Cerebral venous thrombosis without thrombocytopenia after a single dose of COVID-19 (Ad26.COV2.S) vaccine injection: a case report.


Journal

Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology
ISSN: 1590-3478
Titre abrégé: Neurol Sci
Pays: Italy
ID NLM: 100959175

Informations de publication

Date de publication:
May 2022
Historique:
received: 26 11 2021
accepted: 20 02 2022
pubmed: 27 2 2022
medline: 22 4 2022
entrez: 26 2 2022
Statut: ppublish

Résumé

The coronavirus pandemic became the hard challenge for the modern global health system. To date, vaccination is the best strategy against Sars-Cov-2-related illness. About 3 billions of people received at least one of the approved vaccines. The related adverse events were reported during the various experimental phases, but newer and less common side effects are emerging post-marketing. Vaccine-induced thrombocytopenia with thrombosis (VITT) is one of these insidious adverse reactions and it is considered responsible of venous thrombosis, in both the splanchnic and the cerebral circulation. Although its mechanism has been presumably established, resembling that observed in heparin-induced thrombocytopenia, some venous thromboses seem not to recognize this etiology and their pathogenesis remains unknown. Here we described a case of cerebral venous thrombosis after administration of the Ad26.COV2.S, presenting without thrombocytopenia, paving the way for possible novel causes of this vaccine-induced pathological condition. A 45-year-old woman came to our observation for bilateral periorbital headache associated with retro-orbital pain started 8 days after administration of COVID vaccine Jannsen. Ophthalmologic exam showing a bilateral papilledema raised the suspicion of intracranial hypertension. Cerebral magnetic resonance imaging revealed signal alteration with T1-positive contrast enhancement in the right temporal and insular lobes suggestive of cerebral venous thrombosis. The absence of thrombocytopenia and platelet factor 4 (PF-4) antibodies led the clinicians to rule out VITT. The patient was treated successfully with warfarin. Venous thrombosis occurring after COVID-19 vaccination represents an adverse event of special interest. Patients with thrombosis and thrombocytopenia appear to be affected by a general thrombophilic state, sustained by an autoimmune mechanism, and show a higher mortality. Thrombosis without thrombocytopenia's pathogenesis has not yet been clarified, but laboratory data and good response to vitamin K antagonists help clinicians in the differential diagnosis with VITT. Future research will allow us to discover other possible mechanisms and maybe identify a subgroup of patients with a higher risk of developing this medical complication.

Sections du résumé

BACKGROUND BACKGROUND
The coronavirus pandemic became the hard challenge for the modern global health system. To date, vaccination is the best strategy against Sars-Cov-2-related illness. About 3 billions of people received at least one of the approved vaccines. The related adverse events were reported during the various experimental phases, but newer and less common side effects are emerging post-marketing. Vaccine-induced thrombocytopenia with thrombosis (VITT) is one of these insidious adverse reactions and it is considered responsible of venous thrombosis, in both the splanchnic and the cerebral circulation. Although its mechanism has been presumably established, resembling that observed in heparin-induced thrombocytopenia, some venous thromboses seem not to recognize this etiology and their pathogenesis remains unknown. Here we described a case of cerebral venous thrombosis after administration of the Ad26.COV2.S, presenting without thrombocytopenia, paving the way for possible novel causes of this vaccine-induced pathological condition.
CASE PRESENTATION METHODS
A 45-year-old woman came to our observation for bilateral periorbital headache associated with retro-orbital pain started 8 days after administration of COVID vaccine Jannsen. Ophthalmologic exam showing a bilateral papilledema raised the suspicion of intracranial hypertension. Cerebral magnetic resonance imaging revealed signal alteration with T1-positive contrast enhancement in the right temporal and insular lobes suggestive of cerebral venous thrombosis. The absence of thrombocytopenia and platelet factor 4 (PF-4) antibodies led the clinicians to rule out VITT. The patient was treated successfully with warfarin.
CONCLUSION CONCLUSIONS
Venous thrombosis occurring after COVID-19 vaccination represents an adverse event of special interest. Patients with thrombosis and thrombocytopenia appear to be affected by a general thrombophilic state, sustained by an autoimmune mechanism, and show a higher mortality. Thrombosis without thrombocytopenia's pathogenesis has not yet been clarified, but laboratory data and good response to vitamin K antagonists help clinicians in the differential diagnosis with VITT. Future research will allow us to discover other possible mechanisms and maybe identify a subgroup of patients with a higher risk of developing this medical complication.

Identifiants

pubmed: 35217969
doi: 10.1007/s10072-022-05965-5
pii: 10.1007/s10072-022-05965-5
pmc: PMC8880295
doi:

Substances chimiques

Ad26COVS1 JT2NS6183B
COVID-19 Vaccines 0
Vaccines 0

Types de publication

Case Reports Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

2951-2956

Commentaires et corrections

Type : CommentIn

Informations de copyright

© 2022. Fondazione Società Italiana di Neurologia.

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Auteurs

Martina Di Pietro (M)

Department of Neuroscience, Imaging, and Clinical Sciences, "G. D'Annunzio" University of Chieti-Pescara, Chieti, Italy.

Fedele Dono (F)

Department of Neuroscience, Imaging, and Clinical Sciences, "G. D'Annunzio" University of Chieti-Pescara, Chieti, Italy. fedele.dono@unich.it.
Behavioral Neurology and Molecular Neurology Units, Center for Advanced Studies and Technology - CAST-, "G. D'Annunzio" University of Chieti-Pescara, Chieti, Italy. fedele.dono@unich.it.

Stefano Consoli (S)

Department of Neuroscience, Imaging, and Clinical Sciences, "G. D'Annunzio" University of Chieti-Pescara, Chieti, Italy.

Giacomo Evangelista (G)

Department of Neuroscience, Imaging, and Clinical Sciences, "G. D'Annunzio" University of Chieti-Pescara, Chieti, Italy.

Valeria Pozzilli (V)

Department of Neuroscience, Imaging, and Clinical Sciences, "G. D'Annunzio" University of Chieti-Pescara, Chieti, Italy.

Dario Calisi (D)

Department of Neuroscience, Imaging, and Clinical Sciences, "G. D'Annunzio" University of Chieti-Pescara, Chieti, Italy.

Filomena Barbone (F)

Department of Neurology, "SS Annunziata" Hospital, Chieti, Italy.

Laura Bonanni (L)

Department of Medicine and Aging Sciences, "G. D'Annunzio" University of Chieti-Pescara, Chieti, Italy.

Marco Onofrj (M)

Department of Neuroscience, Imaging, and Clinical Sciences, "G. D'Annunzio" University of Chieti-Pescara, Chieti, Italy.

Maria Vittoria De Angelis (MV)

Department of Neurology, Stroke Unit, "SS Annunziata" Hospital, Chieti, Italy.

Stefano L Sensi (SL)

Department of Neuroscience, Imaging, and Clinical Sciences, "G. D'Annunzio" University of Chieti-Pescara, Chieti, Italy. ssensi@unich.it.
Behavioral Neurology and Molecular Neurology Units, Center for Advanced Studies and Technology - CAST-, "G. D'Annunzio" University of Chieti-Pescara, Chieti, Italy. ssensi@unich.it.

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