How to Manage Inflammatory Bowel Disease Patients When They Withdraw Anti-Tumour Necrosis Factor [Anti-TNF] Due to Severe Anti-TNF-Induced Skin Lesions? A Multicentre Cohort Study.


Journal

Journal of Crohn's & colitis
ISSN: 1876-4479
Titre abrégé: J Crohns Colitis
Pays: England
ID NLM: 101318676

Informations de publication

Date de publication:
30 Aug 2022
Historique:
received: 15 12 2021
revised: 01 02 2022
accepted: 24 02 2022
pubmed: 27 2 2022
medline: 2 9 2022
entrez: 26 2 2022
Statut: ppublish

Résumé

Optimal management of patients with inflammatory bowel disease [IBD] after anti-tumour necrosis factor [TNF] discontinuation due to severe induced skin lesions is unclear. Our study aimed to describe dermatological and IBD evolution after anti-TNF discontinuation for this side effect. We conducted a multicentre retrospective study including consecutive IBD patients who discontinued anti-TNF due to severe induced skin lesions. Our objectives were to determine factors associated with dermatological remission [complete disappearance of skin lesions] and with IBD relapse in patients with inactive disease at inclusion, notably the impact of an early switch to another biological agent within 3 months of anti-TNF discontinuation. Among the 181 patients [134 women, 160 Crohn's disease] included in the 13 participating centres, dermatological remission occurred in 110 [62%] patients with a median [interquartile range, IQR] interval of 8.0 [6.8-11.0] months. Scalp location was independently associated with less remission of skin lesions (hazard ratio [HR] = 0.64 [95% CI 0.43-0.94], p = 0.02) while early switch was independently associated with a higher probability of remission of skin lesions (HR = 1.64 [95% CI 1.1-2.5], p = 0.02). Among the 148 patients with inactive IBD at inclusion, disease relapse occurred in 75 [51%] patients with a median [IQR] interval of 26.0 [23.0-39.1] months. Survival rates without IBD relapse at 1 year were 85.8% [95% CI 77.5-94.9] in the early switch group and 59.3% [95% CI 48.9-71.9] in the other group [p < 0.01]. Early switch to a new biological is associated with a higher probability of healing of anti-TNF-induced skin lesions and significantly reduces the risk of IBD relapse.

Sections du résumé

BACKGROUND AND AIMS OBJECTIVE
Optimal management of patients with inflammatory bowel disease [IBD] after anti-tumour necrosis factor [TNF] discontinuation due to severe induced skin lesions is unclear. Our study aimed to describe dermatological and IBD evolution after anti-TNF discontinuation for this side effect.
METHODS METHODS
We conducted a multicentre retrospective study including consecutive IBD patients who discontinued anti-TNF due to severe induced skin lesions. Our objectives were to determine factors associated with dermatological remission [complete disappearance of skin lesions] and with IBD relapse in patients with inactive disease at inclusion, notably the impact of an early switch to another biological agent within 3 months of anti-TNF discontinuation.
RESULTS RESULTS
Among the 181 patients [134 women, 160 Crohn's disease] included in the 13 participating centres, dermatological remission occurred in 110 [62%] patients with a median [interquartile range, IQR] interval of 8.0 [6.8-11.0] months. Scalp location was independently associated with less remission of skin lesions (hazard ratio [HR] = 0.64 [95% CI 0.43-0.94], p = 0.02) while early switch was independently associated with a higher probability of remission of skin lesions (HR = 1.64 [95% CI 1.1-2.5], p = 0.02). Among the 148 patients with inactive IBD at inclusion, disease relapse occurred in 75 [51%] patients with a median [IQR] interval of 26.0 [23.0-39.1] months. Survival rates without IBD relapse at 1 year were 85.8% [95% CI 77.5-94.9] in the early switch group and 59.3% [95% CI 48.9-71.9] in the other group [p < 0.01].
CONCLUSIONS CONCLUSIONS
Early switch to a new biological is associated with a higher probability of healing of anti-TNF-induced skin lesions and significantly reduces the risk of IBD relapse.

Identifiants

pubmed: 35218189
pii: 6537448
doi: 10.1093/ecco-jcc/jjac035
doi:

Substances chimiques

Tumor Necrosis Factor Inhibitors 0
Tumor Necrosis Factor-alpha 0
Infliximab B72HH48FLU
Adalimumab FYS6T7F842

Types de publication

Journal Article Multicenter Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

1202-1210

Informations de copyright

© The Author(s) 2022. Published by Oxford University Press on behalf of European Crohn’s and Colitis Organisation. All rights reserved. For permissions, please email: journals.permissions@oup.com.

Auteurs

C Cottron (C)

CHU de Bordeaux, Hôpital Haut-Lévêque, Service d'Hépato-gastroentérologie et oncologie digestive - Université de Bordeaux, F-33000 Bordeaux, France.

X Treton (X)

Department of Gastroenterology, IBD and Nutrition, Beaujon Hospital, APHP, Paris, France.

R Altwegg (R)

Department of Hepatogastroenterology, Saint Eloi Hospital, CHU de Montpellier, Montpellier, France.

C Reenaers (C)

Department of Gastroenterology, CHU Sart Tilman, Liège, Belgium.

A Amiot (A)

Department of Gastroenterology, Henri Mondor Hospital, APHP, Creteil, France.

M Fumery (M)

Department of Gastroenterology, CHU de Amiens, and Peritox, UMR I-01, France.

L Vuitton (L)

Department of Hepatogastroenterology, CHRU de Besançon, Besançon, France.

L Peyrin-Biroulet (L)

Department of Hepatogastroenterology, Nancy University Hospital, Vandoeuvre les Nancy, France.

G Bouguen (G)

CHU Rennes, Univ Rennes, INSERM, CIC1414, Institut NUMECAN (Nutrition Metabolism and Cancer), F-35000 Rennes, France.

O Dewit (O)

Department of Gastroenterology, Université Catholique de Louvain Saint Luc, Brussels, Belgium.

S Nancey (S)

Department of Gastroenterology, CHU de Lyon, Lyon Sud Hospital, University Claude Bernard Lyon 1, INSERM U1111, Lyon, France.

L Caillo (L)

Department of Hepatogastroenterology, CHU de Nîmes, Nîmes, France.

X Roblin (X)

Department of Hepatogastroenterology, CHU de Saint-Etienne, Hôpital Nord, Université Jean Monnet, Saint-Etienne, France.

M Beylot-Barry (M)

Department of Dermatology, Saint-André Hospital, CHU de Bordeaux, France.

P Rivière (P)

CHU de Bordeaux, Hôpital Haut-Lévêque, Service d'Hépato-gastroentérologie et oncologie digestive - Université de Bordeaux, F-33000 Bordeaux, France.

D Laharie (D)

CHU de Bordeaux, Hôpital Haut-Lévêque, Service d'Hépato-gastroentérologie et oncologie digestive - Université de Bordeaux, F-33000 Bordeaux, France.

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Classifications MeSH