease, characterized by the narrowing of the arteries resulting from the formation of intimal plaques in the wall of arteries. Yet the molecular mechanisms responsible for maintaining the development and progression of atherosclerotic lesions have not been fully defined. In this study, we show that TGF-β activates the endothelial-to-mesenchymal transition (EndMT) in cultured human aortic endothelial cells (HAECs) and this transition is dependent on the key executor of the Wnt signaling pathway

Identifiants

pubmed: 34901211
doi: 10.3389/fcvm.2021.751720
pmc: PMC8652052
doi:

Types de publication

Journal Article

Langues

eng

Pagination

751720

Informations de copyright

Copyright © 2021 Zhang, Rojas, Singh, Musich, Gutierrez, Yao, Thewke and Jiang.

Déclaration de conflit d'intérêts

The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.

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Auteurs

Jinyu Zhang (J)

Department of Biomedical Sciences, James H. Quillen College of Medicine, East Tennessee State University, Johnson City, TN, United States.
Division of Infectious, Inflammatory and Immunologic Diseases, Department of Internal Medicine, Quillen College of Medicine, East Tennessee State University, Johnson City, TN, United States.

Samuel Rojas (S)

Department of Biological Sciences, College of Arts and Sciences, East Tennessee State University, Johnson City, TN, United States.

Sanjay Singh (S)

Department of Biomedical Sciences, James H. Quillen College of Medicine, East Tennessee State University, Johnson City, TN, United States.

Phillip R Musich (PR)

Department of Biomedical Sciences, James H. Quillen College of Medicine, East Tennessee State University, Johnson City, TN, United States.

Matthew Gutierrez (M)

Department of Health Sciences, College of Public Health, East Tennessee State University, Johnson City, TN, United States.

Zhiqiang Yao (Z)

Division of Infectious, Inflammatory and Immunologic Diseases, Department of Internal Medicine, Quillen College of Medicine, East Tennessee State University, Johnson City, TN, United States.

Douglas Thewke (D)

Department of Biomedical Sciences, James H. Quillen College of Medicine, East Tennessee State University, Johnson City, TN, United States.

Yong Jiang (Y)

Department of Biomedical Sciences, James H. Quillen College of Medicine, East Tennessee State University, Johnson City, TN, United States.

Classifications MeSH