Randomized Phase 2 Trial of Abiraterone Acetate Plus Prednisone, Degarelix, or the Combination in Men with Biochemically Recurrent Prostate Cancer After Radical Prostatectomy.

Abiraterone Androgen Androgen deprivation therapy Biochemical recurrence Degarelix Prostate cancer Prostate-specific antigen

Journal

European urology open science
ISSN: 2666-1683
Titre abrégé: Eur Urol Open Sci
Pays: Netherlands
ID NLM: 101771568

Informations de publication

Date de publication:
Dec 2021
Historique:
accepted: 24 09 2021
entrez: 22 12 2021
pubmed: 23 12 2021
medline: 23 12 2021
Statut: epublish

Résumé

Phase 2 trial endpoints that can be utilized in high-risk biochemical recurrence (BCR) after prostatectomy as a way of more rapidly identifying treatments for phase 3 trials are urgently needed. The efficacy of abiraterone acetate plus prednisone (AAP) in BCR is unknown. To compare the rates of complete biochemical responses after testosterone recovery after 8 mo of AAP and degarelix, a gonadotropin-releasing hormone antagonist, alone or in combination. Patients with BCR (prostate-specific antigen [PSA] ≥1.0 ng/ml, PSA doubling time ≤9 mo, no metastases on standard imaging, and testosterone ≥150 ng/dl) after prostatectomy (with or without prior radiotherapy) were included in this study. Patients were randomized to AAP (arm 1), AAP with degarelix (arm 2), or degarelix (arm 3) for 8 mo, and monitored for 18 mo. The primary endpoint was undetectable PSA with testosterone >150 ng/dl at 18 mo. Secondary endpoints were undetectable PSA at 8 mo and time to testosterone recovery. For the 122 patients enrolled, no difference was found between treatments for the primary endpoint (arm 1: 5.1% [95% confidence interval {CI}: 1-17%], arm 2: 17.1% [95% CI: 7-32%], arm 3: 11.9% [95% CI: 4-26%]; arm 1 vs 2, Rates of undetectable PSA levels with testosterone recovery were similar between arms, suggesting that increased androgen suppression with AAP and androgen deprivation therapy (ADT) is unlikely to eradicate recurrent disease compared with ADT alone. We evaluated the use of abiraterone acetate plus prednisone (AAP) and androgen deprivation therapy (ADT), AAP alone, or ADT alone in men with biochemically recurrent, nonmetastatic prostate cancer. While more men who received the combination had an undetectable prostate-specific antigen (PSA) level at 8 mo on treatment, once men came off treatment and testosterone level rose, there was no difference in the rates of undetectable PSA levels. This suggests that the combination is not able to eradicate disease any better than ADT alone.

Sections du résumé

BACKGROUND BACKGROUND
Phase 2 trial endpoints that can be utilized in high-risk biochemical recurrence (BCR) after prostatectomy as a way of more rapidly identifying treatments for phase 3 trials are urgently needed. The efficacy of abiraterone acetate plus prednisone (AAP) in BCR is unknown.
OBJECTIVE OBJECTIVE
To compare the rates of complete biochemical responses after testosterone recovery after 8 mo of AAP and degarelix, a gonadotropin-releasing hormone antagonist, alone or in combination.
DESIGN SETTING AND PARTICIPANTS METHODS
Patients with BCR (prostate-specific antigen [PSA] ≥1.0 ng/ml, PSA doubling time ≤9 mo, no metastases on standard imaging, and testosterone ≥150 ng/dl) after prostatectomy (with or without prior radiotherapy) were included in this study.
INTERVENTION METHODS
Patients were randomized to AAP (arm 1), AAP with degarelix (arm 2), or degarelix (arm 3) for 8 mo, and monitored for 18 mo.
OUTCOME MEASUREMENTS AND STATISTICAL ANALYSIS METHODS
The primary endpoint was undetectable PSA with testosterone >150 ng/dl at 18 mo. Secondary endpoints were undetectable PSA at 8 mo and time to testosterone recovery.
RESULTS AND LIMITATIONS CONCLUSIONS
For the 122 patients enrolled, no difference was found between treatments for the primary endpoint (arm 1: 5.1% [95% confidence interval {CI}: 1-17%], arm 2: 17.1% [95% CI: 7-32%], arm 3: 11.9% [95% CI: 4-26%]; arm 1 vs 2,
CONCLUSIONS CONCLUSIONS
Rates of undetectable PSA levels with testosterone recovery were similar between arms, suggesting that increased androgen suppression with AAP and androgen deprivation therapy (ADT) is unlikely to eradicate recurrent disease compared with ADT alone.
PATIENT SUMMARY RESULTS
We evaluated the use of abiraterone acetate plus prednisone (AAP) and androgen deprivation therapy (ADT), AAP alone, or ADT alone in men with biochemically recurrent, nonmetastatic prostate cancer. While more men who received the combination had an undetectable prostate-specific antigen (PSA) level at 8 mo on treatment, once men came off treatment and testosterone level rose, there was no difference in the rates of undetectable PSA levels. This suggests that the combination is not able to eradicate disease any better than ADT alone.

