Identifying risk of adverse outcomes in COVID-19 patients via artificial intelligence-powered analysis of 12-lead intake electrocardiogram.

12-lead ECG Arrhythmia Artificial intelligence COVID-19 Deep learning Heart failure prognosis Mortality Risk factors

Journal

Cardiovascular digital health journal
ISSN: 2666-6936
Titre abrégé: Cardiovasc Digit Health J
Pays: United States
ID NLM: 101771268

Informations de publication

Date de publication:
Apr 2022
Historique:
pubmed: 11 1 2022
medline: 11 1 2022
entrez: 10 1 2022
Statut: ppublish

Résumé

Adverse events in COVID-19 are difficult to predict. Risk stratification is encumbered by the need to protect healthcare workers. We hypothesize that artificial intelligence (AI) can help identify subtle signs of myocardial involvement in the 12-lead electrocardiogram (ECG), which could help predict complications. Use intake ECGs from COVID-19 patients to train AI models to predict risk of mortality or major adverse cardiovascular events (MACE). We studied intake ECGs from 1448 COVID-19 patients (60.5% male, aged 63.4 ± 16.9 years). Records were labeled by mortality (death vs discharge) or MACE (no events vs arrhythmic, heart failure [HF], or thromboembolic [TE] events), then used to train AI models; these were compared to conventional regression models developed using demographic and comorbidity data. A total of 245 (17.7%) patients died (67.3% male, aged 74.5 ± 14.4 years); 352 (24.4%) experienced at least 1 MACE (119 arrhythmic, 107 HF, 130 TE). AI models predicted mortality and MACE with area under the curve (AUC) values of 0.60 ± 0.05 and 0.55 ± 0.07, respectively; these were comparable to AUC values for conventional models (0.73 ± 0.07 and 0.65 ± 0.10). There were no prominent temporal trends in mortality rate or MACE incidence in our cohort; holdout testing with data from after a cutoff date (June 9, 2020) did not degrade model performance. Using intake ECGs alone, our AI models had limited ability to predict hospitalized COVID-19 patients' risk of mortality or MACE. Our models' accuracy was comparable to that of conventional models built using more in-depth information, but translation to clinical use would require higher sensitivity and positive predictive value. In the future, we hope that mixed-input AI models utilizing both ECG and clinical data may be developed to enhance predictive accuracy.

Sections du résumé

BACKGROUND BACKGROUND
Adverse events in COVID-19 are difficult to predict. Risk stratification is encumbered by the need to protect healthcare workers. We hypothesize that artificial intelligence (AI) can help identify subtle signs of myocardial involvement in the 12-lead electrocardiogram (ECG), which could help predict complications.
OBJECTIVE OBJECTIVE
Use intake ECGs from COVID-19 patients to train AI models to predict risk of mortality or major adverse cardiovascular events (MACE).
METHODS METHODS
We studied intake ECGs from 1448 COVID-19 patients (60.5% male, aged 63.4 ± 16.9 years). Records were labeled by mortality (death vs discharge) or MACE (no events vs arrhythmic, heart failure [HF], or thromboembolic [TE] events), then used to train AI models; these were compared to conventional regression models developed using demographic and comorbidity data.
RESULTS RESULTS
A total of 245 (17.7%) patients died (67.3% male, aged 74.5 ± 14.4 years); 352 (24.4%) experienced at least 1 MACE (119 arrhythmic, 107 HF, 130 TE). AI models predicted mortality and MACE with area under the curve (AUC) values of 0.60 ± 0.05 and 0.55 ± 0.07, respectively; these were comparable to AUC values for conventional models (0.73 ± 0.07 and 0.65 ± 0.10). There were no prominent temporal trends in mortality rate or MACE incidence in our cohort; holdout testing with data from after a cutoff date (June 9, 2020) did not degrade model performance.
CONCLUSION CONCLUSIONS
Using intake ECGs alone, our AI models had limited ability to predict hospitalized COVID-19 patients' risk of mortality or MACE. Our models' accuracy was comparable to that of conventional models built using more in-depth information, but translation to clinical use would require higher sensitivity and positive predictive value. In the future, we hope that mixed-input AI models utilizing both ECG and clinical data may be developed to enhance predictive accuracy.

Identifiants

pubmed: 35005676
doi: 10.1016/j.cvdhj.2021.12.003
pii: S2666-6936(21)00146-8
pmc: PMC8719367
doi:

Types de publication

Journal Article

Langues

eng

Pagination

62-74

Subventions

Organisme : NCATS NIH HHS
ID : UL1 TR002319
Pays : United States

Informations de copyright

© 2021 Heart Rhythm Society.

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Auteurs

Arun R Sridhar (AR)

Division of Cardiology, University of Washington, Seattle, Washington.

Zih-Hua Chen Amber (ZH)

Department of Bioengineering, University of Washington, Seattle, Washington.

Jacob J Mayfield (JJ)

Division of Cardiology, University of Washington, Seattle, Washington.

Alison E Fohner (AE)

Department of Epidemiology, University of Washington, Seattle, Washington.

Panagiotis Arvanitis (P)

Department of Medical Science, Uppsala University, Uppsala, Sweden.

Sarah Atkinson (S)

Division of Cardiology, University of Washington, Seattle, Washington.

Frieder Braunschweig (F)

Department of Medicine, Karolinska Institutet, Stockholm, Sweden.
Department of Cardiology, Karolinska University Hospital, Stockholm, Sweden.

Neal A Chatterjee (NA)

Division of Cardiology, University of Washington, Seattle, Washington.

Alessio Falasca Zamponi (AF)

Department of Medical Science, Uppsala University, Uppsala, Sweden.

Gregory Johnson (G)

Unaffiliated independent researcher, Seattle, Washington.

Sanika A Joshi (SA)

Department of Bioengineering, University of Washington, Seattle, Washington.

Mats C H Lassen (MCH)

Department of Cardiology, Herlev & Gentofte University Hospital, Copenhagen University, Copenhagen, Denmark.

Jeanne E Poole (JE)

Division of Cardiology, University of Washington, Seattle, Washington.

Christopher Rumer (C)

Division of Cardiology, University of Washington, Seattle, Washington.

Kristoffer G Skaarup (KG)

Department of Cardiology, Herlev & Gentofte University Hospital, Copenhagen University, Copenhagen, Denmark.

Tor Biering-Sørensen (T)

Department of Cardiology, Herlev & Gentofte University Hospital, Copenhagen University, Copenhagen, Denmark.

Carina Blomstrom-Lundqvist (C)

Department of Medical Science, Uppsala University, Uppsala, Sweden.

Cecilia M Linde (CM)

Department of Medicine, Karolinska Institutet, Stockholm, Sweden.
Department of Cardiology, Karolinska University Hospital, Stockholm, Sweden.

Mary M Maleckar (MM)

Department of Computational Physiology, Simula Research Laboratory, Oslo, Norway.

Patrick M Boyle (PM)

Department of Bioengineering, University of Washington, Seattle, Washington.
Institute for Stem Cell and Regenerative Medicine, University of Washington, Seattle, Washington.
Center for Cardiovascular Biology, University of Washington, Seattle, Washington.

Classifications MeSH