Obesity-Related Insulin Resistance: The Central Role of Adipose Tissue Dysfunction.

Adipose tissue insulin resistance Ceramides Diacylglycerols Hepatic insulin resistance Lipidomic remodeling Lipotoxicity Muscle insulin resistance

Journal

Handbook of experimental pharmacology
ISSN: 0171-2004
Titre abrégé: Handb Exp Pharmacol
Pays: Germany
ID NLM: 7902231

Informations de publication

Date de publication:
2022
Historique:
pubmed: 23 2 2022
medline: 26 7 2022
entrez: 22 2 2022
Statut: ppublish

Résumé

Obesity is a key player in the onset and progression of insulin resistance (IR), a state by which insulin-sensitive cells fail to adequately respond to insulin action. IR is a reversible condition, but if untreated leads to type 2 diabetes alongside increasing cardiovascular risk. The link between obesity and IR has been widely investigated; however, some aspects are still not fully characterized.In this chapter, we introduce key aspects of the pathophysiology of IR and its intimate connection with obesity. Specifically, we focus on the role of adipose tissue dysfunction (quantity, quality, and distribution) as a driver of whole-body IR. Furthermore, we discuss the obesity-related lipidomic remodeling occurring in adipose tissue, liver, and skeletal muscle. Key mechanisms linking lipotoxicity to IR in different tissues and metabolic alterations (i.e., fatty liver and diabetes) and the effect of weight loss on IR are also reported while highlighting knowledge gaps.

Identifiants

pubmed: 35192055
doi: 10.1007/164_2021_573
doi:

Substances chimiques

Insulins 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

145-164

Informations de copyright

© 2022. The Author(s), under exclusive license to Springer Nature Switzerland AG.

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Auteurs

Gabriele Mocciaro (G)

Institute of Clinical Physiology, National Research Council, Pisa, Italy.

Amalia Gastaldelli (A)

Institute of Clinical Physiology, National Research Council, Pisa, Italy. amalia@ifc.cnr.it.

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