Of vascular defense, hemostasis, cancer, and platelet biology: an evolutionary perspective.
Coagulation
Corona virus disease 2019 (COVID-19)
C–C chemokine receptors (CCR 1,3And 4)
Platelet
Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2)
Toll-like receptor (TLR)
Journal
Cancer metastasis reviews
ISSN: 1573-7233
Titre abrégé: Cancer Metastasis Rev
Pays: Netherlands
ID NLM: 8605731
Informations de publication
Date de publication:
03 2022
03 2022
Historique:
received:
12
11
2021
accepted:
04
01
2022
pubmed:
14
1
2022
medline:
29
3
2022
entrez:
13
1
2022
Statut:
ppublish
Résumé
We have established considerable expertise in studying the role of platelets in cancer biology. From this expertise, we were keen to recognize the numerous venous-, arterial-, microvascular-, and macrovascular thrombotic events and immunologic disorders are caused by severe, acute-respiratory-syndrome coronavirus 2 (SARS-CoV-2) infections. With this offering, we explore the evolutionary connections that place platelets at the center of hemostasis, immunity, and adaptive phylogeny. Coevolutionary changes have also occurred in vertebrate viruses and their vertebrate hosts that reflect their respective evolutionary interactions. As mammals adapted from aquatic to terrestrial life and the heavy blood loss associated with placentalization-based live birth, platelets evolved phylogenetically from thrombocytes toward higher megakaryocyte-blebbing-based production rates and the lack of nuclei. With no nuclei and robust RNA synthesis, this adaptation may have influenced viral replication to become less efficient after virus particles are engulfed. Human platelets express numerous receptors that bind viral particles, which developed from archetypal origins to initiate aggregation and exocytic-release of thrombo-, immuno-, angiogenic-, growth-, and repair-stimulatory granule contents. Whether by direct, evolutionary, selective pressure, or not, these responses may help to contain virus spread, attract immune cells for eradication, and stimulate angiogenesis, growth, and wound repair after viral damage. Because mammalian and marsupial platelets became smaller and more plate-like their biophysical properties improved in function, which facilitated distribution near vessel walls in fluid-shear fields. This adaptation increased the probability that platelets could then interact with and engulf shedding virus particles. Platelets also generate circulating microvesicles that increase membrane surface-area encounters and mark viral targets. In order to match virus-production rates, billions of platelets are generated and turned over per day to continually provide active defenses and adaptation to suppress the spectrum of evolving threats like SARS-CoV-2.
Identifiants
pubmed: 35022962
doi: 10.1007/s10555-022-10019-5
pii: 10.1007/s10555-022-10019-5
pmc: PMC8754476
doi:
Types de publication
Journal Article
Research Support, N.I.H., Extramural
Research Support, Non-U.S. Gov't
Review
Langues
eng
Sous-ensembles de citation
IM
Pagination
147-172Subventions
Organisme : NCI NIH HHS
ID : R01CA177909
Pays : United States
Organisme : University of Texas MD Anderson Cancer Center
ID : Pancreatic Moon Shot
Organisme : NCI NIH HHS
ID : P50 CA098258
Pays : United States
Organisme : University of Texas MD Anderson Cancer Center
ID : IRG
Organisme : NCI NIH HHS
ID : R01 CA177909
Pays : United States
Organisme : NCI NIH HHS
ID : P50CA221707
Pays : United States
Organisme : University of Texas MD Anderson Cancer Center
ID : Ovarian Moon Shot
Organisme : University of Texas MD Anderson Cancer Center
ID : Frank McGraw Memorial Chair in Cancer Research
Organisme : NCI NIH HHS
ID : R01CA218230
Pays : United States
Organisme : University of Texas MD Anderson Cancer Center
ID : Moonshot
Organisme : NCI NIH HHS
ID : P50CA217685
Pays : United States
Organisme : NCI NIH HHS
ID : 3P30CA016672
Pays : United States
Organisme : NCI NIH HHS
ID : R01CA231141
Pays : United States
Organisme : NCI NIH HHS
ID : P01CA117969
Pays : United States
Informations de copyright
© 2022. The Author(s), under exclusive licence to Springer Science+Business Media, LLC, part of Springer Nature.
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