Bone marrow NG2


Journal

Nature cancer
ISSN: 2662-1347
Titre abrégé: Nat Cancer
Pays: England
ID NLM: 101761119

Informations de publication

Date de publication:
03 2021
Historique:
entrez: 7 1 2022
pubmed: 8 1 2022
medline: 8 1 2022
Statut: ppublish

Résumé

In the bone marrow (BM) microenvironment, where breast cancer (BC) disseminated tumour cells (DTCs) can remain dormant for decades, NG2

Identifiants

pubmed: 34993493
doi: 10.1038/s43018-021-00179-8
pmc: PMC8730384
mid: NIHMS1675122
pii: 10.1038/s43018-021-00179-8
doi:

Substances chimiques

Nestin 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Pagination

327-339

Subventions

Organisme : NCI NIH HHS
ID : R01 CA244780
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK112976
Pays : United States
Organisme : NCI NIH HHS
ID : R01 CA218024
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL069438
Pays : United States
Organisme : NCI NIH HHS
ID : R01 CA218578
Pays : United States
Organisme : NCI NIH HHS
ID : R01 CA216248
Pays : United States
Organisme : NCI NIH HHS
ID : R01 CA109182
Pays : United States
Organisme : NIDDK NIH HHS
ID : U01 DK116312
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL145064
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL126705
Pays : United States
Organisme : NCI NIH HHS
ID : K22 CA196750
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK056638
Pays : United States
Organisme : NCI NIH HHS
ID : P30 CA196521
Pays : United States

