Glucosylceramide in T cells regulates the pathology of inflammatory bowel disease.


Journal

Biochemical and biophysical research communications
ISSN: 1090-2104
Titre abrégé: Biochem Biophys Res Commun
Pays: United States
ID NLM: 0372516

Informations de publication

Date de publication:
09 04 2022
Historique:
received: 24 12 2021
accepted: 01 02 2022
pubmed: 16 2 2022
medline: 18 3 2022
entrez: 15 2 2022
Statut: ppublish

Résumé

Inflammatory bowel disease (IBD) is a chronic inflammatory disease in the colon characterized by excessive activation of T cells. Glycosphingolipids (GSLs) are composed of lipid rafts in cellular membranes, and their content is linked to immune cell function. In the present study, we investigated the involvement of GSLs in IBD. Microarray data showed that in IBD patients, the expression of only UDP-glucose ceramide glucosyltransferase (UGCG) decreased among the GSLs synthases. Ad libitum access to dextran sulfate sodium (DSS) resulted in decreased UGCG and glucosylceramide (GlcCer) content in mesenteric lymph nodes and T cells from the spleen. Furthermore, the knockdown of Ugcg in T cells exacerbated the pathogenesis of colitis, which was accompanied by a decrease in Treg levels. Treatment with GlcCer nanoparticles prevented DSS-induced colitis. These results suggested that GlcCer in T cells is involved in the pathogenesis of IBD. Furthermore, GlcCer nanoparticles are a potential efficacious therapeutic target for IBD patients.

Identifiants

pubmed: 35168060
pii: S0006-291X(22)00180-2
doi: 10.1016/j.bbrc.2022.02.004
pii:
doi:

Substances chimiques

Glucosylceramides 0
Dextran Sulfate 9042-14-2
Glucosyltransferases EC 2.4.1.-
ceramide glucosyltransferase EC 2.4.1.80

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

24-30

Informations de copyright

Copyright © 2022 Elsevier Inc. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest Azabu University and Hokkaido University hold patents on the usage of GlcCer nanoparticles. MK, MN, ER, TN, and TY are the inventors in these patents. Remaining authors do not declare any competing interest.

Auteurs

Mariko Komuro (M)

Laboratory of Biochemistry, School of Veterinary Medicine, Azabu University, Sagamihara, Kanagawa, Japan.

Masaki Nagane (M)

Laboratory of Biochemistry, School of Veterinary Medicine, Azabu University, Sagamihara, Kanagawa, Japan; Center for Human and Animal Symbiosis Science, Azabu University, Sagamihara, Kanagawa, Japan. Electronic address: nagane@azabu-u.ac.jp.

Rikito Endo (R)

Faculty of Pharmaceutical Sciences, Hokkaido University, Sapporo, Hokkaido, Japan.

Takashi Nakamura (T)

Faculty of Pharmaceutical Sciences, Hokkaido University, Sapporo, Hokkaido, Japan.

Takayoshi Miyamoto (T)

Laboratory of Biochemistry, School of Veterinary Medicine, Azabu University, Sagamihara, Kanagawa, Japan.

Chiaki Niwa (C)

Laboratory of Biochemistry, School of Veterinary Medicine, Azabu University, Sagamihara, Kanagawa, Japan.

Tomoki Fukuyama (T)

Laboratory of Pharmacology, School of Veterinary Medicine, Azabu University, Sagamihara, Kanagawa, Japan.

Hideyoshi Harashima (H)

Faculty of Pharmaceutical Sciences, Hokkaido University, Sapporo, Hokkaido, Japan.

Naoyuki Aihara (N)

Laboratory of Veterinary Pathology, School of Veterinary Medicine, Azabu University, Sagamihara, Kanagawa, Japan.

Junichi Kamiie (J)

Laboratory of Veterinary Pathology, School of Veterinary Medicine, Azabu University, Sagamihara, Kanagawa, Japan.

Rimina Suzuki (R)

Laboratory of Biochemistry, School of Veterinary Medicine, Azabu University, Sagamihara, Kanagawa, Japan.

Tadashi Yamashita (T)

Laboratory of Biochemistry, School of Veterinary Medicine, Azabu University, Sagamihara, Kanagawa, Japan.

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Classifications MeSH