Temporal Artery Vascular Diseases.

ANCA-associated vasculitis giant cell arteritis temporal arteritis varicella-zoster virus

Journal

Journal of clinical medicine
ISSN: 2077-0383
Titre abrégé: J Clin Med
Pays: Switzerland
ID NLM: 101606588

Informations de publication

Date de publication:
05 Jan 2022
Historique:
received: 07 12 2021
revised: 24 12 2021
accepted: 30 12 2021
entrez: 11 1 2022
pubmed: 12 1 2022
medline: 12 1 2022
Statut: epublish

Résumé

In the presence of temporal arteritis, clinicians often refer to the diagnosis of giant cell arteritis (GCA). However, differential diagnoses should also be evoked because other types of vascular diseases, vasculitis or not, may affect the temporal artery. Among vasculitis, Anti-neutrophil cytoplasmic antibodies (ANCA)-associated vasculitis is probably the most common, and typically affects the peri-adventitial small vessel of the temporal artery and sometimes mimics giant cell arteritis, however, other symptoms are frequently associated and more specific of ANCA-associated vasculitis prompt a search for ANCA. The Immunoglobulin G4-related disease (IgG4-RD) can cause temporal arteritis as well. Some infections can also affect the temporal artery, primarily an infection caused by the varicella-zoster virus (VZV), which has an arterial tropism that may play a role in triggering giant cell arteritis. Drugs, mainly checkpoint inhibitors that are used to treat cancer, can also trigger giant cell arteritis. Furthermore, the temporal artery can be affected by diseases other than vasculitis such as atherosclerosis, calcyphilaxis, aneurysm, or arteriovenous fistula. In this review, these different diseases affecting the temporal artery are described.

Identifiants

pubmed: 35012016
pii: jcm11010275
doi: 10.3390/jcm11010275
pmc: PMC8745856
pii:
doi:

Types de publication

Journal Article Review

Langues

eng

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Auteurs

Hélène Greigert (H)

Department of Internal Medicine and Clinical Immunology, Dijon University Hospital, 21000 Dijon, France.
Department of Vascular Medicine, Dijon University Hospital, 21000 Dijon, France.
Interactions Hôte-Greffon-Tumeur/Ingénierie Cellulaire et Génique, LabEx LipSTIC, INSERM, EFS BFC, UMR1098, Université de Bourgogne Franche-Comté, 21000 Dijon, France.

André Ramon (A)

Department of Rheumatology, Dijon University Hospital, 21000 Dijon, France.

Georges Tarris (G)

Department of Pathology, Dijon University Hospital, 21000 Dijon, France.

Laurent Martin (L)

Department of Pathology, Dijon University Hospital, 21000 Dijon, France.

Bernard Bonnotte (B)

Department of Internal Medicine and Clinical Immunology, Dijon University Hospital, 21000 Dijon, France.
Interactions Hôte-Greffon-Tumeur/Ingénierie Cellulaire et Génique, LabEx LipSTIC, INSERM, EFS BFC, UMR1098, Université de Bourgogne Franche-Comté, 21000 Dijon, France.

Maxime Samson (M)

Department of Internal Medicine and Clinical Immunology, Dijon University Hospital, 21000 Dijon, France.
Interactions Hôte-Greffon-Tumeur/Ingénierie Cellulaire et Génique, LabEx LipSTIC, INSERM, EFS BFC, UMR1098, Université de Bourgogne Franche-Comté, 21000 Dijon, France.

Classifications MeSH