Références

Chaffer, C. L. & Weinberg, R. A. A perspective on cancer cell metastasis. Science 331, 1559–1564 (2011).
doi: 10.1126/science.1203543
Sherry, M. M., Greco, F. A., Johnson, D. H. & Hainsworth, J. D. Metastatic breast cancer confined to the skeletal system. An indolent disease. Am. J. Med. 81, 381–386 (1986).
doi: 10.1016/0002-9343(86)90286-X
Engel, J. et al. The process of metastasisation for breast cancer. Eur. J. Cancer 39, 1794–1806 (2003).
doi: 10.1016/S0959-8049(03)00422-2
Husemann, Y. et al. Systemic spread is an early step in breast cancer. Cancer Cell 13, 58–68 (2008).
doi: 10.1016/j.ccr.2007.12.003
Sanger, N. et al. Disseminated tumor cells in the bone marrow of patients with ductal carcinoma in situ. Int. J. Cancer https://doi.org/10.1002/ijc.25895 (2011).
Braun, S. et al. Cytokeratin-positive cells in the bone marrow and survival of patients with stage I, II, or III breast cancer. N. Engl. J. Med. https://doi.org/10.1056/NEJM200002243420801 (2000).
Chéry, L. et al. Characterization of single disseminated prostate cancer cells reveals tumor cell heterogeneity and identifies dormancy associated pathways. Oncotarget 5, 9939–9951 (2014).
doi: 10.18632/oncotarget.2480
Borgen, E. et al. NR2F1 stratifies dormant disseminated tumor cells in breast cancer patients. Breast Cancer Res. https://doi.org/10.1186/s13058-018-1049-0 (2018).
Naume, B. et al. Clinical outcome with correlation to disseminated tumor cell (DTC) status after DTC-guided secondary adjuvant treatment with docetaxel in early breast cancer. J. Clin. Oncol. 32, 3848–3857 (2014).
doi: 10.1200/JCO.2014.56.9327
Bragado, P. et al. TGF-β2 dictates disseminated tumour cell fate in target organs through TGF-β-RIII and p38α/β signalling. Nat. Cell Biol. 15, 1351–1361 (2013).
doi: 10.1038/ncb2861
Carlson, P. et al. Targeting the perivascular niche sensitizes disseminated tumour cells to chemotherapy. Nat. Cell Biol. https://doi.org/10.1038/s41556-018-0267-0 (2019).
Johnson, R. W. et al. Induction of LIFR confers a dormancy phenotype in breast cancer cells disseminated to the bone marrow. Nat. Cell Biol. 18, 1078–1089 (2016).
doi: 10.1038/ncb3408
Kobayashi, A. et al. Bone morphogenetic protein 7 in dormancy and metastasis of prostate cancer stem-like cells in bone. J. Exp. Med. 208, 2641–2655 (2011).
doi: 10.1084/jem.20110840
Gao, H. et al. The BMP inhibitor Coco reactivates breast cancer cells at lung metastatic sites. Cell 150, 764–779 (2012).
doi: 10.1016/j.cell.2012.06.035
Taichman, R. S. et al. GAS6 receptor status is associated with dormancy and bone metastatic tumor formation. PLoS ONE 8, e61873 (2013).
doi: 10.1371/journal.pone.0061873
Yumoto, K. et al. Axl is required for TGF-β2-induced dormancy of prostate cancer cells in the bone marrow. Sci. Rep. 6, 36520 (2016).
doi: 10.1038/srep36520
Jung, Y. et al. Endogenous GAS6 and Mer receptor signaling regulate prostate cancer stem cells in bone marrow. Oncotarget 7, 25698–25711 (2016).
doi: 10.18632/oncotarget.8365
Yue, X. et al. Leukemia inhibitory factor promotes EMT through STAT3- dependent miR-21 induction. Oncotarget https://doi.org/10.18632/oncotarget.6756 (2015).
Agarwal, P. et al. Mesenchymal niche-specific expression of Cxcl12 controls quiescence of treatment-resistant leukemia stem cells. Cell Stem Cell 24, 769–784 (2019).
doi: 10.1016/j.stem.2019.02.018
Ghajar, C. M. et al. The perivascular niche regulates breast tumour dormancy. Nat. Cell Biol. 15, 807–817 (2013).
doi: 10.1038/ncb2767
Lawson, M. A. et al. Osteoclasts control reactivation of dormant myeloma cells by remodelling the endosteal niche. Nat. Commun. https://doi.org/10.1038/ncomms9983 (2015).
Yu-Lee, L. Y. et al. Osteoblast-secreted factors mediate dormancy of metastatic prostate cancer in the bone via activation of the TGFbRIII–p38MAPK–pS249/ T252RB pathway. Cancer Res. 78, 2911–2924 (2018).
doi: 10.1158/0008-5472.CAN-17-1051
Cackowski, F. C. & Taichman, R. S. Parallels between hematopoietic stem cell and prostate cancer disseminated tumor cell regulation. Bone. 119, 82–86 (2019).
doi: 10.1016/j.bone.2018.02.025
Shiozawa, Y. et al. Human prostate cancer metastases target the hematopoietic stem cell niche to establish footholds in mouse bone marrow. J. Clin. Invest. 121, 1298–1312 (2011).
doi: 10.1172/JCI43414
Wilson, A. et al. Hematopoietic stem cells reversibly switch from dormancy to self-renewal during homeostasis and repair. Cell 135, 1118–1129 (2008).
doi: 10.1016/j.cell.2008.10.048
Pinho, S. & Frenette, P. S. Haematopoietic stem cell activity and interactions with the niche. Nat. Rev. Mol. Cell Biol. 20, 303–320 (2019).
doi: 10.1038/s41580-019-0103-9
Kunisaki, Y. et al. Arteriolar niches maintain haematopoietic stem cell quiescence. Nature 502, 637–643 (2013).
doi: 10.1038/nature12612
Hanoun, M. et al. Acute myelogenous leukemia-induced sympathetic neuropathy promotes malignancy in an altered hematopoietic stem cell Niche. Cell Stem Cell (2014) https://doi.org/10.1016/j.stem.2014.06.020
Maryanovich, M. et al. Adrenergic nerve degeneration in bone marrow drives aging of the hematopoietic stem cell niche. Nat. Med. https://doi.org/10.1038/s41591-018-0030-x (2018).
Harper, K. L. et al. Mechanism of early dissemination and metastasis in Her2
