Neutralizing Antibody Response to Pseudotype Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) Differs Between mRNA-1273 and BNT162b2 Coronavirus Disease 2019 (COVID-19) Vaccines and by History of SARS-CoV-2 Infection.


Journal

Clinical infectious diseases : an official publication of the Infectious Diseases Society of America
ISSN: 1537-6591
Titre abrégé: Clin Infect Dis
Pays: United States
ID NLM: 9203213

Informations de publication

Date de publication:
24 08 2022
Historique:
received: 17 10 2021
pubmed: 21 12 2021
medline: 30 8 2022
entrez: 20 12 2021
Statut: ppublish

Résumé

Data on the development of neutralizing antibodies (nAbs) against SARS-CoV-2 after SARS-CoV-2 infection and after vaccination with mRNA COVID-19 vaccines are limited. From a prospective cohort of 3975 adult essential and frontline workers tested weekly from August 2020 to March 2021 for SARS-CoV-2 infection by reverse transcription-polymerase chain reaction assay irrespective of symptoms, 497 participants had sera drawn after infection (170), vaccination (327), and after both infection and vaccination (50 from the infection population). Serum was collected after infection and each vaccine dose. Serum-neutralizing antibody titers against USA-WA1/2020-spike pseudotype virus were determined by the 50% inhibitory dilution. Geometric mean titers (GMTs) and corresponding fold increases were calculated using t tests and linear mixed-effects models. Among 170 unvaccinated participants with SARS-CoV-2 infection, 158 (93%) developed nAbs with a GMT of 1003 (95% confidence interval, 766-1315). Among 139 previously uninfected participants, 138 (99%) developed nAbs after mRNA vaccine dose 2 with a GMT of 3257 (2596-4052). GMT was higher among those receiving mRNA-1273 vaccine (GMT, 4698; 3186-6926) compared with BNT162b2 vaccine (GMT, 2309; 1825-2919). Among 32 participants with prior SARS-CoV-2 infection, GMT was 21 655 (14 766-31 756) after mRNA vaccine dose 1, without further increase after dose 2. A single dose of mRNA vaccine after SARS-CoV-2 infection resulted in the highest observed nAb response. Two doses of mRNA vaccine in previously uninfected participants resulted in higher nAbs to SARS-CoV-2 than after 1 dose of vaccine or SARS-CoV-2 infection alone. nAb response also differed by mRNA vaccine product.

Sections du résumé

BACKGROUND
Data on the development of neutralizing antibodies (nAbs) against SARS-CoV-2 after SARS-CoV-2 infection and after vaccination with mRNA COVID-19 vaccines are limited.
METHODS
From a prospective cohort of 3975 adult essential and frontline workers tested weekly from August 2020 to March 2021 for SARS-CoV-2 infection by reverse transcription-polymerase chain reaction assay irrespective of symptoms, 497 participants had sera drawn after infection (170), vaccination (327), and after both infection and vaccination (50 from the infection population). Serum was collected after infection and each vaccine dose. Serum-neutralizing antibody titers against USA-WA1/2020-spike pseudotype virus were determined by the 50% inhibitory dilution. Geometric mean titers (GMTs) and corresponding fold increases were calculated using t tests and linear mixed-effects models.
RESULTS
Among 170 unvaccinated participants with SARS-CoV-2 infection, 158 (93%) developed nAbs with a GMT of 1003 (95% confidence interval, 766-1315). Among 139 previously uninfected participants, 138 (99%) developed nAbs after mRNA vaccine dose 2 with a GMT of 3257 (2596-4052). GMT was higher among those receiving mRNA-1273 vaccine (GMT, 4698; 3186-6926) compared with BNT162b2 vaccine (GMT, 2309; 1825-2919). Among 32 participants with prior SARS-CoV-2 infection, GMT was 21 655 (14 766-31 756) after mRNA vaccine dose 1, without further increase after dose 2.
CONCLUSIONS
A single dose of mRNA vaccine after SARS-CoV-2 infection resulted in the highest observed nAb response. Two doses of mRNA vaccine in previously uninfected participants resulted in higher nAbs to SARS-CoV-2 than after 1 dose of vaccine or SARS-CoV-2 infection alone. nAb response also differed by mRNA vaccine product.

