Interstitial lung diseases associated with mutations of poly(A)-specific ribonuclease: A multicentre retrospective study.

PARN idiopathic interstitial pneumonia idiopathic pulmonary fibrosis interstitial lung disease mutation poly(A)-specific ribonuclease telomere homeostasis variant

Journal

Respirology (Carlton, Vic.)
ISSN: 1440-1843
Titre abrégé: Respirology
Pays: Australia
ID NLM: 9616368

Informations de publication

Date de publication:
03 2022
Historique:
revised: 21 10 2021
received: 04 09 2021
accepted: 29 11 2021
pubmed: 5 1 2022
medline: 22 4 2022
entrez: 4 1 2022
Statut: ppublish

Résumé

Poly(A)-specific ribonuclease (PARN) mutations have been associated with familial pulmonary fibrosis. This study aims to describe the phenotype of patients with interstitial lung disease (ILD) and heterozygous PARN mutations. We performed a retrospective, observational, non-interventional study of patients with an ILD diagnosis and a pathogenic heterozygous PARN mutation followed up in a centre of the OrphaLung network. We included 31 patients (29 from 16 kindreds and two sporadic patients). The median age at ILD diagnosis was 59 years (range 54 to 63). In total, 23 (74%) patients had a smoking history and/or fibrogenic exposure. The pulmonary phenotypes were heterogenous, but the most frequent diagnosis was idiopathic pulmonary fibrosis (n = 12, 39%). Haematological abnormalities were identified in three patients and liver disease in two. In total, 21 patients received a specific treatment for ILD: steroids (n = 13), antifibrotic agents (n = 11), immunosuppressants (n = 5) and N-acetyl cysteine (n = 2). The median forced vital capacity decline for the whole sample was 256 ml/year (range -363 to -148). After a median follow-up of 32 months (range 18 to 66), 10 patients had died and six had undergone lung transplantation. The median transplantation-free survival was 54 months (95% CI 29 to ∞). Extra-pulmonary features were less frequent with PARN mutation than telomerase reverse transcriptase (TERT) or telomerase RNA component (TERC) mutation. IPF is common among individuals with PARN mutation, but other ILD subtypes may be observed.

Sections du résumé

BACKGROUND AND OBJECTIVE
Poly(A)-specific ribonuclease (PARN) mutations have been associated with familial pulmonary fibrosis. This study aims to describe the phenotype of patients with interstitial lung disease (ILD) and heterozygous PARN mutations.
METHODS
We performed a retrospective, observational, non-interventional study of patients with an ILD diagnosis and a pathogenic heterozygous PARN mutation followed up in a centre of the OrphaLung network.
RESULTS
We included 31 patients (29 from 16 kindreds and two sporadic patients). The median age at ILD diagnosis was 59 years (range 54 to 63). In total, 23 (74%) patients had a smoking history and/or fibrogenic exposure. The pulmonary phenotypes were heterogenous, but the most frequent diagnosis was idiopathic pulmonary fibrosis (n = 12, 39%). Haematological abnormalities were identified in three patients and liver disease in two. In total, 21 patients received a specific treatment for ILD: steroids (n = 13), antifibrotic agents (n = 11), immunosuppressants (n = 5) and N-acetyl cysteine (n = 2). The median forced vital capacity decline for the whole sample was 256 ml/year (range -363 to -148). After a median follow-up of 32 months (range 18 to 66), 10 patients had died and six had undergone lung transplantation. The median transplantation-free survival was 54 months (95% CI 29 to ∞). Extra-pulmonary features were less frequent with PARN mutation than telomerase reverse transcriptase (TERT) or telomerase RNA component (TERC) mutation.
CONCLUSION
IPF is common among individuals with PARN mutation, but other ILD subtypes may be observed.

Identifiants

pubmed: 34981600
doi: 10.1111/resp.14195
doi:

Substances chimiques

Exoribonucleases EC 3.1.-
poly(A)-specific ribonuclease EC 3.1.13.4

Types de publication

Journal Article Multicenter Study Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

226-235

Informations de copyright

© 2022 Asian Pacific Society of Respirology.

