Ex vivo platelet morphology assessment of chronic myeloid leukemia patients before and after Imatinib treatment.


Journal

Microscopy research and technique
ISSN: 1097-0029
Titre abrégé: Microsc Res Tech
Pays: United States
ID NLM: 9203012

Informations de publication

Date de publication:
Jun 2022
Historique:
revised: 15 12 2021
received: 06 09 2021
accepted: 19 01 2022
pubmed: 18 2 2022
medline: 26 5 2022
entrez: 17 2 2022
Statut: ppublish

Résumé

Chronic myeloid leukemia (CML) is a myeloproliferative disease and the first line treatment is through the administration of Imatinib, a first generation tyrosine kinase inhibitor. Thrombocytosis and bleeding irregularities are common in CML, however, the morphological variations in CML patients' platelets are not well documented. In this study, ex vivo platelet morphology of control participants, as well as CML patients were assessed before and after Imatinib treatment. The topographical and structural morphology of platelets were determined via scanning electron microscopy (SEM) and transmission electron microscopy (TEM). Qualitative data of SEM and TEM revealed that CML patient's platelets were prone to aggregation and coagulation at time of diagnosis; the samples that were not aggregated at time of diagnosis showed typical discoid shaped platelets, which was comparable to control participants' platelets. TEM results of CML patients' platelets at diagnosis showed that internal granular constituents including dense bodies were decreased in comparison to control participants. In all CML patients, platelets appeared activated after 6 months of treatment with Imatinib with membrane structure abnormalities and constituent variations. Research to date has primarily focused on the effects of CML on leukocyte populations, however, the results of the current study implicate the impact of CML pathogenesis on platelets, seemingly as a result of alterations in normal hematopoiesis. In addition, the impact of Imatinib treatment on platelet morphology was also established, indicating an increase in platelet activation. Recognizing and understanding the impact of CML disease progression on platelets is of importance to aid improved patient treatment. RESEARCH HIGHLIGHTS: In the study, results from SEM and TEM indicated that CML patient's platelets were prone to aggregation at time of diagnosis, and activation after Imatnib treatment. Platelet samples that did not aggregate had decreased internal granular constituents.

Identifiants

pubmed: 35174933
doi: 10.1002/jemt.24079
doi:

Substances chimiques

Antineoplastic Agents 0
Protein Kinase Inhibitors 0
Imatinib Mesylate 8A1O1M485B

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

2222-2233

Subventions

Organisme : Cancer Association of South Africa
ID : AOV741
Organisme : Cancer Association of South Africa
ID : AOW228
Organisme : Medical Research Council of South Africa
ID : AOW110
Organisme : National Research Foundation
ID : N00375
Organisme : National Research Foundation
ID : N00591
Organisme : School of Medicine Research Committee of the Faculty of Health Sciences
ID : AOR984
Organisme : Struwig-Germeshuysen Research Trust
ID : AON074

Informations de copyright

© 2022 Wiley Periodicals LLC.

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Auteurs

Lisa Repsold (L)

Department of Physiology, Faculty of Health Sciences, School of Medicine, University of Pretoria, Pretoria, Gauteng, South Africa.

Roger Pool (R)

Department of Haematology, Faculty of Health Sciences, School of Medicine, University of Pretoria, Pretoria, Gauteng, South Africa.

Mohammed Karodia (M)

Department of Haematology, Faculty of Health Sciences, School of Medicine, University of Pretoria, Pretoria, Gauteng, South Africa.

Gregory Tintinger (G)

Department of Internal Medicine, Faculty of Health Sciences, School of Medicine, University of Pretoria, Pretoria, Gauteng, South Africa.

Anna Margaretha Joubert (AM)

Department of Physiology, Faculty of Health Sciences, School of Medicine, University of Pretoria, Pretoria, Gauteng, South Africa.

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