CD39/CD73 Dysregulation of Adenosine Metabolism Increases Decidual Natural Killer Cell Cytotoxicity: Implications in Unexplained Recurrent Spontaneous Abortion.
CD39
CD73
TGF-β
URSA
adenosine
dNK
Journal
Frontiers in immunology
ISSN: 1664-3224
Titre abrégé: Front Immunol
Pays: Switzerland
ID NLM: 101560960
Informations de publication
Date de publication:
2022
2022
Historique:
received:
11
11
2021
accepted:
24
01
2022
entrez:
28
2
2022
pubmed:
1
3
2022
medline:
9
4
2022
Statut:
epublish
Résumé
Unexplained recurrent spontaneous abortion (URSA) is believed to be associated with impaired immunosuppression at the maternal-fetal interface, but the detailed molecular mechanism remains unclear. The ATP-adenosine metabolic pathway regulated by CD39/CD73 has recently been recognized to be important in immunosuppression. This study aimed to investigate the regulation of decidual natural killer (dNK) cells and fetal extravillous trophoblast (EVT) cells by CD39 and CD73 in URSA, as well as the possible regulatory mechanism of CD39/CD73
Identifiants
pubmed: 35222389
doi: 10.3389/fimmu.2022.813218
pmc: PMC8866181
doi:
Substances chimiques
Transforming Growth Factor beta
0
Adenosine Triphosphate
8L70Q75FXE
TOR Serine-Threonine Kinases
EC 2.7.11.1
Adenosine
K72T3FS567
Types de publication
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
813218Informations de copyright
Copyright © 2022 Zhu, Song, Zhou, Han, Yu, Chen, Mansell, Novakovic, Baker, Cannon, Saffery, Chen and Zhang.
Déclaration de conflit d'intérêts
The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
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