Pinho, S. et al. PDGFR and CD51 mark human nestin
doi: 10.1084/jem.20122252
Ranganathan, A. C., Adam, A. P., Zhang, L. & Aguirre-Ghiso, J. A. Tumor cell dormancy induced by p38SAPK and ER-stress signaling: An adaptive advantage for metastatic cells? Cancer Biol. Ther. https://doi.org/10.4161/cbt.5.7.2968 (2006).
Fluegen, G. et al. Phenotypic heterogeneity of disseminated tumour cells is preset by primary tumour hypoxic microenvironments. Nat. Cell Biol. 19, 120–132 (2017).
doi: 10.1038/ncb3465
Sosa, M. S. et al. NR2F1 controls tumour cell dormancy via SOX9- and RARβ-driven quiescence programmes. Nat. Commun. 6, 1–14 (2015).
doi: 10.1038/ncomms7170
Männ, L. et al. CD11c.DTR mice develop a fatal fulminant myocarditis after local or systemic treatment with diphtheria toxin. Eur. J. Immunol. https://doi.org/10.1002/eji.201546245 (2016).
Christiaansen, A. F., Boggiatto, P. M. & Varga, S. M. Limitations of Foxp3
Bennett, C. L. et al. Inducible ablation of mouse Langerhans cells diminishes but fails to abrogate contact hypersensitivity. J. Cell Biol. https://doi.org/10.1083/jcb.200501071 (2005).
Xu, C. et al. Stem cell factor is selectively secreted by arterial endothelial cells in bone marrow. Nat. Commun. https://doi.org/10.1038/s41467-018-04726-3 (2018).
Tikhonova, A. N. et al. The bone marrow microenvironment at single-cell resolution. Nature https://doi.org/10.1038/s41586-019-1104-8 (2019).
Vukicevic, S., Latin, V., Chen, P., Batorsky, R. & Reddi, A. H. Localization of osteogenic protein-1 (bone morphogenetic protein-7) during human embryonic development: High affinity binding to basement membranes. Biochem. Biophys. Res. Commun. 198, 693–700 (1994).
doi: 10.1006/bbrc.1994.1100
Gregory, K. E. et al. The prodomain of BMP-7 targets the BMP-7 complex to the extracellular matrix. J. Biol. Chem. 280, 27970–27980 (2005).
doi: 10.1074/jbc.M504270200
Nakahara, F. et al. Engineering a haematopoietic stem cell niche by revitalizing mesenchymal stromal cells. Nat. Cell Biol. https://doi.org/10.1038/s41556-019-0308-3 (2019).
Aguirre-Ghiso, J. A., Ossowski, L. & Rosenbaum, S. K. Green fluorescent protein tagging of extracellular signal-regulated kinase and p38 pathways reveals novel dynamics of pathway activation during primary and metastatic growth. Cancer Res. 64, 7336–7345 (2004).
doi: 10.1158/0008-5472.CAN-04-0113
Regot, S., Hughey, J. J., Bajar, B. T., Carrasco, S. & Covert, M. W. High-sensitivity measurements of multiple kinase activities in live single cells. Cell 157, 1724–1734 (2014).
doi: 10.1016/j.cell.2014.04.039
Oki, T. et al. A novel cell-cycle-indicator, mVenus-p27K. Sci. Rep. 4, 4012 (2014).
doi: 10.1038/srep04012
Asada, N. et al. Differential cytokine contributions of perivascular haematopoietic stem cell niches. Nat. Cell Biol. https://doi.org/10.1038/ncb3475 (2017).
Zhou, B. O., Yue, R., Murphy, M. M., Peyer, J. G. & Morrison, S. J. Leptin-receptor-expressing mesenchymal stromal cells represent the main source of bone formed by adult bone marrow. Cell Stem Cell https://doi.org/10.1016/j.stem.2014.06.008 (2014).
Paget, S. The distribution of secondary growths in cancer of the breast. Lancet https://doi.org/10.1016/S0140-6736(00)49915-0 (1889).
Yang, Y. et al. Immunocompetent mouse allograft models for development of therapies to target breast cancer metastasis. Oncotarget 8, 30621–30643 (2017).
doi: 10.18632/oncotarget.15695
Boudreau, N. & Bissell, M. J. Extracellular matrix signaling: Integration of form and function in normal and malignant cells. Curr. Opin. Cell Biol. 10, 640–646 (1998).
doi: 10.1016/S0955-0674(98)80040-9
Kenny, P. A. & Bissell, M. J. Tumor reversion: Correction of malignant behavior by microenvironmental cues. Int. J. Cancer 107, 688–695 (2003).
doi: 10.1002/ijc.11491
Turley, R. S. et al. The type III transforming growth factor-β receptor as a novel tumor suppressor gene in prostate cancer. Cancer Res. 67, 1090–1098 (2007).
doi: 10.1158/0008-5472.CAN-06-3117
Ajiboye, S., Sissung, T. M., Sharifi, N. & Fig, W. D. More than an accessory: Implications of type III transforming growth factor-β receptor loss in prostate cancer. BJU Int. 105, 913–916 (2010).
doi: 10.1111/j.1464-410X.2009.08999.x
Dong, M. et al. The type III TGF-β receptor suppresses breast cancer progression. J. Clin. Invest. 117, 206–217 (2007).
doi: 10.1172/JCI29293
Kim, I. Y. et al. Loss of expression of bone morphogenetic protein receptor type II in human prostate cancer cells. Oncogene 23, 7651–7659 (2004).
doi: 10.1038/sj.onc.1207924
Singh, A. et al. Angiocrine signals regulate quiescence and therapy resistance in bone metastasis. JCI Insight https://doi.org/10.1172/jci.insight.125679 (2019).
Mignone, J. L., Kukekov, V., Chiang, A. S., Steindler, D. & Enikolopov, G. Neural stem and progenitor cells in nestin-GFP transgenic mice. J. Comp. Neurol. 469, 311–324 (2004).
doi: 10.1002/cne.10964
Ishtiaq Ahmed, A. S., Bose, G. C., Huang, L. & Azhar, M. Generation of mice carrying a knockout-first and conditional-ready allele of transforming growth factor β2 gene. Genesis 52, 817–826 (2014).
doi: 10.1002/dvg.22795
Ossowski, L. & Reich, E. Changes in malignant phenotype of a human carcinoma conditioned by growth environment. Cell 33, 323–333 (1983).
doi: 10.1016/0092-8674(83)90414-2

