Real world risk of infusion reactions and effectiveness of front-line obinutuzumab plus chlorambucil compared with other frontline treatments for chronic lymphocytic leukemia.


Journal

BMC cancer
ISSN: 1471-2407
Titre abrégé: BMC Cancer
Pays: England
ID NLM: 100967800

Informations de publication

Date de publication:
06 Feb 2022
Historique:
received: 15 06 2021
accepted: 31 01 2022
entrez: 6 2 2022
pubmed: 7 2 2022
medline: 11 3 2022
Statut: epublish

Résumé

Chronic lymphocytic leukemia (CLL) is the most common type of leukemia in North America. Previous studies have shown improved progression free survival (PFS) and response rates in unfit patients treated with obinutuzumab compared to other regimens. The aim of this study was to evaluate the obinutuzumab-chlorambucil regimen in the context of historical treatments and first-dose infusion reactions at CancerCare Manitoba (CCMB). A retrospective chart review was conducted for patients treated with obinutuzumab from January 1, 2014 to December 31, 2017 at CCMB. A minimum data set was extracted for patients treated with other front-line therapies. Descriptive statistics were used to evaluate patient demographics, toxicity, duration and dosing of obinutuzumab treatment. Kaplan-Meier curves were used to evaluate time-to-next-treatment (TTNT), overall survival (OS) and PFS for patients treated with obinutuzumab. A multivariable logistic regression model was used to investigate associations between infusion related reactions (IRRs) and age at treatment, pre-treatment lymphocyte count, cumulative illness rating scale (CIRS) and receipt of prior chemotherapy. Forty seven percent of patients receiving frontline therapy received chlorambucil and obinutuzumab. Sixty-seven patients were treated with obinutuzumab and consisted of 36 males (53.7%) and 31 females (46.3%) with 29 patients (43.3%) over age 75 years. Rates of grade 3 and 4 obinutuzumab IRRs were lower (6%) compared to the CLL11 clinical trial (20%) due to local practices including slower infusion rates and using chlorambucil before starting obinutuzumab treatment. Many patients had difficulty tolerating the full dosage of chlorambucil. Only 26 patients (38.8%) had their dose of chlorambucil escalated to the full dose of 0.5 mg/kg. In addition, only 18 patients (26.9%) received all doses of obinutuzumab and all 12 doses of chlorambucil. In summary, first dose infusion reactions with obinutuzumab can be markedly reduced by using chlorambucil to decrease the lymphocyte count before obinutuzumab and by using a very slow initial obinutuzumab infusion rate. Modifications in chlorambucil dosing and obinutuzumab administration can improve tolerance without significant loss in efficacy.

Sections du résumé

BACKGROUND BACKGROUND
Chronic lymphocytic leukemia (CLL) is the most common type of leukemia in North America. Previous studies have shown improved progression free survival (PFS) and response rates in unfit patients treated with obinutuzumab compared to other regimens. The aim of this study was to evaluate the obinutuzumab-chlorambucil regimen in the context of historical treatments and first-dose infusion reactions at CancerCare Manitoba (CCMB).
METHODS METHODS
A retrospective chart review was conducted for patients treated with obinutuzumab from January 1, 2014 to December 31, 2017 at CCMB. A minimum data set was extracted for patients treated with other front-line therapies. Descriptive statistics were used to evaluate patient demographics, toxicity, duration and dosing of obinutuzumab treatment. Kaplan-Meier curves were used to evaluate time-to-next-treatment (TTNT), overall survival (OS) and PFS for patients treated with obinutuzumab. A multivariable logistic regression model was used to investigate associations between infusion related reactions (IRRs) and age at treatment, pre-treatment lymphocyte count, cumulative illness rating scale (CIRS) and receipt of prior chemotherapy.
RESULTS RESULTS
Forty seven percent of patients receiving frontline therapy received chlorambucil and obinutuzumab. Sixty-seven patients were treated with obinutuzumab and consisted of 36 males (53.7%) and 31 females (46.3%) with 29 patients (43.3%) over age 75 years. Rates of grade 3 and 4 obinutuzumab IRRs were lower (6%) compared to the CLL11 clinical trial (20%) due to local practices including slower infusion rates and using chlorambucil before starting obinutuzumab treatment. Many patients had difficulty tolerating the full dosage of chlorambucil. Only 26 patients (38.8%) had their dose of chlorambucil escalated to the full dose of 0.5 mg/kg. In addition, only 18 patients (26.9%) received all doses of obinutuzumab and all 12 doses of chlorambucil.
CONCLUSIONS CONCLUSIONS
In summary, first dose infusion reactions with obinutuzumab can be markedly reduced by using chlorambucil to decrease the lymphocyte count before obinutuzumab and by using a very slow initial obinutuzumab infusion rate. Modifications in chlorambucil dosing and obinutuzumab administration can improve tolerance without significant loss in efficacy.

