Homocysteine levels correlate with velocimetric parameters in patients with erectile dysfunction undergoing penile duplex ultrasound.


Journal

Andrology
ISSN: 2047-2927
Titre abrégé: Andrology
Pays: England
ID NLM: 101585129

Informations de publication

Date de publication:
05 2022
Historique:
revised: 15 02 2022
received: 04 01 2022
accepted: 24 02 2022
pubmed: 1 3 2022
medline: 23 4 2022
entrez: 28 2 2022
Statut: ppublish

Résumé

Hyperhomocysteinemia may contribute to the development of endothelial dysfunction and, consequently, atherosclerosis, a systemic disease involving the vessels that may affect the cavernous arteries leading to vasculogenic erectile dysfunction. Our study aims therefore to explore the relationship between homocysteine levels and velocimetric parameters detected by basal penile duplex ultrasound such as peak systolic velocity and flaccid penile acceleration in patients with erectile dysfunction. A cross-sectional study was conducted collecting clinical, metabolic, hormonal, and instrumental (basal penile duplex ultrasound) data in patients affected by vasculogenic erectile dysfunction. Data of 126 subjects affected by erectile dysfunction were collected. Mean age was 52.1 ± 12.6 years, whereas mean body mass index was 25.6 ± 4.0 kg/m Hyperhomocysteinemia is highly prevalent in erectile dysfunction patients. The results of our study show that homocysteine levels correlate with velocimetric parameters assessed by basal penile duplex ultrasound, confirming the role of hyperhomocysteinemia in the genesis of erectile dysfunction of arterial origin.

Identifiants

pubmed: 35224883
doi: 10.1111/andr.13169
pmc: PMC9310719
doi:

Substances chimiques

Homocysteine 0LVT1QZ0BA

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

733-739

Informations de copyright

© 2022 American Society of Andrology and European Academy of Andrology.

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Auteurs

Gianmaria Salvio (G)

Division of Endocrinology, Department of Clinical and Molecular Sciences, Polytechnic University of Marche, Ospedali Riuniti, Ancona, Italy.

Alessandro Ciarloni (A)

Division of Endocrinology, Department of Clinical and Molecular Sciences, Polytechnic University of Marche, Ospedali Riuniti, Ancona, Italy.

Simone Cordoni (S)

Division of Endocrinology, Department of Clinical and Molecular Sciences, Polytechnic University of Marche, Ospedali Riuniti, Ancona, Italy.

Melissa Cutini (M)

Division of Endocrinology, Department of Clinical and Molecular Sciences, Polytechnic University of Marche, Ospedali Riuniti, Ancona, Italy.

Nicola Delli Muti (N)

Division of Endocrinology, Department of Clinical and Molecular Sciences, Polytechnic University of Marche, Ospedali Riuniti, Ancona, Italy.

Federica Finocchi (F)

Division of Endocrinology, Department of Clinical and Molecular Sciences, Polytechnic University of Marche, Ospedali Riuniti, Ancona, Italy.

Francesca Firmani (F)

Division of Endocrinology, Department of Clinical and Molecular Sciences, Polytechnic University of Marche, Ospedali Riuniti, Ancona, Italy.

Lara Giovannini (L)

Division of Endocrinology, Department of Clinical and Molecular Sciences, Polytechnic University of Marche, Ospedali Riuniti, Ancona, Italy.

Michele Perrone (M)

Division of Endocrinology, Department of Clinical and Molecular Sciences, Polytechnic University of Marche, Ospedali Riuniti, Ancona, Italy.

Giancarlo Balercia (G)

Division of Endocrinology, Department of Clinical and Molecular Sciences, Polytechnic University of Marche, Ospedali Riuniti, Ancona, Italy.

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