Identifiants

pubmed: 34934969
doi: 10.1016/j.euros.2021.09.015
pii: S2666-1683(21)01696-7
pmc: PMC8655386
doi:

Types de publication

Journal Article

Langues

eng

Pagination

70-78

Subventions

Organisme : NCI NIH HHS
ID : P30 CA008748
Pays : United States

Informations de copyright

© 2021 The Authors.

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Auteurs

Karen A Autio (KA)

Genitourinary Oncology Service, Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, USA.
Weill Cornell Medical College, New York, NY, USA.

Emmanuel S Antonarakis (ES)

University of Minnesota Medical School, Minneapolis, MN, USA.

Tina M Mayer (TM)

Rutgers Cancer Institute of New Jersey, New Brunswick, NJ, USA.

Daniel H Shevrin (DH)

NorthShore University HealthSystem, Evanston, IL, USA.

Mark N Stein (MN)

Columbia University Herbert Irving Comprehensive Cancer Center, New York, NY, USA.

Ulka N Vaishampayan (UN)

University of Michigan, Ann Arbor, MI, USA.

Michael J Morris (MJ)

Genitourinary Oncology Service, Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, USA.
Weill Cornell Medical College, New York, NY, USA.

Susan F Slovin (SF)

Genitourinary Oncology Service, Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, USA.
Weill Cornell Medical College, New York, NY, USA.

Elisabeth I Heath (EI)

Karmanos Cancer Institute, Wayne State University, Detroit, MI, USA.

Scott T Tagawa (ST)

Weill Cornell Medical College, New York, NY, USA.

Dana E Rathkopf (DE)

Genitourinary Oncology Service, Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, USA.
Weill Cornell Medical College, New York, NY, USA.

Matthew I Milowsky (MI)

UNC Lineberger Comprehensive Cancer Center, University of North Carolina, Chapel Hill, NC, USA.

Michael R Harrison (MR)

Duke Cancer Institute Center for Prostate and Urologic Cancers, Duke University, Durham, NC, USA.

Tomasz M Beer (TM)

Knight Cancer Institute, Oregon Health & Science University, Portland, OR, USA.

Arjun V Balar (AV)

New York University, New York, NY, USA.

Andrew J Armstrong (AJ)

Duke Cancer Institute Center for Prostate and Urologic Cancers, Duke University, Durham, NC, USA.

Daniel J George (DJ)

Duke Cancer Institute Center for Prostate and Urologic Cancers, Duke University, Durham, NC, USA.

Channing J Paller (CJ)

Johns Hopkins Sidney Kimmel Comprehensive Cancer Center, Johns Hopkins Medicine, Baltimore, MD, USA.

Arlyn Apollo (A)

Genitourinary Oncology Service, Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, USA.

Daniel C Danila (DC)

Genitourinary Oncology Service, Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, USA.

Julie N Graff (JN)

Knight Cancer Institute, Oregon Health & Science University, Portland, OR, USA.

Luke Nordquist (L)

Urology Cancer Center and GU Research Network, Omaha, NE, USA.

Erica S Dayan Cohn (ES)

Genitourinary Oncology Service, Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, USA.

Kin Tse (K)

Columbia University, New York, NY, USA.

Nicole A Schreiber (NA)

Convergent Therapeutics, New York, NY, USA.

Glenn Heller (G)

Biostatistics Service, Department of Epidemiology and Biostatistics, Memorial Sloan Kettering Cancer Center, New York, NY, USA.

Howard I Scher (HI)

Genitourinary Oncology Service, Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, NY, USA.
Weill Cornell Medical College, New York, NY, USA.
Prostate Cancer Clinical Trials Consortium, New York, NY, USA.

Classifications MeSH