Reappraisal of liver transplantation for erythropoietic protoporphyria: A deadly combination of disease recurrence and biliary complication.


Journal

Pediatric transplantation
ISSN: 1399-3046
Titre abrégé: Pediatr Transplant
Pays: Denmark
ID NLM: 9802574

Informations de publication

Date de publication:
06 2022
Historique:
revised: 23 01 2022
received: 03 09 2021
accepted: 17 02 2022
pubmed: 1 3 2022
medline: 12 5 2022
entrez: 28 2 2022
Statut: ppublish

Résumé

Erythropoietic protoporphyria (EPP) is a rare inherited disorder that causes the accumulation of protoporphyrin in the erythrocytes, skin, and liver. Severe protoporphyric hepatopathy results in liver failure, requiring both liver and bone marrow transplantation as a life-saving procedure and to correct the underlying enzymatic defect, respectively. We report a 20-year-old man who underwent split liver transplantation using a right trisegment and caudate lobe graft for EPP-induced liver failure, but succumbed to a deadly combination of early relapse of EPP and subsequent, intractable, late-onset bile leakage from the cut surface of segment 4. EPP recurrence most likely created a high-risk situation for bile leakage from the non-communicating bile ducts of segment 4; therefore, this case shed light on the potential relationship between EPP recurrence and biliary complications. Physicians should recognize the potentially rapid and life-threatening progression of protoporphyric hepatopathy that leads to liver failure. For young patients with EPP, LT and sequential BMT should thoroughly be considered by a multidisciplinary team as soon as hepatic reserve deterioration becomes evident. Split liver transplantation should preferably be avoided and appropriate post-transplant management is critical before protoporphyrin depositions to the bile duct and hepatocyte causes irreversible damage to the liver graft.

Sections du résumé

BACKGROUND
Erythropoietic protoporphyria (EPP) is a rare inherited disorder that causes the accumulation of protoporphyrin in the erythrocytes, skin, and liver. Severe protoporphyric hepatopathy results in liver failure, requiring both liver and bone marrow transplantation as a life-saving procedure and to correct the underlying enzymatic defect, respectively.
CASE PRESENTATION
We report a 20-year-old man who underwent split liver transplantation using a right trisegment and caudate lobe graft for EPP-induced liver failure, but succumbed to a deadly combination of early relapse of EPP and subsequent, intractable, late-onset bile leakage from the cut surface of segment 4. EPP recurrence most likely created a high-risk situation for bile leakage from the non-communicating bile ducts of segment 4; therefore, this case shed light on the potential relationship between EPP recurrence and biliary complications.
CONCLUSION
Physicians should recognize the potentially rapid and life-threatening progression of protoporphyric hepatopathy that leads to liver failure. For young patients with EPP, LT and sequential BMT should thoroughly be considered by a multidisciplinary team as soon as hepatic reserve deterioration becomes evident. Split liver transplantation should preferably be avoided and appropriate post-transplant management is critical before protoporphyrin depositions to the bile duct and hepatocyte causes irreversible damage to the liver graft.

Identifiants

pubmed: 35225415
doi: 10.1111/petr.14261
doi:

Substances chimiques

Protoporphyrins 0

Types de publication

Case Reports

Langues

eng

Sous-ensembles de citation

IM

Pagination

e14261

Informations de copyright

© 2022 Wiley Periodicals LLC.

Références

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Auteurs

Yutaka Endo (Y)

Department of Surgery, Keio University School of Medicine, Tokyo, Japan.

Taizo Hibi (T)

Department of Pediatric Surgery and Transplantation, Kumamoto University School of Medicine, Kumamoto, Japan.

Masahiro Shinoda (M)

Department of Surgery, Keio University School of Medicine, Tokyo, Japan.
Department of Digestive Center, International University of Health and Welfare Mita Hospital, Tokyo, Japan.

Hideaki Obara (H)

Department of Surgery, Keio University School of Medicine, Tokyo, Japan.

Minoru Kitago (M)

Department of Surgery, Keio University School of Medicine, Tokyo, Japan.

Hiroshi Yagi (H)

Department of Surgery, Keio University School of Medicine, Tokyo, Japan.

Yuta Abe (Y)

Department of Surgery, Keio University School of Medicine, Tokyo, Japan.

Yasushi Hasegawa (Y)

Department of Surgery, Keio University School of Medicine, Tokyo, Japan.

Kentaro Matsubara (K)

Department of Surgery, Keio University School of Medicine, Tokyo, Japan.

Shutaro Hori (S)

Department of Surgery, Keio University School of Medicine, Tokyo, Japan.

Masayuki Tanaka (M)

Department of Surgery, Keio University School of Medicine, Tokyo, Japan.

Satomi Makiuchi (S)

Department of Pathology, Keio University School of Medicine, Tokyo, Japan.

Yutaka Nakano (Y)

Department of Surgery, Keio University School of Medicine, Tokyo, Japan.

Osamu Itano (O)

Department of Gastrointestinal Surgery, International University of Health and Welfare, Chiba, Japan.

Tatsuo Kuroda (T)

Department of Pediatric Surgery, Keio University School of Medicine, Tokyo, Japan.

Yuko Kitagawa (Y)

Department of Surgery, Keio University School of Medicine, Tokyo, Japan.

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