Carotid Artery Plaque Progression: Proposal of a New Predictive Score and Role of Carotid Intima-Media Thickness.


Journal

International journal of environmental research and public health
ISSN: 1660-4601
Titre abrégé: Int J Environ Res Public Health
Pays: Switzerland
ID NLM: 101238455

Informations de publication

Date de publication:
11 01 2022
Historique:
received: 15 11 2021
revised: 05 01 2022
accepted: 08 01 2022
entrez: 21 1 2022
pubmed: 22 1 2022
medline: 1 3 2022
Statut: epublish

Résumé

We aimed to investigate if the carotid intima-media thickness (IMT) at baseline and the HAD We performed a retrospective analysis on real-life prospectively collected data from patients with any detectable carotid plaque at follow up. The plaque score, calculated at baseline (T0) and at a median follow up of 36.6 months (IQR 39.6-34.3) (T3), was defined as 0: no plaque or stenosis < 30%; 1: stenosis in the range 30-49%; 2: in the range 50-69%; 3: in the range 70-99% and 4: occlusion. Carotid IMT was measured at T0 and T3; HAD We included 340 patients with a mean age of 69.9 (9.1) years and 25.3% subjects had plaque progression. Individuals with progression had a median HAD Baseline IMT could be considered a predictor of progression. Patients with progression had an HAD

Sections du résumé

BACKGROUND
We aimed to investigate if the carotid intima-media thickness (IMT) at baseline and the HAD
METHODS
We performed a retrospective analysis on real-life prospectively collected data from patients with any detectable carotid plaque at follow up. The plaque score, calculated at baseline (T0) and at a median follow up of 36.6 months (IQR 39.6-34.3) (T3), was defined as 0: no plaque or stenosis < 30%; 1: stenosis in the range 30-49%; 2: in the range 50-69%; 3: in the range 70-99% and 4: occlusion. Carotid IMT was measured at T0 and T3; HAD
RESULTS
We included 340 patients with a mean age of 69.9 (9.1) years and 25.3% subjects had plaque progression. Individuals with progression had a median HAD
CONCLUSION
Baseline IMT could be considered a predictor of progression. Patients with progression had an HAD

Identifiants

pubmed: 35055580
pii: ijerph19020758
doi: 10.3390/ijerph19020758
pmc: PMC8776120
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

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Auteurs

Nicoletta Brunelli (N)

Headache and Neurosonology Unit, Neurology, Campus Bio-Medico University of Rome, 00128 Rome, Italy.

Claudia Altamura (C)

Headache and Neurosonology Unit, Neurology, Campus Bio-Medico University of Rome, 00128 Rome, Italy.

Carmelina Maria Costa (CM)

Headache and Neurosonology Unit, Neurology, Campus Bio-Medico University of Rome, 00128 Rome, Italy.

Riccardo Altavilla (R)

Neurology and Stroke Unit Department, ASST Santi Paolo e Carlo, 20142 Milan, Italy.

Paola Palazzo (P)

Department of Neurology, Lausanne University Hospital, Rue du Bugnon 46, 1011 Lausanne, Switzerland.
Neurology Unit, Riviera-Chablais Hospital, Route du Vieux-Séquoia 20, 1847 Rennaz, Switzerland.

Paola Maggio (P)

Neurology Unit, ASST Bergamo Est, 24068 Bergamo, Italy.

Marilena Marcosano (M)

Headache and Neurosonology Unit, Neurology, Campus Bio-Medico University of Rome, 00128 Rome, Italy.

Fabrizio Vernieri (F)

Headache and Neurosonology Unit, Neurology, Campus Bio-Medico University of Rome, 00128 Rome, Italy.

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