Identifiants

pubmed: 35123433
doi: 10.1186/s12885-022-09256-2
pii: 10.1186/s12885-022-09256-2
pmc: PMC8818183
doi:

Substances chimiques

Antibodies, Monoclonal, Humanized 0
Chlorambucil 18D0SL7309
obinutuzumab O43472U9X8

Types de publication

Evaluation Study Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

148

Informations de copyright

© 2022. The Author(s).

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Auteurs

Nicole Bourrier (N)

Max Rady College of Medicine, 727 McDermot Ave, Winnipeg, MB, R3E 3P5, Canada.

Ivan Landego (I)

Department of Internal Medicine, Rady Faculty of Health Sciences, University of Manitoba, 727 McDermot Ave, Winnipeg, MB, R3E 3P5, Canada.

Oliver Bucher (O)

Department of Epidemiology, CancerCare Manitoba, 675 McDermot Ave, Winnipeg, MB, R3E 0V9, Canada.

Mandy Squires (M)

CancerCare Manitoba Research Institute, 675 McDermot Ave, Winnipeg, MB, R3E 0V9, Canada.

Erin Streu (E)

Department of Nursing, CancerCare Manitoba, 675 McDermot Ave, Winnipeg, MB, R3E 0V9, Canada.

Irena Hibbert (I)

Department of Nursing, CancerCare Manitoba, 675 McDermot Ave, Winnipeg, MB, R3E 0V9, Canada.

Theresa Whiteside (T)

Department of Nursing, CancerCare Manitoba, 675 McDermot Ave, Winnipeg, MB, R3E 0V9, Canada.

Spencer B Gibson (SB)

CancerCare Manitoba Research Institute, 675 McDermot Ave, Winnipeg, MB, R3E 0V9, Canada.
Department of Biochemistry and Medical Genetics, Max Rady College of Medicine University of Manitoba, 727 McDermot Ave, Winnipeg, MB, R3E 3P5, Canada.
Department of Immunology, Max Rady College of Medicine University of Manitoba, 727 McDermot Ave, Winnipeg, MB, R3E 3P5, Canada.

Marc Geirnaert (M)

Provincial Oncology Drug Program, CancerCare Manitoba, 675 McDermot Ave, Winnipeg, MB, R3E 0V9, Canada.

James B Johnston (JB)

Department of Internal Medicine, Rady Faculty of Health Sciences, University of Manitoba, 727 McDermot Ave, Winnipeg, MB, R3E 3P5, Canada.
CancerCare Manitoba Research Institute, 675 McDermot Ave, Winnipeg, MB, R3E 0V9, Canada.

David E Dawe (DE)

Department of Internal Medicine, Rady Faculty of Health Sciences, University of Manitoba, 727 McDermot Ave, Winnipeg, MB, R3E 3P5, Canada.
CancerCare Manitoba Research Institute, 675 McDermot Ave, Winnipeg, MB, R3E 0V9, Canada.
Department of Medical Oncology and Hematology, CancerCare Manitoba, 675 McDermot Ave, Winnipeg, MB, R3E 0V9, Canada.

Versha Banerji (V)

Max Rady College of Medicine, 727 McDermot Ave, Winnipeg, MB, R3E 3P5, Canada. vbanerji@cancercare.mb.ca.
Department of Internal Medicine, Rady Faculty of Health Sciences, University of Manitoba, 727 McDermot Ave, Winnipeg, MB, R3E 3P5, Canada. vbanerji@cancercare.mb.ca.
CancerCare Manitoba Research Institute, 675 McDermot Ave, Winnipeg, MB, R3E 0V9, Canada. vbanerji@cancercare.mb.ca.
Department of Biochemistry and Medical Genetics, Max Rady College of Medicine University of Manitoba, 727 McDermot Ave, Winnipeg, MB, R3E 3P5, Canada. vbanerji@cancercare.mb.ca.
Department of Medical Oncology and Hematology, CancerCare Manitoba, 675 McDermot Ave, Winnipeg, MB, R3E 0V9, Canada. vbanerji@cancercare.mb.ca.

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