Genetic heterogeneity and subtypes of major depression.


Journal

Molecular psychiatry
ISSN: 1476-5578
Titre abrégé: Mol Psychiatry
Pays: England
ID NLM: 9607835

Informations de publication

Date de publication:
03 2022
Historique:
received: 31 03 2021
accepted: 26 11 2021
revised: 25 11 2021
pubmed: 9 1 2022
medline: 18 5 2022
entrez: 8 1 2022
Statut: ppublish

Résumé

Major depression (MD) is a heterogeneous disorder; however, the extent to which genetic factors distinguish MD patient subgroups (genetic heterogeneity) remains uncertain. This study sought evidence for genetic heterogeneity in MD. Using UK Biobank cohort, the authors defined 16 MD subtypes within eight comparison groups (vegetative symptoms, symptom severity, comorbid anxiety disorder, age at onset, recurrence, suicidality, impairment, and postpartum depression; N ~ 3000-47000). To compare genetic component of these subtypes, subtype-specific genome-wide association studies were performed to estimate SNP-heritability, and genetic correlations within subtype comparison and with other related disorders/traits. The findings indicated that MD subtypes were divergent in their SNP-heritability, and genetic correlations both within subtype comparisons and with other related disorders/traits. Three subtype comparisons (vegetative symptoms, age at onset, and impairment) showed significant differences in SNP-heritability; while genetic correlations within subtype comparisons ranged from 0.55 to 0.86, suggesting genetic profiles are only partially shared among MD subtypes. Furthermore, subtypes that are more clinically challenging, e.g., early-onset, recurrent, suicidal, more severely impaired, had stronger genetic correlations with other psychiatric disorders. MD with atypical-like features showed a positive genetic correlation (+0.40) with BMI while a negative correlation (-0.09) was found in those without atypical-like features. Novel genomic loci with subtype-specific effects were identified. These results provide the most comprehensive evidence to date for genetic heterogeneity within MD, and suggest that the phenotypic complexity of MD can be effectively reduced by studying the subtypes which share partially distinct etiologies.

Identifiants

pubmed: 34997191
doi: 10.1038/s41380-021-01413-6
pii: 10.1038/s41380-021-01413-6
pmc: PMC9106834
mid: NIHMS1771796
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

1667-1675

Subventions

Organisme : Medical Research Council
ID : MC_PC_17228
Pays : United Kingdom
Organisme : NIMH NIH HHS
ID : R01 MH077139
Pays : United States
Organisme : NIMH NIH HHS
ID : R01 MH123724
Pays : United States
Organisme : NIMH NIH HHS
ID : R01 MH124871
Pays : United States
Organisme : NIMH NIH HHS
ID : U01 MH109528
Pays : United States
Organisme : Medical Research Council
ID : MC_QA137853
Pays : United Kingdom

Informations de copyright

© 2021. The Author(s), under exclusive licence to Springer Nature Limited.

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Auteurs

Thuy-Dung Nguyen (TD)

Department of Medical Epidemiology and Biostatistics, Karolinska Institutet, Stockholm, Sweden.
Department of Global Public Health, Karolinska Institutet, Stockholm, Sweden.

Arvid Harder (A)

Department of Medical Epidemiology and Biostatistics, Karolinska Institutet, Stockholm, Sweden.

Ying Xiong (Y)

Department of Medical Epidemiology and Biostatistics, Karolinska Institutet, Stockholm, Sweden.

Kaarina Kowalec (K)

Department of Medical Epidemiology and Biostatistics, Karolinska Institutet, Stockholm, Sweden.
College of Pharmacy, University of Manitoba, Winnipeg, Canada.

Sara Hägg (S)

Department of Medical Epidemiology and Biostatistics, Karolinska Institutet, Stockholm, Sweden.

Na Cai (N)

Helmholtz Pioneer Campus, Helmholtz Zentrum München, Neuherberg, Germany.

Ralf Kuja-Halkola (R)

Department of Medical Epidemiology and Biostatistics, Karolinska Institutet, Stockholm, Sweden.

Christina Dalman (C)

Department of Global Public Health, Karolinska Institutet, Stockholm, Sweden.

Patrick F Sullivan (PF)

Department of Medical Epidemiology and Biostatistics, Karolinska Institutet, Stockholm, Sweden.
Department of Genetics and Psychiatry, University of North Carolina, Chapel Hill, NC, USA.

Yi Lu (Y)

Department of Medical Epidemiology and Biostatistics, Karolinska Institutet, Stockholm, Sweden. lu.yi@ki.se.
Department of Global Public Health, Karolinska Institutet, Stockholm, Sweden. lu.yi@ki.se.

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