Thoracoscopic infrared ablation to create a box lesion as a treatment for atrial fibrillation.

Ablation Atrial fibrillation Box lesion Epicardial ablation Epicardial maze procedure Ex-maze procedure Infrared Infrared coagulator Left atrial appendage amputation Thoracoscopic surgery

Journal

Journal of cardiothoracic surgery
ISSN: 1749-8090
Titre abrégé: J Cardiothorac Surg
Pays: England
ID NLM: 101265113

Informations de publication

Date de publication:
08 Jan 2022
Historique:
received: 18 01 2021
accepted: 29 12 2021
entrez: 8 1 2022
pubmed: 9 1 2022
medline: 12 1 2022
Statut: epublish

Résumé

Creating a box lesion in the posterior wall of the left atrium from the epicardial side of the beating heart remains a challenge. Although a transmural lesion can be created by applying radiofrequency (RF) energy at clampable sites, it is still difficult to create a transmural lesion at unclampable sites because the inner blood flow in the unclampable free wall weakens the thermal effect on the outside. Our aim was to apply the newly developed infrared coagulator to create linear transmural lesions on the beating heart thoracoscopically to treat atrial fibrillation (AF). A 71-year-old male was referred to our hospital with a diagnosis of hypertrophic cardiomyopathy and permanent atrial fibrillation. The patient was first diagnosed with atrial fibrillation 20 years before. Direct current cardioversion had been performed every few years a total of four times, but sinus rhythm restoration had always been temporary. On February 27, 2020, thoracoscopic PV isolation together with infrared roof- and bottom-line ablation to create a box lesion and left atrial appendage amputation (LAAA) were performed. The coagulator could be applied to clinical thoracoscopic surgery to successfully create a box lesion without any complication. The patient restored a regular sinus rhythm, it has been maintained for eleven months, and there have been no adverse events. The infrared coagulator might have enough potential to create transmural lesions on the beating heart in thoracoscopic AF surgery.

Sections du résumé

BACKGROUND BACKGROUND
Creating a box lesion in the posterior wall of the left atrium from the epicardial side of the beating heart remains a challenge. Although a transmural lesion can be created by applying radiofrequency (RF) energy at clampable sites, it is still difficult to create a transmural lesion at unclampable sites because the inner blood flow in the unclampable free wall weakens the thermal effect on the outside. Our aim was to apply the newly developed infrared coagulator to create linear transmural lesions on the beating heart thoracoscopically to treat atrial fibrillation (AF).
CASE PRESENTATION METHODS
A 71-year-old male was referred to our hospital with a diagnosis of hypertrophic cardiomyopathy and permanent atrial fibrillation. The patient was first diagnosed with atrial fibrillation 20 years before. Direct current cardioversion had been performed every few years a total of four times, but sinus rhythm restoration had always been temporary. On February 27, 2020, thoracoscopic PV isolation together with infrared roof- and bottom-line ablation to create a box lesion and left atrial appendage amputation (LAAA) were performed. The coagulator could be applied to clinical thoracoscopic surgery to successfully create a box lesion without any complication. The patient restored a regular sinus rhythm, it has been maintained for eleven months, and there have been no adverse events.
CONCLUSIONS CONCLUSIONS
The infrared coagulator might have enough potential to create transmural lesions on the beating heart in thoracoscopic AF surgery.

Identifiants

pubmed: 34996500
doi: 10.1186/s13019-021-01750-1
pii: 10.1186/s13019-021-01750-1
pmc: PMC8742376
doi:

Types de publication

Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1

Informations de copyright

© 2022. The Author(s).

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Auteurs

Hiroshi Kubota (H)

Department of Cardiovascular Surgery, Kyorin University, 6-20-2, Shinkawa,Tokyo, Mitaka, 181-8611, Japan. kub@ks.kyorin-u.ac.jp.

Toshiya Ohtsuka (T)

Department of Cardiac Surgery, New Heart Watanabe Institute, Tokyo, Japan.

Mikio Ninomiya (M)

Department of Cardiovascular Surgery, Tokyo Metropolitan Tama Medical Center, Tokyo, Japan.

Takahiro Nonaka (T)

Department of Cardiovascular Surgery, Tokyo Metropolitan Tama Medical Center, Tokyo, Japan.

Motoyuki Hisagi (M)

Department of Cardiovascular Surgery, Tokyo Metropolitan Tama Medical Center, Tokyo, Japan.

Hidehito Endo (H)

Department of Cardiovascular Surgery, Kyorin University, 6-20-2, Shinkawa,Tokyo, Mitaka, 181-8611, Japan.

Sachito Minegishi (S)

Department of Cardiovascular Surgery, Kyorin University, 6-20-2, Shinkawa,Tokyo, Mitaka, 181-8611, Japan.

Hiroshi Tsuchiya (H)

Department of Cardiovascular Surgery, Kyorin University, 6-20-2, Shinkawa,Tokyo, Mitaka, 181-8611, Japan.

Yusuke Inaba (Y)

Department of Cardiovascular Surgery, Kyorin University, 6-20-2, Shinkawa,Tokyo, Mitaka, 181-8611, Japan.

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