Potassium Effects on NCC Are Attenuated during Inhibition of Cullin E3-Ubiquitin Ligases.
Animals
Cullin Proteins
/ metabolism
Cyclopentanes
/ pharmacology
Dietary Supplements
Kidney Tubules
/ drug effects
Male
Mice
Mice, Inbred C57BL
Models, Biological
Phosphorylation
/ drug effects
Potassium
/ pharmacology
Pyrimidines
/ pharmacology
Solute Carrier Family 12, Member 3
/ metabolism
Ubiquitin-Protein Ligases
/ metabolism
E3 ligase
NCC
cullins
potassium
Journal
Cells
ISSN: 2073-4409
Titre abrégé: Cells
Pays: Switzerland
ID NLM: 101600052
Informations de publication
Date de publication:
29 12 2021
29 12 2021
Historique:
received:
29
11
2021
revised:
20
12
2021
accepted:
22
12
2021
entrez:
11
1
2022
pubmed:
12
1
2022
medline:
3
3
2022
Statut:
epublish
Résumé
The thiazide-sensitive sodium chloride cotransporter (NCC) plays a vital role in maintaining sodium (Na
Identifiants
pubmed: 35011657
pii: cells11010095
doi: 10.3390/cells11010095
pmc: PMC8750104
pii:
doi:
Substances chimiques
Cullin Proteins
0
Cyclopentanes
0
Pyrimidines
0
Solute Carrier Family 12, Member 3
0
Ubiquitin-Protein Ligases
EC 2.3.2.27
Potassium
RWP5GA015D
pevonedistat
S3AZD8D215
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : American Heart Association-American Stroke Association
ID : 17POST33670206
Pays : United States
Organisme : NIDDK NIH HHS
ID : R01 DK098141
Pays : United States
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