Identifiants

pubmed: 34928334
pii: 6470720
doi: 10.1093/cid/ciab1038
pmc: PMC8755309
doi:

Substances chimiques

Antibodies, Neutralizing 0
Antibodies, Viral 0
COVID-19 Vaccines 0
Spike Glycoprotein, Coronavirus 0
Vaccines, Synthetic 0
mRNA Vaccines 0
spike protein, SARS-CoV-2 0
2019-nCoV Vaccine mRNA-1273 EPK39PL4R4
BNT162 Vaccine N38TVC63NU

Types de publication

Journal Article Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, P.H.S.

Langues

eng

Sous-ensembles de citation

IM

Pagination

e827-e837

Subventions

Organisme : NCATS NIH HHS
ID : UL1 TR002538
Pays : United States
Organisme : NCIRD CDC HHS
Pays : United States

Informations de copyright

© The Author(s) 2021. Published by Oxford University Press for the Infectious Diseases Society of America. All rights reserved. For permissions, e-mail: journals.permissions@oup.com.

Auteurs

Harmony L Tyner (HL)

St. Luke's Regional Health Care System, Duluth, Minnesota, USA.

Jefferey L Burgess (JL)

The Mel and Enid Zuckerman College of Public Health and the College of Medicine, University of Arizona, Tucson, Arizona, USA.

Lauren Grant (L)

Centers for Disease Control and Prevention COVID-19 Response Team, Atlanta, GeorgiaUSA.

Manjusha Gaglani (M)

Baylor Scott and White Health, Temple, Texas, USA.
Texas A&M University College of Medicine, Temple, Texas, USA.

Jennifer L Kuntz (JL)

Kaiser Permanente Northwest Center for Health Research, Portland, Oregon, USA.

Allison L Naleway (AL)

Kaiser Permanente Northwest Center for Health Research, Portland, Oregon, USA.

Natalie J Thornburg (NJ)

Centers for Disease Control and Prevention COVID-19 Response Team, Atlanta, GeorgiaUSA.

Alberto J Caban-Martinez (AJ)

the Leonard M. Miller School of Medicine, University of Miami, Miami, Florida, USA.

Sarang K Yoon (SK)

Department of Family and Preventive Medicine, University of Utah Health, Salt Lake City, Utah, USA.

Meghan K Herring (MK)

Abt Associates, Inc, Rockville, Maryland, USA.

Shawn C Beitel (SC)

The Mel and Enid Zuckerman College of Public Health and the College of Medicine, University of Arizona, Tucson, Arizona, USA.

Lenee Blanton (L)

Centers for Disease Control and Prevention COVID-19 Response Team, Atlanta, GeorgiaUSA.

Janko Nikolich-Zugich (J)

The Mel and Enid Zuckerman College of Public Health and the College of Medicine, University of Arizona, Tucson, Arizona, USA.

Matthew S Thiese (MS)

Department of Family and Preventive Medicine, University of Utah Health, Salt Lake City, Utah, USA.

Jessica Flores Pleasants (JF)

Abt Associates, Inc, Rockville, Maryland, USA.

Ashley L Fowlkes (AL)

Centers for Disease Control and Prevention COVID-19 Response Team, Atlanta, GeorgiaUSA.

Karen Lutrick (K)

The Mel and Enid Zuckerman College of Public Health and the College of Medicine, University of Arizona, Tucson, Arizona, USA.

Kayan Dunnigan (K)

Baylor Scott and White Health, Temple, Texas, USA.

Young M Yoo (YM)

Centers for Disease Control and Prevention COVID-19 Response Team, Atlanta, GeorgiaUSA.

Spencer Rose (S)

Baylor Scott and White Health, Temple, Texas, USA.

Holly Groom (H)

Kaiser Permanente Northwest Center for Health Research, Portland, Oregon, USA.

Jennifer Meece (J)

Marshfield Clinic Research Institute, Marshfield, Wisconsin, USA.

Meredith G Wesley (MG)

Abt Associates, Inc, Rockville, Maryland, USA.

Natasha Schaefer-Solle (N)

the Leonard M. Miller School of Medicine, University of Miami, Miami, Florida, USA.

Paola Louzado-Feliciano (P)

the Leonard M. Miller School of Medicine, University of Miami, Miami, Florida, USA.

Laura J Edwards (LJ)

Abt Associates, Inc, Rockville, Maryland, USA.

Lauren E W Olsho (LEW)

Abt Associates, Inc, Rockville, Maryland, USA.

Mark G Thompson (MG)

Centers for Disease Control and Prevention COVID-19 Response Team, Atlanta, GeorgiaUSA.

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