Death Receptor 6 as a Prognostic Marker in Low-grade Glioma.


Journal

Anticancer research
ISSN: 1791-7530
Titre abrégé: Anticancer Res
Pays: Greece
ID NLM: 8102988

Informations de publication

Date de publication:
Mar 2022
Historique:
received: 01 11 2021
revised: 30 11 2021
accepted: 07 12 2021
entrez: 27 2 2022
pubmed: 28 2 2022
medline: 8 3 2022
Statut: ppublish

Résumé

Low-grade gliomas (LGG) are heterogenous tumours, causing variable survivals in patients. Identifying molecular markers for a more accurate prognosis is, therefore, important. Since death receptor 6 (DR6) is up-regulated in gliomas and shows an aberrant signalling network, we tested its suitability as a prognostic marker. DR6 was investigated in patient samples via PCR and western blot. Clinical data were analysed and compared to The Cancer Genome Atlas (TCGA) 'brain lower grade glioma' dataset. DR6 was found to be enhanced in LGG and its expression increased in recurrent LGG. The receptor showed a protective effect in primary LGG with a significantly elongated progression-free survival that was confirmed in the TCGA study. This effect was reversed in relapsed LGG in which cases with high DR6 expression reveal a shorter overall survival. DR6 is an interesting candidate for further studies regarding its effect as a prognostic marker, playing an opposing role in primary and relapsed LGG.

Sections du résumé

BACKGROUND/AIM OBJECTIVE
Low-grade gliomas (LGG) are heterogenous tumours, causing variable survivals in patients. Identifying molecular markers for a more accurate prognosis is, therefore, important. Since death receptor 6 (DR6) is up-regulated in gliomas and shows an aberrant signalling network, we tested its suitability as a prognostic marker.
MATERIALS AND METHODS METHODS
DR6 was investigated in patient samples via PCR and western blot. Clinical data were analysed and compared to The Cancer Genome Atlas (TCGA) 'brain lower grade glioma' dataset.
RESULTS RESULTS
DR6 was found to be enhanced in LGG and its expression increased in recurrent LGG. The receptor showed a protective effect in primary LGG with a significantly elongated progression-free survival that was confirmed in the TCGA study. This effect was reversed in relapsed LGG in which cases with high DR6 expression reveal a shorter overall survival.
CONCLUSION CONCLUSIONS
DR6 is an interesting candidate for further studies regarding its effect as a prognostic marker, playing an opposing role in primary and relapsed LGG.

Identifiants

pubmed: 35220214
pii: 42/3/1237
doi: 10.21873/anticanres.15591
doi:

Substances chimiques

Biomarkers, Tumor 0
Receptors, Tumor Necrosis Factor 0
TNFRSF21 protein, human 0
Isocitrate Dehydrogenase EC 1.1.1.41
IDH1 protein, human EC 1.1.1.42.

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1237-1245

Informations de copyright

Copyright © 2022 International Institute of Anticancer Research (Dr. George J. Delinasios), All rights reserved.

Auteurs

Sarah K Stegmann (SK)

Laboratory for Neurooncology and Experimental Neurosurgery, Department of General Neurosurgery, Center for Neurosurgery, Cologne, Germany.
Faculty of Medicine and University Hospital Cologne, Cologne, Germany.

Saskia Kuhl (S)

Laboratory for Neurooncology and Experimental Neurosurgery, Department of General Neurosurgery, Center for Neurosurgery, Cologne, Germany.
Faculty of Medicine and University Hospital Cologne, Cologne, Germany.

Nam Gyu Im (NG)

Faculty of Medicine and University Hospital Cologne, Cologne, Germany.

Dang Thuy Diem-Dinh (DT)

Laboratory for Neurooncology and Experimental Neurosurgery, Department of General Neurosurgery, Center for Neurosurgery, Cologne, Germany.
Faculty of Medicine and University Hospital Cologne, Cologne, Germany.

Lukas Goertz (L)

Laboratory for Neurooncology and Experimental Neurosurgery, Department of General Neurosurgery, Center for Neurosurgery, Cologne, Germany.
Faculty of Medicine and University Hospital Cologne, Cologne, Germany.

Roland Goldbrunner (R)

Laboratory for Neurooncology and Experimental Neurosurgery, Department of General Neurosurgery, Center for Neurosurgery, Cologne, Germany.
Faculty of Medicine and University Hospital Cologne, Cologne, Germany.

Marco Timmer (M)

Laboratory for Neurooncology and Experimental Neurosurgery, Department of General Neurosurgery, Center for Neurosurgery, Cologne, Germany; marco.timmer@uk-koeln.de.
Faculty of Medicine and University Hospital Cologne, Cologne, Germany.

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Classifications MeSH