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Auteurs

Quentin Philippot (Q)

Service de Pneumologie A, Hôpital Bichat, APHP, Paris, France.

Caroline Kannengiesser (C)

INSERM, Unité 1152, Université de Paris, Paris, France.
Laboratoire de Génétique, Hôpital Bichat, APHP, Paris, France.

Marie Pierre Debray (MP)

INSERM, Unité 1152, Université de Paris, Paris, France.
Service de Radiologie, Hôpital Bichat, APHP, Paris, France.

Clément Gauvain (C)

Service d'Oncologie Thoracique, CHU de Lille, Lille, France.

Ibrahima Ba (I)

Laboratoire de Génétique, Hôpital Bichat, APHP, Paris, France.

Margherita Vieri (M)

Department of Hematology, Oncology, Hemostaseology, and Stem Cell Transplantation, Medical Faculty, RWTH Aachen University, Aachen, Germany.

Anne Gondouin (A)

Service de Pneumologie, CHU de Besançon, Besancon, France.

Jean-Marc Naccache (JM)

Service de Pneumologie, Hôpital Tenon, APHP, Paris, France.

Martine Reynaud-Gaubert (M)

Service de Pneumologie, Hôpital Nord, APHM, Marseille, France.

Yurdagul Uzunhan (Y)

Service de Pneumologie, Hôpital Avicenne, APHP, Bobigny, France.

Benjamin Bondue (B)

Service de Pneumologie, Hôpital Erasme, Brussels, Belgium.

Dominique Israël-Biet (D)

Service de Pneumologie, Hôpital Européen George Pompidou, APHP, Paris, France.

Philippe Dieudé (P)

INSERM, Unité 1152, Université de Paris, Paris, France.
Service de Rhumatologie, Hôpital Bichat, APHP, Paris, France.

Cécile Fourrage (C)

Service de Génétique Hôpital Necker Enfants Malades, APHP, Paris, France.
Plateforme de Bio-informatique, Institut Imagine, Université de Paris, Paris, France.

Elodie Lainey (E)

Laboratoire d'Hématologie Hôpital Robert Debré, APHP, Paris, France.

Effrosyne Manali (E)

2nd Pulmonary department, Attikon University Hospital, Athens, Greece.

Spyros Papiris (S)

2nd Pulmonary department, Attikon University Hospital, Athens, Greece.

Lidwine Wemeau (L)

Service de Pneumologie, CHRU de Lille, Lille, France.

Sandrine Hirschi (S)

Service de Pneumologie, CHU de Strasbourg, Strasbourg, France.

Hervé Mal (H)

INSERM, Unité 1152, Université de Paris, Paris, France.
Service de Pneumologie B, Hôpital Bichat, APHP, Paris, France.

Hilario Nunes (H)

Service de Pneumologie, Hôpital Avicenne, APHP, Bobigny, France.

Frédéric Schlemmer (F)

Unité de Pneumologie, Université Paris-Est Créteil, APHP, Hôpitaux Universitaires Henri Mondor, Créteil, France.

Elodie Blanchard (E)

Service de Pneumologie, CHU de Bordeaux, Bordeaux, France.

Fabian Beier (F)

Department of Hematology, Oncology, Hemostaseology, and Stem Cell Transplantation, Medical Faculty, RWTH Aachen University, Aachen, Germany.

Vincent Cottin (V)

Coordonnateur OrphaLung, Centre coordonnateur national de référence des maladies pulmonaires rares, Service de Pneumologie, Hôpital Louis Pradel, Université de Lyon, INRAE, member of Radico-ILD, Lyon, France.
RespiFil, ERN-LUNG, Lyon, France.

Bruno Crestani (B)

Service de Pneumologie A, Hôpital Bichat, APHP, Paris, France.
INSERM, Unité 1152, Université de Paris, Paris, France.

Raphaël Borie (R)

Service de Pneumologie A, Hôpital Bichat, APHP, Paris, France.
INSERM, Unité 1152, Université de Paris, Paris, France.

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