The role of Intestinal-Fatty Acid Binding Proteins and Chitinase-3-Like Protein 1 across the spectrum of dysglycemia.


Journal

Diabetes & metabolic syndrome
ISSN: 1878-0334
Titre abrégé: Diabetes Metab Syndr
Pays: Netherlands
ID NLM: 101462250

Informations de publication

Date de publication:
Jan 2022
Historique:
received: 03 09 2021
revised: 07 12 2021
accepted: 09 12 2021
pubmed: 24 12 2021
medline: 1 4 2022
entrez: 23 12 2021
Statut: ppublish

Résumé

Recent studies underlie the importance of intestinal permeability and chronic inflammation in the pathogenesis of T2DM. Our study compared the concentrations of FABP2 and YKL40 as markers of intestinal permeability and inflammation among normoglycemia, prediabetes and T2DM. We recruited 122 participants (45 normoglycemic, 26 prediabetes, and 51 T2DM) of whom we measured the fasting serum levels of FABP2 and YKL-40 using ELISA method. The levels of FABP2 were significantly higher in the T2DM group [2.890 (1.880-4.070)] in comparison to both prediabetes [2.025 (1.145-2.343), p = 0.0085] and normoglycemia group [1.72 (1.250-2.645), p = 0.011]. The levels of YKL-40 were also significantly higher in the T2DM group [68.70 (44.61-166.6)] in comparison to both prediabetes [28.85 (20.64-41.53), p < 0.0001] and normoglycemia group [28.64 (19.25-43.87), p < 0.001]. Our study observed that the levels of FABP2 and YKL-40 were highest in the T2DM group supporting the available evidences on the role of intestinal permeability disruption and chronic low-grade inflammation in the pathogenesis of T2DM.

Sections du résumé

BACKGROUND AND AIMS OBJECTIVE
Recent studies underlie the importance of intestinal permeability and chronic inflammation in the pathogenesis of T2DM. Our study compared the concentrations of FABP2 and YKL40 as markers of intestinal permeability and inflammation among normoglycemia, prediabetes and T2DM.
METHODS METHODS
We recruited 122 participants (45 normoglycemic, 26 prediabetes, and 51 T2DM) of whom we measured the fasting serum levels of FABP2 and YKL-40 using ELISA method.
RESULTS RESULTS
The levels of FABP2 were significantly higher in the T2DM group [2.890 (1.880-4.070)] in comparison to both prediabetes [2.025 (1.145-2.343), p = 0.0085] and normoglycemia group [1.72 (1.250-2.645), p = 0.011]. The levels of YKL-40 were also significantly higher in the T2DM group [68.70 (44.61-166.6)] in comparison to both prediabetes [28.85 (20.64-41.53), p < 0.0001] and normoglycemia group [28.64 (19.25-43.87), p < 0.001].
CONCLUSIONS CONCLUSIONS
Our study observed that the levels of FABP2 and YKL-40 were highest in the T2DM group supporting the available evidences on the role of intestinal permeability disruption and chronic low-grade inflammation in the pathogenesis of T2DM.

Identifiants

pubmed: 34942410
pii: S1871-4021(21)00386-6
doi: 10.1016/j.dsx.2021.102366
pii:
doi:

Substances chimiques

Biomarkers 0
CHI3L1 protein, human 0
Chitinase-3-Like Protein 1 0
FABP2 protein, human 0
Fatty Acid-Binding Proteins 0
Chitinases EC 3.2.1.14

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

102366

Informations de copyright

Copyright © 2021 Diabetes India. Published by Elsevier Ltd. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest The authors declare that they have no competing interests.

Auteurs

Imelda R Sianipar (IR)

Department of Medical Physiology, Faculty of Medicine, Universitas Indonesia, Indonesia. Electronic address: imelda.rosalyn@ui.ac.id.

Sestramita Sestramita (S)

Graduate Student of Master Program in Biomedical Science, Faculty of Medicine, Universitas Indonesia, Indonesia.

Tika Pradnjaparamita (T)

Metabolic, Cardiovascular and Aging Research Cluster, The Indonesian Medical Education and Research Institute, Faculty of Medicine, Universitas Indonesia, Jakarta, Indonesia.

Em Yunir (E)

Metabolic, Cardiovascular and Aging Research Cluster, The Indonesian Medical Education and Research Institute, Faculty of Medicine, Universitas Indonesia, Jakarta, Indonesia; Division of Endocrinology, Metabolism and Diabetes, Department of Internal Medicine, dr. Cipto Mangunkusumo National General Hospital/Faculty of Medicine Universitas Indonesia, Indonesia.

Dante S Harbuwono (DS)

Metabolic, Cardiovascular and Aging Research Cluster, The Indonesian Medical Education and Research Institute, Faculty of Medicine, Universitas Indonesia, Jakarta, Indonesia; Division of Endocrinology, Metabolism and Diabetes, Department of Internal Medicine, dr. Cipto Mangunkusumo National General Hospital/Faculty of Medicine Universitas Indonesia, Indonesia.

Pradana Soewondo (P)

Metabolic, Cardiovascular and Aging Research Cluster, The Indonesian Medical Education and Research Institute, Faculty of Medicine, Universitas Indonesia, Jakarta, Indonesia; Division of Endocrinology, Metabolism and Diabetes, Department of Internal Medicine, dr. Cipto Mangunkusumo National General Hospital/Faculty of Medicine Universitas Indonesia, Indonesia. Electronic address: pradana.soewondo@ui.ac.id.

Dicky L Tahapary (DL)

Metabolic, Cardiovascular and Aging Research Cluster, The Indonesian Medical Education and Research Institute, Faculty of Medicine, Universitas Indonesia, Jakarta, Indonesia; Division of Endocrinology, Metabolism and Diabetes, Department of Internal Medicine, dr. Cipto Mangunkusumo National General Hospital/Faculty of Medicine Universitas Indonesia, Indonesia. Electronic address: dicky.tahapary@ui.ac.id.

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Classifications MeSH