Why do humans undergo an adiposity rebound? Exploring links with the energetic costs of brain development in childhood using MRI-based 4D measures of total cerebral blood flow.


Journal

International journal of obesity (2005)
ISSN: 1476-5497
Titre abrégé: Int J Obes (Lond)
Pays: England
ID NLM: 101256108

Informations de publication

Date de publication:
05 2022
Historique:
received: 25 09 2021
accepted: 07 01 2022
revised: 30 12 2021
pubmed: 10 2 2022
medline: 3 5 2022
entrez: 9 2 2022
Statut: ppublish

Résumé

Individuals typically show a childhood nadir in adiposity termed the adiposity rebound (AR). The AR serves as an early predictor of obesity risk, with early rebounders often at increased risk; however, it is unclear why this phenomenon occurs, which could impede understandings of weight gain trajectories. The brain's energy requirements account for a lifetime peak of 66% of the body's resting metabolic expenditure during childhood, around the age of the AR, and relates inversely to weight gain, pointing to a potential energy trade-off between brain development and adiposity. However, no study has compared developmental trajectories of brain metabolism and adiposity in the same individuals, which would allow a preliminary test of a brain-AR link. We used cubic splines and generalized additive models to compare age trajectories of previously collected MRI-based 4D flow measures of total cerebral blood flow (TCBF), a proxy for cerebral energy use, to the body mass index (BMI) in a cross-sectional sample of 82 healthy individuals (0-60 years). We restricted our AR analysis to pre-pubertal individuals (0-12 years, n = 42), predicting that peak TCBF would occur slightly after the BMI nadir, consistent with evidence that lowest BMI typically precedes the nadir in adiposity. TCBF and the BMI showed inverse trajectories throughout childhood, while the estimated age at peak TCBF (5.6 years) was close but slightly later than the estimated age of the BMI nadir (4.9 years). The timing of peak TCBF in this sample points to a likely concordance between peak brain energetics and the nadir in adiposity. Inverse age trajectories between TCBF and BMI support the hypothesis that brain metabolism is a potentially important influence on early life adiposity. These findings also suggest that experiences influencing the pattern of childhood brain energy use could be important predictors of body composition trajectories.

Sections du résumé

BACKGROUND
Individuals typically show a childhood nadir in adiposity termed the adiposity rebound (AR). The AR serves as an early predictor of obesity risk, with early rebounders often at increased risk; however, it is unclear why this phenomenon occurs, which could impede understandings of weight gain trajectories. The brain's energy requirements account for a lifetime peak of 66% of the body's resting metabolic expenditure during childhood, around the age of the AR, and relates inversely to weight gain, pointing to a potential energy trade-off between brain development and adiposity. However, no study has compared developmental trajectories of brain metabolism and adiposity in the same individuals, which would allow a preliminary test of a brain-AR link.
METHODS
We used cubic splines and generalized additive models to compare age trajectories of previously collected MRI-based 4D flow measures of total cerebral blood flow (TCBF), a proxy for cerebral energy use, to the body mass index (BMI) in a cross-sectional sample of 82 healthy individuals (0-60 years). We restricted our AR analysis to pre-pubertal individuals (0-12 years, n = 42), predicting that peak TCBF would occur slightly after the BMI nadir, consistent with evidence that lowest BMI typically precedes the nadir in adiposity.
RESULTS
TCBF and the BMI showed inverse trajectories throughout childhood, while the estimated age at peak TCBF (5.6 years) was close but slightly later than the estimated age of the BMI nadir (4.9 years).
CONCLUSIONS
The timing of peak TCBF in this sample points to a likely concordance between peak brain energetics and the nadir in adiposity. Inverse age trajectories between TCBF and BMI support the hypothesis that brain metabolism is a potentially important influence on early life adiposity. These findings also suggest that experiences influencing the pattern of childhood brain energy use could be important predictors of body composition trajectories.

Identifiants

pubmed: 35136192
doi: 10.1038/s41366-022-01065-8
pii: 10.1038/s41366-022-01065-8
pmc: PMC9050592
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1044-1050

Subventions

Organisme : NCI NIH HHS
ID : P30 CA008748
Pays : United States

Informations de copyright

© 2022. The Author(s).

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Auteurs

Jacob E Aronoff (JE)

Department of Anthropology, Northwestern University, Evanston, IL, USA. aronoff@u.northwestern.edu.

Ann Ragin (A)

Department of Radiology, Northwestern University Feinberg School of Medicine, Chicago, IL, USA.

Can Wu (C)

Department of Medical Physics, Memorial Sloan Kettering Cancer Center, New York, NY, USA.

Michael Markl (M)

Department of Radiology, Northwestern University Feinberg School of Medicine, Chicago, IL, USA.
Department of Biomedical Engineering, Northwestern University McCormick School of Engineering, Chicago, IL, USA.

Susanne Schnell (S)

Department of Radiology, Northwestern University Feinberg School of Medicine, Chicago, IL, USA.
Department of Medical Physics, Institute of Physics, University of Greifswald, Greifswald, Germany.

Ali Shaibani (A)

Department of Radiology, Northwestern University Feinberg School of Medicine, Chicago, IL, USA.
Department of Neurosurgery, Northwestern University Feinberg School of Medicine, Chicago, IL, USA.

Clancy Blair (C)

Department of Population Health, NYU School of Medicine, New York, NY, USA.
Department of Applied Psychology, NYU Steinhardt, New York, NY, USA.

Christopher W Kuzawa (CW)

Department of Anthropology, Northwestern University, Evanston, IL, USA. kuzawa@northwestern.edu.
Institute for Policy Research, Northwestern University, Evanston, IL, USA. kuzawa@northwestern.edu.

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