Liver pathological alterations in fetal rabbit model of congenital diaphragmatic hernia.

congenital diaphragmatic hernia fetal liver pediatric translational research rabbits

Journal

Congenital anomalies
ISSN: 1741-4520
Titre abrégé: Congenit Anom (Kyoto)
Pays: Australia
ID NLM: 9306292

Informations de publication

Date de publication:
May 2022
Historique:
revised: 14 11 2021
received: 14 08 2021
accepted: 20 12 2021
pubmed: 19 2 2022
medline: 6 5 2022
entrez: 18 2 2022
Statut: ppublish

Résumé

To date, fetal liver implication is not a well-understood phenomenon in congenital diaphragmatic hernia (CDH). We evaluated the fetal morphologic changes on liver growth after surgical procedure in CDH experimental model. A diaphragmatic defect at gestational day E25 and tracheal occlusion (TO) at E27 were surgically created in rabbit fetuses. Five experimental groups were assessed: control group, left CDH, right CDH, CDH + TO, and TO alone. Body and organ growth were measured. For histological evaluation of the CDH effect, liver sections were collected. Left-CDH group had livers with increased leukocyte infiltration in comparison with controls (p = 0.02). Increased capillary sinusoid congestion and hepatocyte vacuolation were greater in left-CDH compared with the right-CDH group (p = 0.05). Capillary sinusoid congestion and interstitial edema were more evident in the left-CDH compared with CDH + TO group (p = 0.05). Increases in sinusoid congestion, hepatocyte vacuolation, and interstitial edema were also greater in the CDH + TO compared with controls (p ≤ 0.02). Intrathoracic liver weight was higher in right-CDH compared with left-CDH group (p < 0.001). Total lung weights (TLW) were significantly lower in both left-CDH compared with controls (p < 0.001), CDH + TO (p = 0.01), and TO (p < 0.01) and in right-CDH compared with CDH + TO (p < 0.01) and TO (p < 0.01). Decreased kidney and heart weights were also recorded. Hemodynamics and structural fetal liver changes in laterality were noted in CDH model. Regulation of intrathoracic liver weights seems to be disturbed by the absence of diaphragmic contact. Pulmonary injury is supported by the effect of a first hit, while the growth of internal organs suggests a multisystemic remodeling related to the fetal adaptation.

Identifiants

pubmed: 35178773
doi: 10.1111/cga.12462
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

105-112

Informations de copyright

© 2022 Japanese Teratology Society.

Références

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Auteurs

Gloria Pelizzo (G)

Department of Biomedical and Clinical Sciences "L. Sacco", University of Milan, Milan, Italy.
Department of Pediatric Surgery, "V Buzzi" Children's Hospital, University of Milan, Milan, Italy.

José L Peiro (JL)

Center for Fetal and Placental Research, Division of Pediatric General and Thoracic Surgery, Cincinnati Children's Hospital Medical Center (CCHMC), Cincinnati, Ohio, USA.
University of Cincinnati, College of Medicine, Cincinnati, Ohio, USA.

Vincenzo Villanacci (V)

Institute of Pathology, ASST-Spedali Civili di Brescia, Brescia, Italy.

Laurenço Sbragia (L)

Department of Surgery and Anatomy, Ribeirão Preto Medical School, University of São Paulo, Ribeirão Preto, Brazil.

Marc Oria (M)

Center for Fetal and Placental Research, Division of Pediatric General and Thoracic Surgery, Cincinnati Children's Hospital Medical Center (CCHMC), Cincinnati, Ohio, USA.
University of Cincinnati, College of Medicine, Cincinnati, Ohio, USA.

Annalisa De Silvestri (A)

Biometry & Clinical Epidemiology, Scientific Direction, Fondazione IRCCS Policlinico San Matteo, Pavia, Italy.

Emanuela Mazzon (E)

IRCCS Centro Neurolesi "Bonino-Pulejo", Messina, Italy.

Valeria Calcaterra (V)

Pediatric and Adolescent Unit, Department of Internal Medicine, University of Pavia, Pavia, Italy.
Pediatric Department, "V Buzzi" Children's Hospital, Milan, Italy.

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