8-Isoprostanes and Asymmetric Dimethylarginine as Predictors of Mortality in Patients Following Coronary Bypass Surgery: A Long-Term Follow-Up Study.

8-iso-prostaglandin F2α asymmetric dimethylarginine coronary artery bypass grafting long-term cardiovascular death oxidative stress

Journal

Journal of clinical medicine
ISSN: 2077-0383
Titre abrégé: J Clin Med
Pays: Switzerland
ID NLM: 101606588

Informations de publication

Date de publication:
04 Jan 2022
Historique:
received: 18 11 2021
revised: 27 12 2021
accepted: 31 12 2021
entrez: 11 1 2022
pubmed: 12 1 2022
medline: 12 1 2022
Statut: epublish

Résumé

We previously demonstrated that enhanced oxidative stress and reduced nitric oxide bioavailability are associated with unfavorable outcomes early after coronary artery bypass grafting. It is not known whether these processes may impact long-term results. We sought to assess whether during long-term follow-up, markers of oxidative stress and nitric oxide bioavailability may predict cardiovascular mortality following bypass surgery. We studied 152 consecutive patients (118 men, age 65.2 ± 8.3 years) who underwent elective, primary, isolated on-pump bypass surgery. We measured plasma 8-iso-prostaglandin F2α and asymmetric dimethylarginine before surgery and twice after surgery (18-36 h and 5-7 days). We assessed all-cause and cardiovascular death in relation to these two biomarkers during a mean follow-up time of 11.7 years. The overall mortality was 44.7% (4.7 per 100 patient-years) and cardiovascular mortality was 21.0% (2.2 per 100 patient-years). Baseline 8-iso-prostaglandin F2α was associated with cardiovascular mortality (HR 1 pg/mL 1.010, 95% CI 1.001-1.021, Our findings indicate that in advanced coronary disease, increased oxidative stress, reflected by 8-iso-prostaglandin F2α before bypass surgery and enhanced asymmetric dimethylarginine accumulation just after the surgery are associated with cardiovascular death during long-term follow-up.

Sections du résumé

BACKGROUND BACKGROUND
We previously demonstrated that enhanced oxidative stress and reduced nitric oxide bioavailability are associated with unfavorable outcomes early after coronary artery bypass grafting. It is not known whether these processes may impact long-term results. We sought to assess whether during long-term follow-up, markers of oxidative stress and nitric oxide bioavailability may predict cardiovascular mortality following bypass surgery.
METHODS METHODS
We studied 152 consecutive patients (118 men, age 65.2 ± 8.3 years) who underwent elective, primary, isolated on-pump bypass surgery. We measured plasma 8-iso-prostaglandin F2α and asymmetric dimethylarginine before surgery and twice after surgery (18-36 h and 5-7 days). We assessed all-cause and cardiovascular death in relation to these two biomarkers during a mean follow-up time of 11.7 years.
RESULTS RESULTS
The overall mortality was 44.7% (4.7 per 100 patient-years) and cardiovascular mortality was 21.0% (2.2 per 100 patient-years). Baseline 8-iso-prostaglandin F2α was associated with cardiovascular mortality (HR 1 pg/mL 1.010, 95% CI 1.001-1.021,
CONCLUSIONS CONCLUSIONS
Our findings indicate that in advanced coronary disease, increased oxidative stress, reflected by 8-iso-prostaglandin F2α before bypass surgery and enhanced asymmetric dimethylarginine accumulation just after the surgery are associated with cardiovascular death during long-term follow-up.

Identifiants

pubmed: 35011987
pii: jcm11010246
doi: 10.3390/jcm11010246
pmc: PMC8745691
pii:
doi:

Types de publication

Journal Article

Langues

eng

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Auteurs

Aleksandra Gołąb (A)

Faculty of Medicine and Dentistry, Pomeranian Medical University, 70-204 Szczecin, Poland.

Dariusz Plicner (D)

Unit of Experimental Cardiology and Cardiac Surgery, Faculty of Medicine and Health Sciences, Andrzej Frycz Modrzewski Krakow University, 30-705 Krakow, Poland.
Department of Cardiovascular Surgery and Transplantation, John Paul II Hospital, 31-202 Krakow, Poland.

Anna Rzucidło-Hymczak (A)

Department of Pediatric Infectious Diseases and Pediatric Hepatology, John Paul II Hospital, 31-202 Krakow, Poland.

Lidia Tomkiewicz-Pająk (L)

Department of Cardiac and Vascular Diseases, John Paul II Hospital, 31-202 Krakow, Poland.
Institute of Cardiology, Jagiellonian University Medical College, 31-008 Krakow, Poland.

Bogusław Gawęda (B)

Division of Cardiovascular Surgery, St. Jadwiga Provincial Clinical Hospital, Medical College of Rzeszow University, 35-310 Rzeszow, Poland.

Bogusław Kapelak (B)

Department of Cardiovascular Surgery and Transplantation, John Paul II Hospital, 31-202 Krakow, Poland.
Institute of Cardiology, Jagiellonian University Medical College, 31-008 Krakow, Poland.

Anetta Undas (A)

Institute of Cardiology, Jagiellonian University Medical College, 31-008 Krakow, Poland.
Center for Research and Innovative Technology, John Paul II Hospital, 31-202 Krakow, Poland.

Classifications MeSH