Auteurs

Ana Rita Nobre (AR)

Division of Hematology and Oncology, Department of Medicine and Department of Otolaryngology, Department of Oncological Sciences, Black Family Stem Cell Institute, The Tisch Cancer Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Abel Salazar Biomedical Sciences Institute, University of Porto, Porto, Portugal.

Emma Risson (E)

Division of Hematology and Oncology, Department of Medicine and Department of Otolaryngology, Department of Oncological Sciences, Black Family Stem Cell Institute, The Tisch Cancer Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.
Université de Lyon, Lyon, France.

Deepak K Singh (DK)

Division of Hematology and Oncology, Department of Medicine and Department of Otolaryngology, Department of Oncological Sciences, Black Family Stem Cell Institute, The Tisch Cancer Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.

Julie S Di Martino (JS)

Division of Hematology and Oncology, Department of Medicine, The Tisch Cancer Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.

Julie F Cheung (JF)

Division of Hematology and Oncology, Department of Medicine and Department of Otolaryngology, Department of Oncological Sciences, Black Family Stem Cell Institute, The Tisch Cancer Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.

Jiapeng Wang (J)

Department of Cell Biology and Anatomy, University of South Carolina School of Medicine, Columbia, SC, USA.

John Johnson (J)

Department of Cell Biology and Anatomy, University of South Carolina School of Medicine, Columbia, SC, USA.

Hege G Russnes (HG)

Division of Surgery and Cancer Medicine, Department of Oncology, Oslo University Hospital, Oslo, Norway.
Division of Laboratory Medicine, Department of Pathology, Oslo University Hospital, Oslo, Norway.

Jose Javier Bravo-Cordero (JJ)

Division of Hematology and Oncology, Department of Medicine, The Tisch Cancer Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA.

Alexander Birbrair (A)

Ruth L. and David S. Gottesman Institute for Stem Cell and Regenerative Medicine Research, Albert Einstein College of Medicine, Bronx, NY, USA.
Department of Pathology, Federal University of Minas Gerais, Belo Horizonte, Brazil.

Bjorn Naume (B)

Division of Cancer Medicine, Department of Oncology, Oslo University Hospital, Oslo, Norway.
Institute of Clinical Medicine, University of Oslo, Oslo, Norway.

Mohamad Azhar (M)

Department of Cell Biology and Anatomy, University of South Carolina School of Medicine, Columbia, SC, USA.

Paul S Frenette (PS)

Ruth L. and David S. Gottesman Institute for Stem Cell and Regenerative Medicine Research, Albert Einstein College of Medicine, Bronx, NY, USA.

Julio A Aguirre-Ghiso (JA)

Division of Hematology and Oncology, Department of Medicine and Department of Otolaryngology, Department of Oncological Sciences, Black Family Stem Cell Institute, The Tisch Cancer Institute, Icahn School of Medicine at Mount Sinai, New York, NY, USA. julio.aguirre-ghiso@mssm.edu.

Articles similaires

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male

Classifications MeSH